Myopathy caused by mammalian target of rapamycin complex 1 (mTORC1) inactivation is not reversed by restoring mitochondrial function
about
Dynamic modelling of pathways to cellular senescence reveals strategies for targeted interventionsAgonism of the 5-hydroxytryptamine 1F receptor promotes mitochondrial biogenesis and recovery from acute kidney injuryMitochondria are required for pro-ageing features of the senescent phenotype.Regulation of mTORC1 and its impact on gene expression at a glance.IL-6 regulation on skeletal muscle mitochondrial remodeling during cancer cachexia in the ApcMin/+ mouse.Alterations to mTORC1 signaling in the skeletal muscle differentially affect whole-body metabolismDifferential response of skeletal muscles to mTORC1 signaling during atrophy and hypertrophy.Regulation of fatty acid metabolism by mTOR in adult murine hearts occurs independently of changes in PGC-1αRapamycin doses sufficient to extend lifespan do not compromise muscle mitochondrial content or endurance.The transcriptional coactivator PGC-1α is dispensable for chronic overload-induced skeletal muscle hypertrophy and metabolic remodeling.Autophagy in skeletal muscle homeostasis and in muscular dystrophiesContractile activity-induced mitochondrial biogenesis and mTORC1.Loss of mTOR signaling affects cone function, cone structure and expression of cone specific proteins without affecting cone survival.mTORC1 is Required for Brown Adipose Tissue Recruitment and Metabolic Adaptation to Cold.A Skeletal Muscle Model of Infantile-onset Pompe Disease with Patient-specific iPS Cells
P2860
Q27321555-7CC41D11-77BC-474C-B76F-E48DA121045BQ33943971-799A5DC2-E46A-4C5F-847D-0CA5715553E9Q34046100-47406014-E04A-4EA9-8B1A-3A5F6793E2F4Q34342717-D77E9989-9A80-4E43-8ED0-6A4F26D2D77FQ36200582-91D124AA-060C-4010-B37E-5B56D1A54242Q36709204-DDC9AB75-0DA2-4349-9CD4-CFC224FFD0A7Q36755594-33900E8F-F3BF-4FEC-899C-21E448885160Q37054943-003972CC-3724-4D01-ADDC-44C027353906Q37152784-2E8B6CF9-816C-4864-AF79-93907A095F95Q37395056-525BD7AE-CC36-4333-AB87-0B66B98C8B3DQ38202870-2BDE91AB-3DC6-4FAF-A78A-C77ED1C4A7EEQ39330883-6BDB0471-47B4-4CEE-9787-49F4BF4455DAQ41051208-D9DA9896-2428-48B3-AEC0-E2F061E0C8C1Q42364391-BA42C749-6DE5-4584-98BF-E713ABE61A41Q42677230-D92F3095-2C2E-4667-892B-1FAAFF63442F
P2860
Myopathy caused by mammalian target of rapamycin complex 1 (mTORC1) inactivation is not reversed by restoring mitochondrial function
description
2011 nî lūn-bûn
@nan
2011 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2011 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2011年の論文
@ja
2011年論文
@yue
2011年論文
@zh-hant
2011年論文
@zh-hk
2011年論文
@zh-mo
2011年論文
@zh-tw
2011年论文
@wuu
name
Myopathy caused by mammalian t ...... storing mitochondrial function
@ast
Myopathy caused by mammalian t ...... storing mitochondrial function
@en
Myopathy caused by mammalian t ...... storing mitochondrial function
@nl
type
label
Myopathy caused by mammalian t ...... storing mitochondrial function
@ast
Myopathy caused by mammalian t ...... storing mitochondrial function
@en
Myopathy caused by mammalian t ...... storing mitochondrial function
@nl
prefLabel
Myopathy caused by mammalian t ...... storing mitochondrial function
@ast
Myopathy caused by mammalian t ...... storing mitochondrial function
@en
Myopathy caused by mammalian t ...... storing mitochondrial function
@nl
P2093
P2860
P50
P3181
P356
P1476
Myopathy caused by mammalian t ...... storing mitochondrial function
@en
P2093
Agnès Conjard-Duplany
Francesco Zorzato
Klaas Romanino
Laetitia Mazelin
Laurent Schaeffer
Markus A Rüegg
Pere Puigserver
Verena Albert
P2860
P304
20808-20813
P3181
P356
10.1073/PNAS.1111448109
P407
P577
2011-12-05T00:00:00Z