Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
about
Na(+)/monocarboxylate transport (SMCT) protein expression correlates with survival in colon cancer: molecular characterization of SMCTEffects of T208E activating mutation on MARK2 protein structure and dynamics: Modeling and simulation.Par1b promotes hepatic-type lumen polarity in Madin Darby canine kidney cells via myosin II- and E-cadherin-dependent signaling.Differential roles for actin polymerization and a myosin II motor in assembly of the epithelial apical junctional complex.Loss of the Par-1b/MARK2 polarity kinase leads to increased metabolic rate, decreased adiposity, and insulin hypersensitivity in vivoCoordinated protein sorting, targeting and distribution in polarized cellsPKCeta is required for beta1gamma2/beta3gamma2- and PKD-mediated transport to the cell surface and the organization of the Golgi apparatusTransporters on demand: intracellular reservoirs and cycling of bile canalicular ABC transporters.Polarization and myelination in myelinating glia.Extending from PARs in Caenorhabditis elegans to homologues in Haemonchus contortus and other parasitic nematodes.LKB1 induces apical trafficking of Silnoon, a monocarboxylate transporter, in Drosophila melanogasterHepatocyte polarityRole of membrane traffic in the generation of epithelial cell asymmetry.Which in vitro models could be best used to study hepatocyte polarity?Remodeling epithelial cell organization: transitions between front-rear and apical-basal polarityAutotaxin, an ectoenzyme that produces lysophosphatidic acid, promotes the entry of lymphocytes into secondary lymphoid organs.Apical trafficking in epithelial cells: signals, clusters and motors.Rho signaling and tight junction functions.Molecular regulation of lumen morphogenesis.The unique polarity phenotype of hepatocytes.Morphogenesis of liver epithelial cells.The special case of hepatocytes: unique tissue architecture calls for a distinct mode of cell division.Role of Polarity Proteins in the Generation and Organization of Apical Surface Protrusions.JAM-A is both essential and inhibitory to development of hepatic polarity in WIF-B cells.Build them up and break them down: Tight junctions of cell lines expressing typical hepatocyte polarity with a varied repertoire of claudins.Rab11-FIP1 phosphorylation by MARK2 regulates polarity in MDCK cells.Epithelial Morphogenesis during Liver Development
P2860
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P2860
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
description
2004 nî lūn-bûn
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2004 թուականի Սեպտեմբերին հրատարակուած գիտական յօդուած
@hyw
2004 թվականի սեպտեմբերին հրատարակված գիտական հոդված
@hy
2004年の論文
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2004年論文
@yue
2004年論文
@zh-hant
2004年論文
@zh-hk
2004年論文
@zh-mo
2004年論文
@zh-tw
2004年论文
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name
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@ast
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@en
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@nl
type
label
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@ast
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@en
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@nl
prefLabel
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@ast
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@en
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@nl
P2093
P2860
P356
P1476
Par-1 promotes a hepatic mode of apical protein trafficking in MDCK cells
@en
P2093
Anne Müsch
David Cohen
Enrique Rodriguez-Boulan
P2860
P304
P356
10.1073/PNAS.0403684101
P407
P577
2004-09-21T00:00:00Z