Fibroblast growth factor-19 action in the brain reduces food intake and body weight and improves glucose tolerance in male rats. - Wikidata - wikimedia - TriplyDB

Fibroblast growth factor-19 action in the brain reduces food intake and body weight and improves glucose tolerance in male rats.

Fibroblast growth factor-19 action in the brain reduces food intake and body weight and improves glucose tolerance in male rats.

http://www.wikidata.org/entity/Q36486182

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Recent advances in metabolic and bariatric surgery Fibroblast Growth Factor Signaling in Metabolic Regulation Brain regulation of energy balance and body weight.Obese patients after gastric bypass surgery have lower brain-hedonic responses to food than after gastric banding.The Role and Potential Therapeutic Implications of the Fibroblast Growth Factors in Energy Balance and Type 2 Diabetes.Gut-Brain Cross-Talk in Metabolic Control.Tissue-specific actions of the metabolic hormones FGF15/19 and FGF21.Gut-brain mechanisms controlling glucose homeostasis.FGF1 and FGF19 reverse diabetes by suppression of the hypothalamic-pituitary-adrenal axis.Bariatric surgery: prevalence, predictors, and mechanisms of diabetes remission.Temporal changes in bile acid levels and 12α-hydroxylation after Roux-en-Y gastric bypass surgery in type 2 diabetes.Bile Acids as Hormones: The FXR-FGF15/19 Pathway Regulation of bile acid homeostasis by the intestinal Diet1-FGF15/19 axis.Glucose intolerance induced by blockade of central FGF receptors is linked to an acute stress response Minireview: Roles of Fibroblast Growth Factors 19 and 21 in Metabolic Regulation and Chronic Diseases Metabolic effects of intestinal absorption and enterohepatic cycling of bile acids.Impact of gut hormone FGF-19 on type-2 diabetes and mitochondrial recovery in a prospective study of obese diabetic women undergoing bariatric surgery Metabolic fibroblast growth factors (FGFs): Mediators of energy homeostasis.Link Between Increased Satiety Gut Hormones and Reduced Food Reward After Gastric Bypass Surgery for Obesity.Central injection of fibroblast growth factor 1 induces sustained remission of diabetic hyperglycemia in rodents.FGF19 action in the brain induces insulin-independent glucose lowering A Genetic Screen Identifies Hypothalamic Fgf15 as a Regulator of Glucagon Secretion.Carbohydrate feeding dissociates the postprandial FGF19 response from circulating bile acid levels in humans.Cooperation between brain and islet in glucose homeostasis and diabetes.Digestive physiology of the pig symposium: intestinal bile acid sensing is linked to key endocrine and metabolic signaling pathways.Non-Neuronal Cells in the Hypothalamic Adaptation to Metabolic Signals Integration of satiety signals by the central nervous system.Mechanisms underlying weight loss after bariatric surgery.The endocrinology of food intake.Fibroblast growth factor 19-targeted therapies for the treatment of metabolic disease.Therapeutic potential of the endocrine fibroblast growth factors FGF19, FGF21 and FGF23.The Contributing Role of Bile Acids to Metabolic Improvements After Obesity and Metabolic Surgery.FGF1 - a new weapon to control type 2 diabetes mellitus.FGF19: How gut talks to brain to keep your sugar down.Central action of FGF19 reduces hypothalamic AGRP/NPY neuron activity and improves glucose metabolism.Fibroblast growth factor 19 entry into brain.Bile acids at the cross-roads of gut microbiome-host cardiometabolic interactions.Bile Acid Signaling Pathways from the Enterohepatic Circulation to the Central Nervous System.FGF19, FGF21, and an FGFR1/β-Klotho-Activating Antibody Act on the Nervous System to Regulate Body Weight and Glycemia.Bile acids in glucose metabolism in health and disease.

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Fibroblast growth factor-19 action in the brain reduces food intake and body weight and improves glucose tolerance in male rats.

http://www.wikidata.org/entity/Q36486182

description

2012 nî lūn-bûn

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2012年の論文

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年学术文章

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2012年學術文章

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2012年學術文章

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2012年學術文章

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name

Fibroblast growth factor-19 ac ...... lucose tolerance in male rats.

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Fibroblast growth factor-19 ac ...... lucose tolerance in male rats.

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type

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Fibroblast growth factor-19 ac ...... lucose tolerance in male rats.

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Fibroblast growth factor-19 ac ...... lucose tolerance in male rats.

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Fibroblast growth factor-19 ac ...... lucose tolerance in male rats.

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Fibroblast growth factor-19 ac ...... lucose tolerance in male rats.

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Fibroblast growth factor-19 ac ...... glucose tolerance in male rats

@en

P2093

Ruth Gutierrez-Aguilar

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Shrawan G Gaitonde

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Stephen C Woods

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P2860

Tissue-specific expression of betaKlotho and fibroblast growth factor (FGF) receptor isoforms determines metabolic activity of FGF19 and FGF21

FGF19 as a postprandial, insulin-independent activator of hepatic protein and glycogen synthesis

Central nervous system mechanisms linking the consumption of palatable high-fat diets to the defense of greater adiposity

FGFR4 prevents hyperlipidemia and insulin resistance but underlies high-fat diet induced fatty liver

Fundamentals of FGF19 & FGF21 action in vitro and in vivo

Central nervous system control of food intake

Fibroblast growth factor 15 functions as an enterohepatic signal to regulate bile acid homeostasis

Therapeutic utilities of fibroblast growth factor 19.

Identification of FGFR4-activating mutations in human rhabdomyosarcomas that promote metastasis in xenotransplanted models.

Fibroblast growth factor 21 action in the brain increases energy expenditure and insulin sensitivity in obese rats

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9-15

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10.1210/EN.2012-1891

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1

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154

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Randy J. Seeley

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2012-11-26T00:00:00Z

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23183168

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