JNK1 is not essential for TNF-mediated joint disease. - Wikidata - wikimedia - TriplyDB

JNK1 is not essential for TNF-mediated joint disease.

JNK1 is not essential for TNF-mediated joint disease.

http://www.wikidata.org/entity/Q24799357

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Fibroblast-like synoviocytes: key effector cells in rheumatoid arthritis MAPK signalling in rheumatoid joint destruction: can we unravel the puzzle?c-Jun N-Terminal Kinases Mediate a Wide Range of Targets in the Metastatic Cascade JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis JNK-1 deficiency limits macrophage-mediated antigen-induced arthritis.Increased sensitivity to apoptosis induced by methotrexate is mediated by JNK.Anti-Inflammatory Effects and Joint Protection in Collagen-Induced Arthritis after Treatment with IQ-1S, a Selective c-Jun N-Terminal Kinase Inhibitor.From cellular receptors to transduction-transcription pathways for cytokines: at which level should the inhibition be targeted in inflammation?c-Jun N-Terminal Kinase in Inflammation and Rheumatic Diseases.Mitogen activated protein kinase inhibitors: where are we now and where are we going?Mitogen-activated protein kinases as therapeutic targets for rheumatoid arthritis.JNK1, but not JNK2, is required in two mechanistically distinct models of inflammatory arthritis Stimulation of macrophage TNFalpha production by orthopaedic wear particles requires activation of the ERK1/2/Egr-1 and NF-kappaB pathways but is independent of p38 and JNK.A multitude of kinases--which are the best targets in treating rheumatoid arthritis?

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P2860

JNK1 is not essential for TNF-mediated joint disease.

http://www.wikidata.org/entity/Q24799357

description

2004 nî lūn-bûn

@nan

2004 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2004 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2004年の論文

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2004年学术文章

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2004年学术文章

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2004年学术文章

@zh-hans

2004年学术文章

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2004年学术文章

@zh-sg

2004年學術文章

@yue

name

JNK1 is not essential for TNF-mediated joint disease

@nl

JNK1 is not essential for TNF-mediated joint disease.

@ast

JNK1 is not essential for TNF-mediated joint disease.

@en

type

label

JNK1 is not essential for TNF-mediated joint disease

@nl

JNK1 is not essential for TNF-mediated joint disease.

@ast

JNK1 is not essential for TNF-mediated joint disease.

@en

prefLabel

JNK1 is not essential for TNF-mediated joint disease

@nl

JNK1 is not essential for TNF-mediated joint disease.

@ast

JNK1 is not essential for TNF-mediated joint disease.

@en

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Arthritis Research and Therapy

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JNK1 is not essential for TNF-mediated joint disease.

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P2093

Günter Steiner

http://www.w3.org/2001/XMLSchema#string

Jean-Pierre David

http://www.w3.org/2001/XMLSchema#string

Kurt Redlich

http://www.w3.org/2001/XMLSchema#string

Marcus Köller

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Silvia Hayer

http://www.w3.org/2001/XMLSchema#string

P2860

Transgenic mice expressing human tumour necrosis factor: a predictive genetic model of arthritis

Protection against cartilage and bone destruction by systemic interleukin-4 treatment in established murine type II collagen-induced arthritis

Randomised double-blind comparison of chimeric monoclonal antibody to tumour necrosis factor alpha (cA2) versus placebo in rheumatoid arthritis

Regulation of c-Jun N-terminal kinase by MEKK-2 and mitogen-activated protein kinase kinase kinases in rheumatoid arthritis

JNK2 contains a specificity-determining region responsible for efficient c-Jun binding and phosphorylation

c-Jun N-terminal kinase is required for metalloproteinase expression and joint destruction in inflammatory arthritis

Mitogen-activated protein kinase pathways mediated by ERK, JNK, and p38 protein kinases

Infliximab and methotrexate in the treatment of rheumatoid arthritis. Anti-Tumor Necrosis Factor Trial in Rheumatoid Arthritis with Concomitant Therapy Study Group

Osteoclasts are essential for TNF-alpha-mediated joint destruction.

Signal transduction networks in rheumatoid arthritis.

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10.1186/AR1473

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