JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis - Wikidata - wikimedia - TriplyDB

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

http://www.wikidata.org/entity/Q34438110

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Cellular targeting in autoimmunity Novel synthetic toll-like receptor 4/MD2 ligands attenuate sterile inflammation Suppression of collagen-induced arthritis in growth arrest and DNA damage-inducible protein 45β-deficient mice Anti-Inflammatory Effects and Joint Protection in Collagen-Induced Arthritis after Treatment with IQ-1S, a Selective c-Jun N-Terminal Kinase Inhibitor.Subthreshold IKK activation modulates the effector functions of primary mast cells and allows specific targeting of transformed mast cells.Selective involvement of ERK and JNK mitogen-activated protein kinases in early rheumatoid arthritis (1987 ACR criteria compared to 2010 ACR/EULAR criteria): a prospective study aimed at identification of diagnostic and prognostic biomarkers as well Regulation of inflammatory arthritis by the upstream kinase mitogen activated protein kinase kinase 7 in the c-Jun N-terminal kinase pathway c-Jun N-Terminal Kinase in Inflammation and Rheumatic Diseases.Neutrophils orchestrate their own recruitment in murine arthritis through C5aR and FcγR signaling.Pathways for bone loss in inflammatory disease.Mast cells: an expanding pathophysiological role from allergy to other disorders.Network Motifs in JNK Signaling.n-3 Polyunsaturated fatty acids and mast cell activation.JNK1, but not JNK2, is required in two mechanistically distinct models of inflammatory arthritis Megakaryocytes compensate for Kit insufficiency in murine arthritis.[6]-Shogaol inhibits the production of proinflammatory cytokines via regulation of NF-κB and phosphorylation of JNK in HMC-1 cells.Activation/Inhibition of mast cells by supra-optimal antigen concentrations ERK2 and JNK1 contribute to TNF-α-induced IL-8 expression in synovial fibroblasts.Cutting edge: nitrogen bisphosphonate-induced inflammation is dependent upon mast cells and IL-1.Effects of sodium selenite on c-Jun N-terminal kinase signalling pathway induced by oxidative stress in human chondrocytes and c-Jun N-terminal kinase expression in patients with Kashin-Beck disease, an endemic osteoarthritis.Mast cells as sources of cytokines, chemokines, and growth factors.Fc receptor beta chain deficiency exacerbates murine arthritis in the anti-type II collagen antibody-induced experimental model.

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JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

http://www.wikidata.org/entity/Q34438110

description

2010 nî lūn-bûn

@nan

2010 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի դեկտեմբերին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

@zh-hant

2010年論文

@zh-hk

2010年論文

@zh-mo

2010年論文

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2010年论文

@wuu

name

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

@ast

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

@en

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

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type

label

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

@ast

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

@en

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

@nl

prefLabel

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

@ast

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

@en

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

@nl

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PNAS.1016401107

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Proceedings of the National Academy of Sciences of the United States of America

P1476

JNK1 controls mast cell degranulation and IL-1{beta} production in inflammatory arthritis

@en

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Brian Crain

http://www.w3.org/2001/XMLSchema#string

Gary S Firestein

http://www.w3.org/2001/XMLSchema#string

Jun-ichi Kashiwakura

http://www.w3.org/2001/XMLSchema#string

Maripat Corr

http://www.w3.org/2001/XMLSchema#string

Monica Guma

http://www.w3.org/2001/XMLSchema#string

Toshiaki Kawakami

http://www.w3.org/2001/XMLSchema#string

Yuko Kawakami

http://www.w3.org/2001/XMLSchema#string

P2860

Rapid and specific conversion of precursor interleukin 1 beta (IL-1 beta) to an active IL-1 species by human mast cell chymase

Molecular cloning of the interleukin-1 beta converting enzyme

JNK1 is not essential for TNF-mediated joint disease.

Signal transduction by the JNK group of MAP kinases

A novel heterodimeric cysteine protease is required for interleukin-1 beta processing in monocytes

Effects of a selective inhibitor of the Abl tyrosine kinase on the growth of Bcr-Abl positive cells

c-Jun can recruit JNK to phosphorylate dimerization partners via specific docking interactions

c-Jun N-terminal kinase is required for metalloproteinase expression and joint destruction in inflammatory arthritis

Neutrophils in a mouse model of autoantibody-mediated arthritis: critical producers of Fc receptor gamma, the receptor for C5a, and lymphocyte function-associated antigen 1

Mitogen-activated protein (MAP) kinase pathways: regulation and physiological functions

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22122-22127

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scholarly article

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10.1073/PNAS.1016401107

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107

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