Long terminal repeat enhancer core sequences in proviruses adjacent to c-myc in T-cell lymphomas induced by a murine retrovirus.
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Structures of endogenous nonecotropic murine leukemia virus (MLV) long terminal repeats in wild mice: implication for evolution of MLVsSequence-specific and/or stereospecific constraints of the U3 enhancer elements of MCF 247-W are important for pathogenicity.Tandemization of a subregion of the enhancer sequences from SRS 19-6 murine leukemia virus associated with T-lymphoid but not other leukemias.Sint1, a common integration site in SL3-3-induced T-cell lymphomas, harbors a putative proto-oncogene with homology to the septin gene familyFeline leukemia virus long terminal repeat activates collagenase IV gene expression through AP-1Core-binding factor influences the disease specificity of Moloney murine leukemia virusIncreased induction of osteopetrosis, but unaltered lymphomagenicity, by murine leukemia virus SL3-3 after mutation of a nuclear factor 1 site in the enhancerNovel principles of gamma-retroviral insertional transcription activation in murine leukemia virus-induced end-stage tumors.Ikaros, a lymphoid-cell-specific transcription factor, contributes to the leukemogenic phenotype of a mink cell focus-inducing murine leukemia virusDisruption of hematopoiesis and thymopoiesis in the early premalignant stages of infection with SL3-3 murine leukemia virus.Duplication of U3 sequences in the long terminal repeat of mink cell focus-inducing viruses generates redundancies of transcription factor binding sites important for the induction of thymomasSelection for c-myc integration sites in polyclonal T-cell lymphomas.Stability of AML1 (core) site enhancer mutations in T lymphomas induced by attenuated SL3-3 murine leukemia virus mutants.Increased lymphomagenicity and restored disease specificity of AML1 site (core) mutant SL3-3 murine leukemia virus by a second-site enhancer variant evolved in vivo.The feline leukemia virus long terminal repeat contains a potent genetic determinant of T-cell lymphomagenicityAn SL3-3 murine leukemia virus enhancer variant more pathogenic than the wild type obtained by assisted molecular evolution in vivoControl of pathogenicity and disease specificity of a T-lymphomagenic gammaretrovirus by E-box motifs but not by an overlapping glucocorticoid response elementLeukemia virus long terminal repeat activates NFkappaB pathway by a TLR3-dependent mechanism.Mutation of all Runx (AML1/core) sites in the enhancer of T-lymphomagenic SL3-3 murine leukemia virus unmasks a significant potential for myeloid leukemia induction and favors enhancer evolution toward induction of other disease patterns.Analysis of wild-type and mutant SL3-3 murine leukemia virus insertions in the c-myc promoter during lymphomagenesis reveals target site hot spots, virus-dependent patterns, and frequent error-prone gap repair.Substitution of feline leukemia virus long terminal repeat sequences into murine leukemia virus alters the pattern of insertional activation and identifies new common insertion sitesTranscriptional activity of core binding factor-alpha (AML1) and beta subunits on murine leukemia virus enhancer cores.Selection of reversions and suppressors of a mutation in the CBF binding site of a lymphomagenic retrovirusSuppressor mutations within the core binding factor (CBF/AML1) binding site of a T-cell lymphomagenic retrovirus.CBF, Myb, and Ets binding sites are important for activity of the core I element of the murine retrovirus SL3-3 in T lymphocytes.The secondary structure of the R region of a murine leukemia virus is important for stimulation of long terminal repeat-driven gene expression.B-Cell lymphoma induction by akv murine leukemia viruses harboring one or both copies of the tandem repeat in the U3 enhancer.Woodchuck hepatitis virus enhancer I and enhancer II are both involved in N-myc2 activation in woodchuck liver tumors.Tumorigenic potential of a recombinant retrovirus containing sequences from Moloney murine leukemia virus and feline leukemia virusThe exogenous form of Jaagsiekte retrovirus is specifically associated with a contagious lung cancer of sheep.Sequence tags of provirus integration sites in DNAs of tumors induced by the murine retrovirus SL3-3.Transcriptional activation of a retrovirus enhancer by CBF (AML1) requires a second factor: evidence for cooperativity with c-Myb.Mapping of a major osteomagenic determinant of murine leukemia virus RFB-14 to non-long terminal repeat sequences.Second-site proviral enhancer alterations in lymphomas induced by enhancer mutants of SL3-3 murine leukemia virus: negative effect of nuclear factor 1 binding site.Importance of a c-Myb binding site for lymphomagenesis by the retrovirus SL3-3.Structure and function of the long terminal repeats of feline leukemia viruses derived from naturally occurring acute myeloid leukemias in cats.Recombinant Origins of Pathogenic and Nonpathogenic Mouse Gammaretroviruses with Polytropic Host Range.Xenotropic Mouse Gammaretroviruses Isolated from Pre-Leukemic Tissues Include a Recombinant
P2860
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P2860
Long terminal repeat enhancer core sequences in proviruses adjacent to c-myc in T-cell lymphomas induced by a murine retrovirus.
description
1995 nî lūn-bûn
@nan
1995年の論文
@ja
1995年論文
@yue
1995年論文
@zh-hant
1995年論文
@zh-hk
1995年論文
@zh-mo
1995年論文
@zh-tw
1995年论文
@wuu
1995年论文
@zh
1995年论文
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name
Long terminal repeat enhancer ...... nduced by a murine retrovirus.
@en
Long terminal repeat enhancer ...... nduced by a murine retrovirus.
@nl
type
label
Long terminal repeat enhancer ...... nduced by a murine retrovirus.
@en
Long terminal repeat enhancer ...... nduced by a murine retrovirus.
@nl
prefLabel
Long terminal repeat enhancer ...... nduced by a murine retrovirus.
@en
Long terminal repeat enhancer ...... nduced by a murine retrovirus.
@nl
P2093
P2860
P1433
P1476
Long terminal repeat enhancer ...... induced by a murine retrovirus
@en
P2093
P2860
P304
P407
P577
1995-01-01T00:00:00Z