African ancestry allelic variation at the MYH9 gene contributes to increased susceptibility to non-diabetic end-stage kidney disease in Hispanic Americans - Wikidata - wikimedia - TriplyDB

African ancestry allelic variation at the MYH9 gene contributes to increased susceptibility to non-diabetic end-stage kidney disease in Hispanic Americans

African ancestry allelic variation at the MYH9 gene contributes to increased susceptibility to non-diabetic end-stage kidney disease in Hispanic Americans

http://www.wikidata.org/entity/Q24602407

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APOL1 genetic variants in focal segmental glomerulosclerosis and HIV-associated nephropathy Missense mutations in the APOL1 gene are highly associated with end stage kidney disease risk previously attributed to the MYH9 gene Genetic causation of neointimal hyperplasia in hemodialysis vascular access dysfunction Developments in renal pharmacogenomics and applications in chronic kidney disease Knockdown of ApoL1 in Zebrafish Larvae Affects the Glomerular Filtration Barrier and the Expression of Nephrin Genetic background of patients from a university medical center in Manhattan: implications for personalized medicine MYH-9 Related Platelet Disorders: Strategies for Management and Diagnosis Analysis of coding variants identified from exome sequencing resources for association with diabetic and non-diabetic nephropathy in African Americans Admixture mapping of end stage kidney disease genetic susceptibility using estimated mutual information ancestry informative markers.Dense mapping of MYH9 localizes the strongest kidney disease associations to the region of introns 13 to 15.The population genetics of chronic kidney disease: insights from the MYH9-APOL1 locus.Characterization of genome-wide association-identified variants for atrial fibrillation in African Americans.APOL1 risk variants, race, and progression of chronic kidney disease A risk allele for focal segmental glomerulosclerosis in African Americans is located within a region containing APOL1 and MYH9 Polymorphisms in the non-muscle myosin heavy chain gene (MYH9) are associated with lower glomerular filtration rate in mixed ancestry diabetic subjects from South Africa.APOL1 null alleles from a rural village in India do not correlate with glomerulosclerosis.Recent developments in HIV-related kidney disease.Epistatic role of the MYH9/APOL1 region on familial hematuria genes.The MYH9/APOL1 region and chronic kidney disease in European-Americans.Racial and ethnic differences in kidney function decline among persons without chronic kidney disease MYH9 and APOL1 gene polymorphisms and the risk of CKD in patients with lupus nephritis from an admixture population.African origins and chronic kidney disease susceptibility in the human immunodeficiency virus era.APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy Population-based risk assessment of APOL1 on renal disease.Polymorphisms in MYH9 are associated with diabetic nephropathy in European Americans Role of MYH9 and APOL1 in African and non-African populations with lupus nephritis.Estimating ancestral proportions in a multi-ethnic US sample: implications for studies of admixed populations.Genetic variation in APOL1 and MYH9 genes is associated with chronic kidney disease among Nigerians.Stress and the kidney.Genetic contribution and associated pathophysiology in end-stage renal disease Background strain and the differential susceptibility of podocyte-specific deletion of Myh9 on murine models of experimental glomerulosclerosis and HIV nephropathy European genetic ancestry is associated with a decreased risk of lupus nephritis MYH9 and APOL1 are both associated with sickle cell disease nephropathy Pathogenesis and therapy of focal segmental glomerulosclerosis: an update.Podocyte-specific deletion of Myh9 encoding nonmuscle myosin heavy chain 2A predisposes mice to glomerulopathy.Mapping genetic determinants of kidney damage in rat models.Hispanic Americans living in the United States and their risk for obesity, diabetes and kidney disease: Genetic and environmental considerations.A null variant in the apolipoprotein L3 gene is associated with non-diabetic nephropathy.Genetics of Sickle Cell-Associated Cardiovascular Disease: An Expert Review with Lessons Learned in Africa.Admixture mapping: from paradigms of race and ethnicity to population history.

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African ancestry allelic variation at the MYH9 gene contributes to increased susceptibility to non-diabetic end-stage kidney disease in Hispanic Americans

http://www.wikidata.org/entity/Q24602407

description

2010 nî lūn-bûn

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2010 թուականի Մայիսին հրատարակուած գիտական յօդուած

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2010 թվականի մայիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

African ancestry allelic varia ...... disease in Hispanic Americans

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African ancestry allelic varia ...... disease in Hispanic Americans

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African ancestry allelic varia ...... disease in Hispanic Americans

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type

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African ancestry allelic varia ...... disease in Hispanic Americans

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African ancestry allelic varia ...... disease in Hispanic Americans

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African ancestry allelic varia ...... disease in Hispanic Americans

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African ancestry allelic varia ...... disease in Hispanic Americans

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African ancestry allelic varia ...... disease in Hispanic Americans

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African ancestry allelic varia ...... disease in Hispanic Americans

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Human Molecular Genetics

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African ancestry allelic varia ...... disease in Hispanic Americans

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Cheryl A Winkler

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George W Nelson

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Guennady Yudkovsky

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Saharon Rosset

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Sara Selig

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Shay Tzur

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Sivan Bercovici

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Walter G Wasser

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P2860

Positionally cloned gene for a novel glomerular protein--nephrin--is mutated in congenital nephrotic syndrome

Admixture dynamics in Hispanics: a shift in the nuclear genetic ancestry of a South American population isolate

MYH9 is a major-effect risk gene for focal segmental glomerulosclerosis

MYH9 is associated with nondiabetic end-stage renal disease in African Americans

A New Statistical Method for Haplotype Reconstruction from Population Data

Haploview: analysis and visualization of LD and haplotype maps

NPHS2, encoding the glomerular protein podocin, is mutated in autosomal recessive steroid-resistant nephrotic syndrome

Mutations in ACTN4, encoding alpha-actinin-4, cause familial focal segmental glomerulosclerosis

CD2-associated protein haploinsufficiency is linked to glomerular disease susceptibility

Trends in the epidemiology of focal segmental glomerulosclerosis.

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1816-27

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scholarly article

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1903744

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10.1093/HMG/DDQ040

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9

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19

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Jeffrey B. Kopp

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2010-05-01T00:00:00Z

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41419362

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20144966

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2850615

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