hMSH4-hMSH5 adenosine nucleotide processing and interactions with homologous recombination machinery
about
DNA mismatch repair: molecular mechanism, cancer, and ageingPostreplicative mismatch repair.Variation in crossover frequencies perturb crossover assurance without affecting meiotic chromosome segregation in Saccharomyces cerevisiaeThe Saccharomyces cerevisiae Mlh1-Mlh3 heterodimer is an endonuclease that preferentially binds to Holliday junctions.FancJ (Brip1) loss-of-function allele results in spermatogonial cell depletion during embryogenesis and altered processing of crossover sites during meiotic prophase I in miceDNA repair mechanisms and the bypass of DNA damage in Saccharomyces cerevisiaeReplication protein A (AtRPA1a) is required for class I crossover formation but is dispensable for meiotic DNA break repair.Genetic analysis of baker's yeast Msh4-Msh5 reveals a threshold crossover level for meiotic viability.Separable Crossover-Promoting and Crossover-Constraining Aspects of Zip1 Activity during Budding Yeast MeiosisThe role of OsMSH4 in male and female gamete development in rice meiosis.Roles for mismatch repair family proteins in promoting meiotic crossing over.MutS Homologues hMSH4 and hMSH5: Genetic Variations, Functions, and Implications in Human DiseasesReassessment of the role of Mut S homolog 5 in Ig class switch recombination shows lack of involvement in cis- and trans-switching.Structural insights into Saccharomyces cerevisiae Msh4-Msh5 complex function using homology modelingVBP1 facilitates proteasome and autophagy-mediated degradation of MutS homologue hMSH4.Modulation of homology-directed repair in T98G glioblastoma cells due to interactions between wildtype p53, Rad51 and HCMV IE1-72.A strand invasion 3' polymerization intermediate of mammalian homologous recombination.Absence of SUN1 and SUN2 proteins in Arabidopsis thaliana leads to a delay in meiotic progression and defects in synapsis and recombination.Archaeal MutS5 tightly binds to Holliday junction similarly to eukaryotic MutSγ.Formation of meiotic holliday junctionA homozygous donor splice-site mutation in the meiotic gene MSH4 causes primary ovarian insufficiency.The polymorphic hMSH5 C85T allele augments radiotherapy-induced spermatogenic impairment.
P2860
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P2860
hMSH4-hMSH5 adenosine nucleotide processing and interactions with homologous recombination machinery
description
2008 nî lūn-bûn
@nan
2008 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի հունվարին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@ast
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@en
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@nl
type
label
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@ast
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@en
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@nl
prefLabel
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@ast
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@en
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@nl
P2093
P2860
P356
P1476
hMSH4-hMSH5 adenosine nucleoti ...... logous recombination machinery
@en
P2093
Christoph Schmutte
Kang-Sup Shim
Richard Fishel
Samir Acharya
Timothy Snowden
P2860
P304
P356
10.1074/JBC.M704060200
P407
P577
2008-01-04T00:00:00Z