The estrogenic effect of bisphenol A disrupts pancreatic beta-cell function in vivo and induces insulin resistance - Wikidata - wikimedia - TriplyDB

The estrogenic effect of bisphenol A disrupts pancreatic beta-cell function in vivo and induces insulin resistance

The estrogenic effect of bisphenol A disrupts pancreatic beta-cell function in vivo and induces insulin resistance

http://www.wikidata.org/entity/Q24536253

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Association of urinary bisphenol a concentration with heart disease: evidence from NHANES 2003/06 Is bisphenol-A exposure during pregnancy associated with blood glucose levels or diagnosis of gestational diabetes?Basic exploratory research versus guideline-compliant studies used for hazard evaluation and risk assessment: bisphenol A as a case study Hormones and endocrine-disrupting chemicals: low-dose effects and nonmonotonic dose responses The estrogenic endocrine disrupting chemical bisphenol A (BPA) and obesity Endocrine-disrupting chemicals: an Endocrine Society scientific statement Why public health agencies cannot depend on good laboratory practices as a criterion for selecting data: the case of bisphenol A Prenatal exposure to bisphenol a at environmentally relevant doses adversely affects the murine female reproductive tract later in life Concentrations of urinary phthalate metabolites are associated with increased waist circumference and insulin resistance in adult U.S. males In vivo effects of bisphenol A in laboratory rodent studies Prenatal Exposure to BPA and Offspring Outcomes: The Diabesogenic Behavior of BPA Neurological Effects of Bisphenol A and its Analogues Bisphenol-A and Female Infertility: A Possible Role of Gene-Environment Interactions Polycystic ovary syndrome: do endocrine-disrupting chemicals play a role?Early life factors and type 2 diabetes mellitus Insulin resistance and environmental pollutants: experimental evidence and future perspectives Environmental chemicals and type 2 diabetes: an updated systematic review of the epidemiologic evidence Pancreatic insulin content regulation by the estrogen receptor ER alpha Exposure to bisphenol-A during pregnancy partially mimics the effects of a high-fat diet altering glucose homeostasis and gene expression in adult male mice Influence of Bisphenol A on Type 2 Diabetes Mellitus Environmental causes of cancer: endocrine disruptors as carcinogens EDC-2: The Endocrine Society's Second Scientific Statement on Endocrine-Disrupting Chemicals Bisphenol A affects early bovine embryo development and metabolism that is negated by an oestrogen receptor inhibitor Association of urinary bisphenol a concentration with type-2 diabetes mellitus Bisphenol-A and disparities in birth outcomes: a review and directions for future research Association of Bisphenol A Exposure With Hypertension and Early Macrovascular Diseases in Chinese Adults: A Cross-Sectional Study Urinary bisphenol A and obesity: NHANES 2003-2006 Urinary bisphenol a concentration and angiography-defined coronary artery stenosis The paradox of progress: environmental disruption of metabolism and the diabetes epidemic Scientific Statement on the Diagnostic Criteria, Epidemiology, Pathophysiology, and Molecular Genetics of Polycystic Ovary Syndrome.Disconcordance in statistical models of bisphenol A and chronic disease outcomes in NHANES 2003-08 Bisphenol-A: epigenetic reprogramming and effects on reproduction and behavior Obesity, diabetes, and associated costs of exposure to endocrine-disrupting chemicals in the European Union Sex differences in the association of urinary bisphenol-A concentration with selected indices of glucose homeostasis among U.S. adults Maternal bisphenol a exposure during pregnancy and its association with adipokines in Mexican-American children Rapid insulinotropic action of low doses of bisphenol-A on mouse and human islets of Langerhans: role of estrogen receptor β Short-term treatment with bisphenol-A leads to metabolic abnormalities in adult male mice Insulinotropic effect of the non-steroidal compound STX in pancreatic β-cells Perinatal bisphenol A exposure and adult glucose homeostasis: identifying critical windows of exposure Mechanisms of estradiol-induced insulin secretion by the G protein-coupled estrogen receptor GPR30/GPER in pancreatic beta-cells

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The estrogenic effect of bisphenol A disrupts pancreatic beta-cell function in vivo and induces insulin resistance

http://www.wikidata.org/entity/Q24536253

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2006 nî lūn-bûn

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2006 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2006 թվականի հունվարին հրատարակված գիտական հոդված

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2006年の論文

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The estrogenic effect of bisph ...... and induces insulin resistance

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The estrogenic effect of bisph ...... and induces insulin resistance

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The estrogenic effect of bisph ...... and induces insulin resistance

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The estrogenic effect of bisph ...... and induces insulin resistance

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The estrogenic effect of bisph ...... and induces insulin resistance

@en

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Cristina Ripoll

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Esther Fuentes

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P2860

Biotransformations of bisphenol A in a mammalian model: answers and new questions raised by low-dose metabolic fate studies in pregnant CD1 mice

An extensive new literature concerning low-dose effects of bisphenol A shows the need for a new risk assessment

Developmental effects of endocrine-disrupting chemicals in wildlife and humans

Junctional communication of pancreatic beta cells contributes to the control of insulin secretion and glucose tolerance

Linking dioxins to diabetes: epidemiology and biologic plausibility

Parent bisphenol A accumulation in the human maternal-fetal-placental unit

Origins of obesity.

The environmental estrogen bisphenol a inhibits estradiol-induced hippocampal synaptogenesis.

Xenoestrogens at picomolar to nanomolar concentrations trigger membrane estrogen receptor-alpha-mediated Ca2+ fluxes and prolactin release in GH3/B6 pituitary tumor cells

Low doses of bisphenol A and diethylstilbestrol impair Ca2+ signals in pancreatic alpha-cells through a nonclassical membrane estrogen receptor within intact islets of Langerhans.

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10.1289/EHP.8451

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249884984

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16393666

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