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Structural basis for the heterodimeric interaction between the acute leukaemia-associated transcription factors AML1 and CBFbetaALY is a common coactivator of RUNX1 and c-Myb on the type B leukemogenic virus enhancerTranscriptional control in the segmentation gene network of DrosophilaRunx1 binds as a dimeric complex to overlapping Runx1 sites within a palindromic element in the human GM-CSF enhancerCloning and characterization of subunits of the T-cell receptor and murine leukemia virus enhancer core-binding factorAML1 is expressed in skeletal muscle and is regulated by innervationIntersection of FOXO- and RUNX1-mediated gene expression programs in single breast epithelial cells during morphogenesis and tumor progression.t(8;21)(q22;q22) Fusion proteins preferentially bind to duplicated AML1/RUNX1 DNA-binding sequences to differentially regulate gene expression.Core-binding factor influences the disease specificity of Moloney murine leukemia virusType B leukemogenic virus has a T-cell-specific enhancer that binds AML-1.Conservation and function of the transcriptional regulatory protein RuntRunx2 is a common target of transforming growth factor beta1 and bone morphogenetic protein 2, and cooperation between Runx2 and Smad5 induces osteoblast-specific gene expression in the pluripotent mesenchymal precursor cell line C2C12.Runx transcription factors repress human and murine c-Myc expression in a DNA-binding and C-terminally dependent manner.SERPINB13 is a novel RUNX1 target geneAn ancient Pygo-dependent Wnt enhanceosome integrated by Chip/LDB-SSDP.Cell type dependent regulation of multidrug resistance-1 gene expression by AML1-ETO.A system of repressor gradients spatially organizes the boundaries of Bicoid-dependent target genes.c-Myb and core-binding factor/PEBP2 display functional synergy but bind independently to adjacent sites in the T-cell receptor delta enhancer.The t(12;21) translocation converts AML-1B from an activator to a repressor of transcription.Cooperative binding of Ets-1 and core binding factor to DNAThe t(8;21) fusion protein interferes with AML-1B-dependent transcriptional activation.The Runt domain of AML1 (RUNX1) binds a sequence-conserved RNA motif that mimics a DNA element.A quantitative proteomics approach identifies ETV6 and IKZF1 as new regulators of an ERG-driven transcriptional network.Post-translational Regulation of Runx2 in Bone and Cartilage.Mutation of all Runx (AML1/core) sites in the enhancer of T-lymphomagenic SL3-3 murine leukemia virus unmasks a significant potential for myeloid leukemia induction and favors enhancer evolution toward induction of other disease patterns.An AML-1 consensus sequence binds an osteoblast-specific complex and transcriptionally activates the osteocalcin gene.Transcriptional activity of core binding factor-alpha (AML1) and beta subunits on murine leukemia virus enhancer cores.Developmental cis-regulatory analysis of the cyclin D gene in the sea urchin Strongylocentrotus purpuratus.Selection of reversions and suppressors of a mutation in the CBF binding site of a lymphomagenic retrovirusSuppressor mutations within the core binding factor (CBF/AML1) binding site of a T-cell lymphomagenic retrovirus.ETS-core binding factor: a common composite motif in antigen receptor gene enhancers.Long terminal repeat enhancer core sequences in proviruses adjacent to c-myc in T-cell lymphomas induced by a murine retrovirus.Transcriptional activation of a retrovirus enhancer by CBF (AML1) requires a second factor: evidence for cooperativity with c-Myb.The potent enhancer activity of the polycythemic strain of spleen focus-forming virus in hematopoietic cells is governed by a binding site for Sp1 in the upstream control region and by a unique enhancer core motif, creating an exclusive target for PCompetitive binding of viral E2 protein and mammalian core-binding factor to transcriptional control sequences of human papillomavirus type 8 and bovine papillomavirus type 1.RUNX1 and RUNX2 upregulate Galectin-3 expression in human pituitary tumors.A bacterial one-hybrid system for determining the DNA-binding specificity of transcription factors.CBFβ stabilizes HIV Vif to counteract APOBEC3 at the expense of RUNX1 target gene expression.Heterodimerization of AML1/ETO with CBFβ is required for leukemogenesis but not for myeloproliferation.Precocious Phenotypic Transcription-Factor Expression During Early Development.
P2860
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P2860
description
1993 nî lūn-bûn
@nan
1993 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
1993 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
name
Sequence specificity of the core-binding factor
@ast
Sequence specificity of the core-binding factor
@en
Sequence specificity of the core-binding factor
@nl
type
label
Sequence specificity of the core-binding factor
@ast
Sequence specificity of the core-binding factor
@en
Sequence specificity of the core-binding factor
@nl
prefLabel
Sequence specificity of the core-binding factor
@ast
Sequence specificity of the core-binding factor
@en
Sequence specificity of the core-binding factor
@nl
P2093
P2860
P3181
P1433
P1476
Sequence specificity of the core-binding factor
@en
P2093
P2860
P304
P3181
P407
P577
1993-04-01T00:00:00Z