Central nervous system mechanisms linking the consumption of palatable high-fat diets to the defense of greater adiposity - Wikidata - wikimedia - TriplyDB

Central nervous system mechanisms linking the consumption of palatable high-fat diets to the defense of greater adiposity

Central nervous system mechanisms linking the consumption of palatable high-fat diets to the defense of greater adiposity

http://www.wikidata.org/entity/Q26853424

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The Obese Brain--Effects of Bariatric Surgery on Energy Balance Neurocircuitry Intranasal insulin therapy for cognitive impairment and neurodegeneration: current state of the art FXR is a molecular target for the effects of vertical sleeve gastrectomy Insulin increases central apolipoprotein E levels as revealed by an improved technique for collection of cerebrospinal fluid from rats Neuroendocrine circuits governing energy balance and stress regulation: functional overlap and therapeutic implications Intestine-specific deletion of acyl-CoA:monoacylglycerol acyltransferase (MGAT) 2 protects mice from diet-induced obesity and glucose intolerance.Neurobiology of food intake in health and disease.Loss of melanocortin-4 receptor function attenuates HPA responses to psychological stress Counterregulation of insulin by leptin as key component of autonomic regulation of body weight.CB1 cannabinoid receptor in SF1-expressing neurons of the ventromedial hypothalamus determines metabolic responses to diet and leptin.Central IKKβ inhibition prevents air pollution mediated peripheral inflammation and exaggeration of type II diabetes.Wired on sugar: the role of the CNS in the regulation of glucose homeostasis.Protein quality and the protein to carbohydrate ratio within a high fat diet influences energy balance and the gut microbiota in C57BL/6J mice.Single rapamycin administration induces prolonged downward shift in defended body weight in rats Metabolic Impairments Precede Changes in Hunger and Food Intake Following Short-Term Administration of Second-Generation Antipsychotics Food, health, and complexity: towards a conceptual understanding to guide collaborative public health action Gut-brain nutrient signaling. Appetition vs. satiation.Fibroblast growth factor-19 action in the brain reduces food intake and body weight and improves glucose tolerance in male rats.The liver: Key in regulating appetite and body weight The neurobiology of food intake in an obesogenic environment.Abdominal fat is associated with a greater brain reward response to high-calorie food cues in Hispanic women.Leptin and the brain: then and now.An inconvenient truth about obesity.Knockout of inositol-requiring enzyme 1α in pro-opiomelanocortin neurons decreases fat mass via increasing energy expenditure.Activation and regulation of the pattern recognition receptors in obesity-induced adipose tissue inflammation and insulin resistance.Associations between BMI and fat taste sensitivity in humans.Fat sensing and metabolic syndrome.Amylin receptor signaling in the ventral tegmental area is physiologically relevant for the control of food intake.Leptin resistance in diet-induced obesity: the role of hypothalamic inflammation.Downregulation of inflammatory markers by conjugated linoleic acid isomers in human cultured astrocytes.Red blood cell β-adrenergic receptors contribute to diet-induced energy expenditure by increasing O2 supply.Neuronal Rap1 Regulates Energy Balance, Glucose Homeostasis, and Leptin Actions Oxygen Consumption Rate and Energy Expenditure in Mice: Indirect Calorimetry.Physiology. Food as a hormone.Whey protein isolate counteracts the effects of a high-fat diet on energy intake and hypothalamic and adipose tissue expression of energy balance-related genes.The impact of ageing, fasting and high-fat diet on central and peripheral glucose tolerance and glucose-sensing neural networks in the arcuate nucleus.High-fat diet-induced downregulation of anorexic leukemia inhibitory factor in the brain stem.High-fat diet-related stimulation of sweetness desire is greater in women than in men despite high vegetable intake.

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P2860

Central nervous system mechanisms linking the consumption of palatable high-fat diets to the defense of greater adiposity

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2012 nî lūn-bûn

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2012 թուականի Փետրուարին հրատարակուած գիտական յօդուած

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J.CMET.2011.12.013

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Cell Metabolism

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Central nervous system mechani ...... e defense of greater adiposity

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Karen K. Ryan

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Randy J. Seeley

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P2860

Inflammation, stress, and diabetes

Toll-like receptor signalling

Two defects contribute to hypothalamic leptin resistance in mice with diet-induced obesity

Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance

The full-length leptin receptor has signaling capabilities of interleukin 6-type cytokine receptors

Endoplasmic reticulum stress and the inflammatory basis of metabolic disease

Diabetes mellitus: a "thrifty" genotype rendered detrimental by "progress"?

Signal integration in the endoplasmic reticulum unfolded protein response

The role of pattern-recognition receptors in innate immunity: update on Toll-like receptors

Inflammation and metabolic disorders

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