APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy - Wikidata - wikimedia - TriplyDB

APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

http://www.wikidata.org/entity/Q35595499

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The APOL1 genotype of African American kidney transplant recipients does not impact 5-year allograft survival APOL1 Kidney Disease Risk Variants: An Evolving Landscape Estimating kidney function in HIV-infected adults in Kenya: comparison to a direct measure of glomerular filtration rate by iohexol clearance Insights into kidney diseases from genome-wide association studies.Personalized Medicine: New Perspectives for the Diagnosis and the Treatment of Renal Diseases APOL1 null alleles from a rural village in India do not correlate with glomerulosclerosis.Defining nephrotic syndrome from an integrative genomics perspective.MYH9 and APOL1 gene polymorphisms and the risk of CKD in patients with lupus nephritis from an admixture population.Hyperglycemia enhances kidney cell injury in HIVAN through down-regulation of vitamin D receptors Clinical phenotype of APOL1 nephropathy in young relatives of patients with end-stage renal disease.Human apolipoprotein L1 (ApoL1) in cancer and chronic kidney disease.Genetic variants and cell-free hemoglobin processing in sickle cell nephropathy.APOL1 Risk Variants Are Strongly Associated with HIV-Associated Nephropathy in Black South Africans HIV-associated nephropathy patients with and without apolipoprotein L1 gene variants have similar clinical and pathological characteristics The association between chronic renal failure and renal cell carcinoma may differ between black and white Americans.Association of APOL1 Genotype with Renal Histology among Black HIV-Positive Patients Undergoing Kidney Biopsy.Genetic variation in APOL1 and MYH9 genes is associated with chronic kidney disease among Nigerians.Baseline kidney function as predictor of mortality and kidney disease progression in HIV-positive patients.Background strain and the differential susceptibility of podocyte-specific deletion of Myh9 on murine models of experimental glomerulosclerosis and HIV nephropathy APOL1 variants associate with increased risk of CKD among African Americans.Host APOL1 genotype is independently associated with proteinuria in HIV infection Impact of APOL1 polymorphism and IL-1β priming in the entry and persistence of HIV-1 in human podocytes.The kidney as a reservoir for HIV-1 after renal transplantation Plasma apolipoprotein L1 levels do not correlate with CKD.Hypertension in the teenager.Renoprotection and the Bardoxolone Methyl Story - Is This the Right Way Forward? A Novel View of Renoprotection in CKD Trials: A New Classification Scheme for Renoprotective Agents.Biomarkers for incident CKD: a new framework for interpreting the literature.Predictors of HIV-associated nephropathy.APOL1 nephropathy: from gene to mechanisms of kidney injury.Current State and Future Trends to Optimize the Care of African Americans with End-Stage Renal Disease.Roles of APOL1 G1 and G2 variants in sickle cell disease patients: kidney is the main target.Integrating APOL1 Gene Variants Into Renal Transplantation: Considerations Arising From the American Society of Transplantation Expert Conference.Diminished impact of ethnicity as a risk factor for chronic kidney disease in the current HIV treatment era.Role of race in kidney transplant outcomes in children with focal segmental glomerulosclerosis.A tripartite complex of suPAR, APOL1 risk variants and αvβ3 integrin on podocytes mediates chronic kidney disease.Impact of early versus deferred antiretroviral therapy on estimated glomerular filtration rate in HIV-positive individuals in the START trial.APOL1-associated glomerular disease among African-American children: a collaboration of the Chronic Kidney Disease in Children (CKiD) and Nephrotic Syndrome Study Network (NEPTUNE) cohorts.Unique X-linked familial FSGS with co-segregating heart block disorder is associated with a mutation in the NXF5 gene.APOL1-Mediated Cell Injury Involves Disruption of Conserved Trafficking Processes.HLA-DQA1 and APOL1 as Risk Loci for Childhood-Onset Steroid-Sensitive and Steroid-Resistant Nephrotic Syndrome.

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APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

http://www.wikidata.org/entity/Q35595499

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2011 nî lūn-bûn

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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2011年论文

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2011年论文

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name

APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

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APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

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type

label

APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

@ast

APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

@en

prefLabel

APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

@ast

APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

@en

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Journal of the American Society of Nephrology

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APOL1 variants increase risk for FSGS and HIVAN but not IgA nephropathy

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P2093

Ali G Gharavi

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Ami Patel

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Anand N Mhatre

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Anil K Lawani

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Bruce A Julian

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Christina M Wyatt

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David J Cohen

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Gerald B Appel

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Holly J Snyder

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Jan Novak

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P2860

African ancestry allelic variation at the MYH9 gene contributes to increased susceptibility to non-diabetic end-stage kidney disease in Hispanic Americans

Association of trypanolytic ApoL1 variants with kidney disease in African Americans

Missense mutations in the APOL1 gene are highly associated with end stage kidney disease risk previously attributed to the MYH9 gene

MYH9 is a major-effect risk gene for focal segmental glomerulosclerosis

MYH9 is associated with nondiabetic end-stage renal disease in African Americans

PLINK: a tool set for whole-genome association and population-based linkage analyses

Defects in cell adhesion and the visceral endoderm following ablation of nonmuscle myosin heavy chain II-A in mice

Admixture mapping of end stage kidney disease genetic susceptibility using estimated mutual information ancestry informative markers.

Dense mapping of MYH9 localizes the strongest kidney disease associations to the region of introns 13 to 15.

Prkdc participates in mitochondrial genome maintenance and prevents Adriamycin-induced nephropathy in mice.

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1991-1996

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10.1681/ASN.2011040434

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11

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22

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21997397

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3231781

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