Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward - Wikidata - wikimedia - TriplyDB

Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

http://www.wikidata.org/entity/Q24619737

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Dopamine signaling in reward-related behaviors The dopamine d1-d2 receptor heteromer in striatal medium spiny neurons: evidence for a third distinct neuronal pathway in Basal Ganglia Transcriptional and epigenetic mechanisms of addiction Addiction-related gene regulation: risks of exposure to cognitive enhancers vs. other psychostimulants Dopamine in motivational control: rewarding, aversive, and alerting Rapid regulation of depression-related behaviours by control of midbrain dopamine neurons Cocaine alters BDNF expression and neuronal migration in the embryonic mouse forebrain Comprehensive qPCR profiling of gene expression in single neuronal cells Autism Spectrum Disorders and Drug Addiction: Common Pathways, Common Molecules, Distinct Disorders?Dopaminergic reward sensitivity can promote adolescent health: A new perspective on the mechanism of ventral striatum activation Optogenetics: 10 years of microbial opsins in neuroscience.Making Sense of Optogenetics Obesity and addiction: neurobiological overlaps Virogenetic and optogenetic mechanisms to define potential therapeutic targets in psychiatric disorders Establishing causality for dopamine in neural function and behavior with optogenetics Insights into cortical oscillations arising from optogenetic studies Structural and synaptic plasticity in stress-related disorders Optogenetics in psychiatric diseases Molecular, cellular, and structural mechanisms of cocaine addiction: a key role for microRNAs Cardiac optogenetics Lateral hypothalamus, nucleus accumbens, and ventral pallidum roles in eating and hunger: interactions between homeostatic and reward circuitry Individual differences in response to positive and negative stimuli: endocannabinoid-based insight on approach and avoidance behaviors Neural integration of reward, arousal, and feeding: recruitment of VTA, lateral hypothalamus, and ventral striatal neurons Optogenetic strategies to investigate neural circuitry engaged by stress.Investigating striatal function through cell-type-specific manipulations Circuit dynamics of adaptive and maladaptive behaviour Genetically targeted magnetic control of the nervous system Separate circuitries encode the hedonic and nutritional values of sugar Activation of D2 dopamine receptor-expressing neurons in the nucleus accumbens increases motivation.BDNF rescues BAF53b-dependent synaptic plasticity and cocaine-associated memory in the nucleus accumbens Pain inhibition by optogenetic activation of specific anterior cingulate cortical neurons Cocaine inhibits dopamine D2 receptor signaling via sigma-1-D2 receptor heteromers Aversive stimuli drive drug seeking in a state of low dopamine tone Direct regulation of diurnal Drd3 expression and cocaine reward by NPAS2.Understanding the Functional Plasticity in Neural Networks of the Basal Ganglia in Cocaine Use Disorder: A Role for Allosteric Receptor-Receptor Interactions in A2A-D2 Heteroreceptor Complexes β-catenin mediates stress resilience through Dicer1/microRNA regulation The ins and outs of the striatum: role in drug addiction Glial modulators as potential treatments of psychostimulant abuse Mechanisms of psychostimulant-induced structural plasticity PDE10A inhibitors stimulate or suppress motor behavior dependent on the relative activation state of the direct and indirect striatal output pathways

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Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

http://www.wikidata.org/entity/Q24619737

description

2010 nî lūn-bûn

@nan

2010 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հոտեմբերին հրատարակված գիտական հոդված

@hy

2010年の論文

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2010年学术文章

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2010年学术文章

@zh-cn

2010年学术文章

@zh-hans

2010年学术文章

@zh-my

2010年学术文章

@zh-sg

2010年學術文章

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name

Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

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Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

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Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

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label

Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

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Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

@en

Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

@nl

prefLabel

Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

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Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

@en

Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

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SCIENCE.1188472

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Cell type-specific loss of BDNF signaling mimics optogenetic control of cocaine reward

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Allyson K Friedman

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Diane Damez-Werno

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Dipesh Chaudhury

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Ezekiell Mouzon

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HaoSheng Sun

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Herbert E Covington

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Ja Wook Koo

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Ming-Hu Han

http://www.w3.org/2001/XMLSchema#string

Murtaza Mogri

http://www.w3.org/2001/XMLSchema#string

Pamela J Kennedy

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NEURAL MECHANISMS OF ADDICTION: The Role of Reward-Related Learning and Memory

Targeted disruption of cocaine-activated nucleus accumbens neurons prevents context-specific sensitization

NMDA-mediated activation of the tyrosine phosphatase STEP regulates the duration of ERK signaling

Delta FosB mediates epigenetic desensitization of the c-fos gene after chronic amphetamine exposure

Expression of mutant NMDA receptors in dopamine D1 receptor-containing cells prevents cocaine sensitization and decreases cocaine preference

Parallel Organization of Functionally Segregated Circuits Linking Basal Ganglia and Cortex

The functional anatomy of basal ganglia disorders

A gene expression atlas of the central nervous system based on bacterial artificial chromosomes

Making new connections: role of ERK/MAP kinase signaling in neuronal plasticity.

Expression of the transcription factor deltaFosB in the brain controls sensitivity to cocaine.

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10.1126/SCIENCE.1188472

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6002

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330

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Eric J. Nestler

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2010-10-15T00:00:00Z

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47429341

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20947769

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