Tumours with PI3K activation are resistant to dietary restriction - Wikidata - wikimedia - TriplyDB

Tumours with PI3K activation are resistant to dietary restriction

Tumours with PI3K activation are resistant to dietary restriction

http://www.wikidata.org/entity/Q24650286

about

Cancer as a metabolic disease Altered dietary methionine differentially impacts glutathione and methionine metabolism in long-living growth hormone-deficient Ames dwarf and wild-type mice Lithocholic bile acid selectively kills neuroblastoma cells, while sparing normal neuronal cells Links between metabolism and cancer Dysregulated metabolism contributes to oncogenesis Why does obesity promote cancer? Epidemiology, biology, and open questions Cancer metabolic reprogramming: importance, main features, and potentials for precise targeted anti-cancer therapies Metabolic homeostasis as the cornerstone of aging The role of obesity in gastrointestinal cancer: evidence and opinion Benefits of short-term dietary restriction in mammals Physiological control of germline development Deconvoluting the obesity and breast cancer link: secretome, soil and seed interactions Cellular and molecular mechanisms of metformin: an overview Energy balance and metabolism after cancer treatment Diabetes and cancer: a consensus report.Dietary and pharmacological modification of the insulin/IGF-1 system: exploiting the full repertoire against cancer The interplay between obesity and cancer: a fly view The eEF2 kinase confers resistance to nutrient deprivation by blocking translation elongation Plants vs. cancer: a review on natural phytochemicals in preventing and treating cancers and their druggability Epithelial cancers in the post-genomic era: should we reconsider our lifestyle?Resveratrol inhibits protein translation in hepatic cells Novel protein kinase signaling systems regulating lifespan identified by small molecule library screening using Drosophila Activation of β-catenin by oncogenic PIK3CA and EGFR promotes resistance to glucose deprivation by inducing a strong antioxidant response Small is beautiful: insulin-like growth factors and their role in growth, development, and cancer The dietary isothiocyanate sulforaphane modulates gene expression and alternative gene splicing in a PTEN null preclinical murine model of prostate cancer.Starved epithelial cells uptake extracellular matrix for survival.Dynamic switch of negative feedback regulation in Drosophila Akt-TOR signaling.Insulin signaling promotes germline proliferation in C. elegans.Calorie restriction and cancer prevention: metabolic and molecular mechanisms.ERGO: a pilot study of ketogenic diet in recurrent glioblastoma.Monoacylglycerol lipase regulates a fatty acid network that promotes cancer pathogenesis.Akt phosphorylation and regulation of transketolase is a nodal point for amino acid control of purine synthesis Fasting and differential chemotherapy protection in patients Fasting cycles retard growth of tumors and sensitize a range of cancer cell types to chemotherapy.PIK3CA hotspot mutation scanning by a novel and highly sensitive high-resolution small amplicon melting analysis method.Molecular genetics of prostate cancer: new prospects for old challenges NRF2, cancer and calorie restriction FOXO1: a potential target for human diseases Fasting vs dietary restriction in cellular protection and cancer treatment: from model organisms to patients.Metformin and other biguanides in oncology: advancing the research agenda.

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P2860

Tumours with PI3K activation are resistant to dietary restriction

http://www.wikidata.org/entity/Q24650286

description

2009 nî lūn-bûn

@nan

2009 թուականի Ապրիլին հրատարակուած գիտական յօդուած

@hyw

2009 թվականի ապրիլին հրատարակված գիտական հոդված

@hy

2009年の論文

@ja

2009年論文

@yue

2009年論文

@zh-hant

2009年論文

@zh-hk

2009年論文

@zh-mo

2009年論文

@zh-tw

2009年论文

@wuu

name

Tumours with PI3K activation are resistant to dietary restriction

@ast

Tumours with PI3K activation are resistant to dietary restriction

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Tumours with PI3K activation are resistant to dietary restriction

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type

label

Tumours with PI3K activation are resistant to dietary restriction

@ast

Tumours with PI3K activation are resistant to dietary restriction

@en

Tumours with PI3K activation are resistant to dietary restriction

@nl

prefLabel

Tumours with PI3K activation are resistant to dietary restriction

@ast

Tumours with PI3K activation are resistant to dietary restriction

@en

Tumours with PI3K activation are resistant to dietary restriction

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Tumours with PI3K activation are resistant to dietary restriction

@en

P2093

David M Sabatini

http://www.w3.org/2001/XMLSchema#string

Nada Y Kalaany

http://www.w3.org/2001/XMLSchema#string

P2860

mTOR interacts with raptor to form a nutrient-sensitive complex that signals to the cell growth machinery

Binding of ras to phosphoinositide 3-kinase p110alpha is required for ras-driven tumorigenesis in mice

AKT/PKB signaling: navigating downstream

CellProfiler: image analysis software for identifying and quantifying cell phenotypes

Phosphatidylinositol 3-kinase mutations identified in human cancer are oncogenic

High frequency of mutations of the PIK3CA gene in human cancers

PIK3CA mutations correlate with hormone receptors, node metastasis, and ERBB2, and are mutually exclusive with PTEN loss in human breast carcinoma

CellProfiler: free, versatile software for automated biological image analysis

Prostate-specific deletion of the murine Pten tumor suppressor gene leads to metastatic prostate cancer

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scholarly article

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92056

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10.1038/NATURE07782

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David M Sabatini

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24194553

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1040350294

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19279572

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2692085

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