Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain
about
Temperature dependence of erythromelalgia mutation L858F in sodium channel Nav1.7Na(v) 1.8-null mice show stimulus-dependent deficits in spinal neuronal activity.Pain as a channelopathyDistinct Nav1.7-dependent pain sensations require different sets of sensory and sympathetic neuronsSynthetic ciguatoxins selectively activate Nav1.8-derived chimeric sodium channels expressed in HEK293 cellsA Nav1.7 channel mutation associated with hereditary erythromelalgia contributes to neuronal hyperexcitability and displays reduced lidocaine sensitivityNeuropathic pain develops normally in mice lacking both Na(v)1.7 and Na(v)1.8.Conotoxins That Could Provide Analgesia through Voltage Gated Sodium Channel InhibitionPrimary erythromelalgia: a reviewAMPK: An emerging target for modification of injury-induced pain plasticity.Endogenous opioids contribute to insensitivity to pain in humans and mice lacking sodium channel Nav1.7Nav1.7 and other voltage-gated sodium channels as drug targets for pain reliefNeurobiology: a channel sets the gain on painPain genesGlobal Nav1.7 knockout mice recapitulate the phenotype of human congenital indifference to painModeling nociception in zebrafish: a way forward for unbiased analgesic discoveryMouse DRG Cell Line with Properties of NociceptorsInhibition of Inactive States of Tetrodotoxin-Sensitive Sodium Channels Reduces Spontaneous Firing of C-Fiber Nociceptors and Produces Analgesia in Formalin and Complete Freund's Adjuvant Models of PainTransient opening of the perineurial barrier for analgesic drug deliveryContinuous delta-opioid receptor activation reduces neuronal voltage-gated sodium channel (NaV1.7) levels through activation of protein kinase C in painful diabetic neuropathyTargeting neuroprotection as an alternative approach to preventing and treating neuropathic painCellular and Molecular Mechanisms of PainEstradiol upregulates voltage-gated sodium channel 1.7 in trigeminal ganglion contributing to hyperalgesia of inflamed TMJ.Voltage-gated Na+ channels in neuropathic pain.Loss-of-function mutations in sodium channel Nav1.7 cause anosmia.In vivo characterization of distinct modality-specific subsets of somatosensory neurons using GCaMP.Pharmacological characterisation of the highly NaV1.7 selective spider venom peptide Pn3a.Sensory-Evoked Intrinsic Imaging Signals in the Olfactory Bulb Are Independent of Neurovascular Coupling.Molecular profiling of murine sensory neurons in the nodose and dorsal root ganglia labeled from the peritoneal cavity.Nav1.7 expression is increased in painful human dental pulpAquaporin-1 tunes pain perception by interaction with Na(v)1.8 Na+ channels in dorsal root ganglion neurons.Identifying the Ion Channels Responsible for Signaling Gastro-Intestinal Based PainVoltage-gated Na+ currents in human dorsal root ganglion neurons.Loss-of-function mutations in SLC30A8 protect against type 2 diabetesAn in vivo tethered toxin approach for the cell-autonomous inactivation of voltage-gated sodium channel currents in nociceptors.Challenges in the development of novel treatment strategies for neuropathic pain.Significant determinants of mouse pain behaviour.An approach to identify microRNAs involved in neuropathic pain following a peripheral nerve injury.Tetrodotoxin (TTX) as a therapeutic agent for pain.Identification of molecular pathologies sufficient to cause neuropathic excitability in primary somatosensory afferents using dynamical systems theory.
P2860
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P2860
Nociceptor-specific gene deletion reveals a major role for Nav1.7 (PN1) in acute and inflammatory pain
description
2004 nî lūn-bûn
@nan
2004 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2004年の論文
@ja
2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
@wuu
name
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@ast
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@en
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@nl
type
label
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@ast
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@en
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@nl
prefLabel
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@ast
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@en
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@nl
P2093
P2860
P3181
P356
P1476
Nociceptor-specific gene delet ...... in acute and inflammatory pain
@en
P2093
Anthony H Dickenson
Elizabeth A Matthews
John N Wood
L Caroline Stirling
Mark D Baker
Mohammed A Nassar
P2860
P304
12706-12711
P3181
P356
10.1073/PNAS.0404915101
P407
P577
2004-08-16T00:00:00Z