Allotopic expression of mitochondrial-encoded genes in mammals: achieved goal, undemonstrated mechanism or impossible task? - Wikidata - wikimedia - TriplyDB

Allotopic expression of mitochondrial-encoded genes in mammals: achieved goal, undemonstrated mechanism or impossible task?

Allotopic expression of mitochondrial-encoded genes in mammals: achieved goal, undemonstrated mechanism or impossible task?

http://www.wikidata.org/entity/Q24603218

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Genetic manipulation for inherited neurodegenerative diseases: myth or reality?Evolutionary defined role of the mitochondrial DNA in fertility, disease and ageing Toward a therapy for mitochondrial disease ST proteins, a new family of plant tandem repeat proteins with a DUF2775 domain mainly found in Fabaceae and Asteraceae.Modeling of antigenomic therapy of mitochondrial diseases by mitochondrially addressed RNA targeting a pathogenic point mutation in mitochondrial DNA Mitochondrial disorders and the eye.Differential targeting of VDAC3 mRNA isoforms influences mitochondria morphology The Tn7 transposition regulator TnsC interacts with the transposase subunit TnsB and target selector TnsD Small mitochondrial-targeted RNAs modulate endogenous mitochondrial protein expression in vivo.Medical management of hereditary optic neuropathies.Therapies in inborn errors of oxidative metabolism.Mitochondrial optic neuropathies - disease mechanisms and therapeutic strategies.The optic nerve: a "mito-window" on mitochondrial neurodegeneration Automated hierarchical classification of protein domain subfamilies based on functionally-divergent residue signatures Transcriptome sequencing of rhizome tissue of Sinopodophyllum hexandrum at two temperatures.Mitochondrial genomes are retained by selective constraints on protein targeting.Leber's Hereditary Optic Neuropathy-Gene Therapy: From Benchtop to Bedside.Genetic relationship of Sardinella lemuru from lombok strait with fish rich in omega-3 fatty acid.Stable nuclear expression of ATP8 and ATP6 genes rescues a mtDNA Complex V null mutant The wooly mutation (wly) on mouse chromosome 11 is associated with a genetic defect in Fam83g Nuclear expression of mitochondrial ND4 leads to the protein assembling in complex I and prevents optic atrophy and visual loss.Mitochondrial Diseases Part III: Therapeutic interventions in mouse models of OXPHOS deficiencies.Hepatic Bmal1 Regulates Rhythmic Mitochondrial Dynamics and Promotes Metabolic Fitness.Staphylococcal pathogenicity island interference with helper phage reproduction is a paradigm of molecular parasitism Isolation and Expression Analysis of CYP9A11 and Cytochrome P450 Reductase Gene in the Beet Armyworm (Lepidoptera: Noctuidae).Polygenic molecular architecture underlying non-sexual cell aggregation in budding yeast.De novo assembly and characterization of transcriptome using Illumina paired-end sequencing and identification of CesA gene in ramie (Boehmeria nivea L. Gaud)Emerging therapies for mitochondrial disorders.Gene Therapy for Leber Hereditary Optic Neuropathy: Initial Results.From serendipity to mitochondria-targeted nanocarriers.Treatment of hereditary optic neuropathies.Sparse Markov chain-based semi-supervised multi-instance multi-label method for protein function prediction.Genome-wide analysis of VQ motif-containing proteins in Moso bamboo (Phyllostachys edulis).A Review of Mitochondrial Optic Neuropathies: From Inherited to Acquired Forms Therapeutic strategies for Leber's hereditary optic neuropathy: A current update.Leber hereditary optic neuropathy: bridging the translational gap.Transcriptome analysis of stem wood of Nothapodytes nimmoniana (Graham) Mabb. identifies genes associated with biosynthesis of camptothecin, an anti-carcinogenic molecule.Mitochondrial DNA Haplogroup A Decreases the Risk of Drug Addiction but Conversely Increases the Risk of HIV-1 Infection in Chinese Addicts.Expression and in silico characterization of Phenylalanine ammonium lyase against karnal bunt (Tilletia indica) in wheat (Triticum aestivum).Computational structural and functional analysis of hypothetical proteins of Staphylococcus aureus.

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Allotopic expression of mitochondrial-encoded genes in mammals: achieved goal, undemonstrated mechanism or impossible task?

http://www.wikidata.org/entity/Q24603218

description

2011 nî lūn-bûn

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2011 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2011 թվականի հունվարին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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Allotopic expression of mitoch ...... mechanism or impossible task?

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Allotopic expression of mitoch ...... mechanism or impossible task?

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Allotopic expression of mitoch ...... mechanism or impossible task?

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Allotopic expression of mitoch ...... mechanism or impossible task?

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Allotopic expression of mitoch ...... mechanism or impossible task?

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Allotopic expression of mitoch ...... mechanism or impossible task?

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Nucleic Acids Research

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Allotopic expression of mitoch ...... mechanism or impossible task?

@en

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Acisclo Pérez-Martos

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Ester Perales-Clemente

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Jose Antonio Enríquez

http://www.w3.org/2001/XMLSchema#string

Patricio Fernández-Silva

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Rebeca Acín-Pérez

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P2860

Limitations of allotopic expression of mitochondrial genes in mammalian cells

Blue native PAGE

Pattern of organization of human mitochondrial pseudogenes in the nuclear genome

Unusual location of a mitochondrial gene. Subunit III of cytochrome C oxidase is encoded in the nucleus of Chlamydomonad algae.

Assembly of functional proton-translocating ATPase complex in yeast mitochondria with cytoplasmically synthesized subunit 8, a polypeptide normally encoded within the organelle.

Structure and evolution of organelle genomes.

Why have organelles retained genomes?

Rescue of a deficiency in ATP synthesis by transfer of MTATP6, a mitochondrial DNA-encoded gene, to the nucleus.

An algal nucleus-encoded subunit of mitochondrial ATP synthase rescues a defect in the analogous human mitochondrial-encoded subunit.

Control of gene expression by redox potential and the requirement for chloroplast and mitochondrial genomes.

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10.1093/NAR/GKQ769

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1

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39

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20823090

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3017613

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