Plasma complement components and activation fragments: associations with age-related macular degeneration genotypes and phenotypes - Wikidata - wikimedia - TriplyDB

Plasma complement components and activation fragments: associations with age-related macular degeneration genotypes and phenotypes

Plasma complement components and activation fragments: associations with age-related macular degeneration genotypes and phenotypes

http://www.wikidata.org/entity/Q24644120

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Relationship between Oxidative Stress, Circadian Rhythms, and AMD Inflammation and its role in age-related macular degeneration The complotype: dictating risk for inflammation and infection Complement system activation and endothelial dysfunction in patients with age-related macular degeneration (AMD): possible relationship between AMD and atherosclerosis Animal models of age related macular degeneration Genetic and environmental underpinnings to age-related ocular diseases Inhibiting Alternative Pathway Complement Activation by Targeting the Factor D Exosite Fit for the Eye: Aptamers in Ocular Disorders Genetic influences on plasma CFH and CFHR1 concentrations and their role in susceptibility to age-related macular degeneration The role of vascular endothelial growth factor-induced activation of NADPH oxidase in choroidal endothelial cells and choroidal neovascularization Inherited mitochondrial DNA variants can affect complement, inflammation and apoptosis pathways: insights into mitochondrial-nuclear interactions.The alternative pathway is required, but not alone sufficient, for retinal pathology in mouse laser-induced choroidal neovascularization.Risk models for progression to advanced age-related macular degeneration using demographic, environmental, genetic, and ocular factors.R102G polymorphism of the C3 gene associated with exudative age-related macular degeneration in a French population Therapeutic effect of oral bisphosphonates on choroidal neovascularization in the human eye.A common complement C3 variant is associated with protection against wet age-related macular degeneration in a Japanese population Fragment Bb: evidence for activation of the alternative pathway of the complement system in pregnant women with acute pyelonephritis.Complement system in pathogenesis of AMD: dual player in degeneration and protection of retinal tissue.Meningococcal disease and the complement system.Age-related macular degeneration: genetic and environmental factors of disease Zinc supplementation inhibits complement activation in age-related macular degeneration.Emerging roles for nuclear receptors in the pathogenesis of age-related macular degeneration.The significance of the complement system for the pathogenesis of age-related macular degeneration - current evidence and translation into clinical application Alterations of choroidal blood flow regulation in young healthy subjects with complement factor H polymorphism.The role of macrophage class a scavenger receptors in a laser-induced murine choroidal neovascularization model.The effect of lutein supplementation on blood plasma levels of complement factor D, C5a and C3d.Common polymorphisms in C3, factor B, and factor H collaborate to determine systemic complement activity and disease risk Impact of the common genetic associations of age-related macular degeneration upon systemic complement component C3d levels Interleukin-17 retinotoxicity is prevented by gene transfer of a soluble interleukin-17 receptor acting as a cytokine blocker: implications for age-related macular degeneration.Complement component C5a promotes expression of IL-22 and IL-17 from human T cells and its implication in age-related macular degeneration.CFH Y402H polymorphism is associated with elevated vitreal GM-CSF and choroidal macrophages in the postmortem human eye.Complement factor D in age-related macular degeneration Intravitreal inhibition of complement C5a reduces choroidal neovascularization in mice.Systemic and Ocular Long Pentraxin 3 in Patients with Age-Related Macular Degeneration.Rare genetic variants in the CFI gene are associated with advanced age-related macular degeneration and commonly result in reduced serum factor I levels Age-Related Macular Degeneration: A Disease of Systemic or Local Complement Dysregulation?STAT3 Activation in Circulating Monocytes Contributes to Neovascular Age-Related Macular Degeneration.Analysis of Risk Alleles and Complement Activation Levels in Familial and Non-Familial Age-Related Macular Degeneration Genetic insights into age-related macular degeneration: controversies addressing risk, causality, and therapeutics.Differential Expression of Complement Markers in Normal and AMD Transmitochondrial Cybrids

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Plasma complement components and activation fragments: associations with age-related macular degeneration genotypes and phenotypes

http://www.wikidata.org/entity/Q24644120

description

2009 nî lūn-bûn

@nan

2009 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի դեկտեմբերին հրատարակված գիտական հոդված

@hy

2009年の論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年論文

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2009年论文

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name

Plasma complement components a ...... ation genotypes and phenotypes

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Plasma complement components a ...... ation genotypes and phenotypes

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Plasma complement components a ...... ation genotypes and phenotypes

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Plasma complement components a ...... ation genotypes and phenotypes

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Plasma complement components a ...... ation genotypes and phenotypes

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Plasma complement components a ...... ation genotypes and phenotypes

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Plasma complement components a ...... ation genotypes and phenotypes

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Plasma complement components a ...... ation genotypes and phenotypes

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Plasma complement components a ...... ation genotypes and phenotypes

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Investigative Ophthalmology & Visual Science

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Plasma complement components a ...... ation genotypes and phenotypes

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P2093

Bernard Rosner

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Johanna M Seddon

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John P Atkinson

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M Elizabeth Hartnett

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Patricia C Giclas

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Robyn Reynolds

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P2860

Human adipsin is identical to complement factor D and is expressed at high levels in adipose tissue

Drusen complement components C3a and C5a promote choroidal neovascularization

A common haplotype in the complement regulatory gene factor H (HF1/CFH) predisposes individuals to age-related macular degeneration

Susceptibility genes for age-related maculopathy on chromosome 10q26.

Complement factor H polymorphism in age-related macular degeneration

Variation near complement factor I is associated with risk of advanced AMD

CFH haplotypes without the Y402H coding variant show strong association with susceptibility to age-related macular degeneration

A variant of mitochondrial protein LOC387715/ARMS2, not HTRA1, is strongly associated with age-related macular degeneration

An integrated hypothesis that considers drusen as biomarkers of immune-mediated processes at the RPE-Bruch's membrane interface in aging and age-related macular degeneration

Complement factor H variant increases the risk of age-related macular degeneration

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5818-27

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scholarly article

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1056604

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10.1167/IOVS.09-3928

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12

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50

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2009-12-01T00:00:00Z

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26723261

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19661236

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2826794

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