Tuberculosis chemotherapy: the influence of bacillary stress and damage response pathways on drug efficacy.
about
Comprehensive functional analysis of Mycobacterium tuberculosis toxin-antitoxin systems: implications for pathogenesis, stress responses, and evolutionTriazaspirodimethoxybenzoyls as Selective Inhibitors of Mycobacterial Lipoamide Dehydrogenase,Spontaneous emergence of multiple drug resistance in tuberculosis before and during therapyTwo-drug antimicrobial chemotherapy: a mathematical model and experiments with Mycobacterium marinumEssential roles for imuA'- and imuB-encoded accessory factors in DnaE2-dependent mutagenesis in Mycobacterium tuberculosisInduced ectopic expression of HigB toxin in Mycobacterium tuberculosis results in growth inhibition, reduced abundance of a subset of mRNAs and cleavage of tmRNABactericidal activity of an imidazo[1, 2-a]pyridine using a mouse M. tuberculosis infection modelMycolic acids as diagnostic markers for tuberculosis case detection in humans and drug efficacy in miceMetabolic Perspectives on PersistenceWhy is long-term therapy required to cure tuberculosis?Identification of gene targets against dormant phase Mycobacterium tuberculosis infectionsDistinct specificities of Mycobacterium tuberculosis and mammalian proteasomes for N-acetyl tripeptide substrates.The three RelE homologs of Mycobacterium tuberculosis have individual, drug-specific effects on bacterial antibiotic toleranceActive starvation responses mediate antibiotic tolerance in biofilms and nutrient-limited bacteriaRole of the DinB homologs Rv1537 and Rv3056 in Mycobacterium tuberculosisVapC toxins from Mycobacterium tuberculosis are ribonucleases that differentially inhibit growth and are neutralized by cognate VapB antitoxins.Either non-homologous ends joining or homologous recombination is required to repair double-strand breaks in the genome of macrophage-internalized Mycobacterium tuberculosisDifferent transcriptional profiles of RAW264.7 infected with Mycobacterium tuberculosis H37Rv and BCG identified via deep sequencingIdentification of novel inhibitors of nonreplicating Mycobacterium tuberculosis using a carbon starvation modelSecreted acid phosphatase (SapM) of Mycobacterium tuberculosis is indispensable for arresting phagosomal maturation and growth of the pathogen in guinea pig tissuesDrug tolerance in replicating mycobacteria mediated by a macrophage-induced efflux mechanism.In-Vivo Gene Signatures of Mycobacterium tuberculosis in C3HeB/FeJ Mice.Strain specific transcriptional response in Mycobacterium tuberculosis infected macrophages.Eradication of bacterial persisters with antibiotic-generated hydroxyl radicals.Mycobacterium tuberculosis RecG binds and unwinds model DNA substrates with a preference for Holliday junctions.Multifunctional essentiality of succinate metabolism in adaptation to hypoxia in Mycobacterium tuberculosis.Immune activation of the host cell induces drug tolerance in Mycobacterium tuberculosis both in vitro and in vivo.A Flow Cytometry Method for Rapidly Assessing Mycobacterium tuberculosis Responses to Antibiotics with Different Modes of ActionProblems in diagnosis and treatment of tuberculosis infectionTuberculosis genes expressed during persistence and reactivation in the resistant rabbit model.The DosR regulon of M. tuberculosis and antibacterial tolerance.Extensively drug-resistant tuberculosis: new strains, new challenges.The uracil DNA glycosylase UdgB of Mycobacterium smegmatis protects the organism from the mutagenic effects of cytosine and adenine deamination.A replication clock for Mycobacterium tuberculosis.DNA repair in Mycobacterium tuberculosis revisited.A TetR family transcriptional factor directly regulates the expression of a 3-methyladenine DNA glycosylase and physically interacts with the enzyme to stimulate its base excision activity in Mycobacterium bovis BCGAntimicrobial treatment improves mycobacterial survival in nonpermissive growth conditions.MenA is a promising drug target for developing novel lead molecules to combat Mycobacterium tuberculosis.DNA repair systems and the pathogenesis of Mycobacterium tuberculosis: varying activities at different stages of infection.Metabolic regulation of antibiotic resistance.
P2860
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P2860
Tuberculosis chemotherapy: the influence of bacillary stress and damage response pathways on drug efficacy.
description
2006 nî lūn-bûn
@nan
2006 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2006年の論文
@ja
2006年論文
@yue
2006年論文
@zh-hant
2006年論文
@zh-hk
2006年論文
@zh-mo
2006年論文
@zh-tw
2006年论文
@wuu
name
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@ast
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@en
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@nl
type
label
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@ast
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@en
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@nl
prefLabel
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@ast
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@en
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@nl
P2860
P356
P1476
Tuberculosis chemotherapy: the ...... nse pathways on drug efficacy.
@en
P2860
P304
P356
10.1128/CMR.00060-05
P407
P577
2006-07-01T00:00:00Z