Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes - Wikidata - wikimedia - TriplyDB

Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

http://www.wikidata.org/entity/Q26860894

about

Role of bone morphogenetic protein-7 in renal fibrosis The Spectrum of Renal Allograft Failure Renal Oxygenation in the Pathophysiology of Chronic Kidney Disease.Signalling pathways involved in hypoxia-induced renal fibrosis.Bone marrow-derived multipotent stromal cells promote myocardial fibrosis and reverse remodeling of the left ventricle.Association of genetic variants with dyslipidemia and chronic kidney disease in a longitudinal population-based genetic epidemiological study Interstitial pericytes decrease in aged mouse kidneys.Cells of renin lineage express hypoxia inducible factor 2α following experimental ureteral obstruction.Targeted complement inhibition and microvasculature in transplants: a therapeutic perspective.Expanding roles of the hypoxia-response network in chronic kidney disease.Stromal and immune cells in gut fibrosis: the myofibroblast and the scarface.Cooperative Oxygen Sensing by the Kidney and Carotid Body in Blood Pressure Control.Polycythemia, capillary rarefaction, and focal glomerulosclerosis in two adolescents born extremely low birth weight and premature.The hypoxic tumour microenvironment.Emerging role of Twist1 in fibrotic diseases.Hypoxia induces pulmonary fibroblast proliferation through NFAT signaling.Capillary rarefaction from the kidney point of view.

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P2860

Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

http://www.wikidata.org/entity/Q26860894

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2014 nî lūn-bûn

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2014 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած

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2014 թվականի նոյեմբերին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

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Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

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Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

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Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

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Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

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Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

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Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

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Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

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Kidney international supplements

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Hypoxia and fibrosis in chronic kidney disease: crossing at pericytes

@en

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Imari Mimura

http://www.w3.org/2001/XMLSchema#string

Kumi Shoji

http://www.w3.org/2001/XMLSchema#string

Masaomi Nangaku

http://www.w3.org/2001/XMLSchema#string

Takahisa Kawakami

http://www.w3.org/2001/XMLSchema#string

Tetsuhiro Tanaka

http://www.w3.org/2001/XMLSchema#string

P2860

Hypoxia promotes fibrogenesis in vivo via HIF-1 stimulation of epithelial-to-mesenchymal transition

Pericytes: developmental, physiological, and pathological perspectives, problems, and promises

Hypoxia-induced VEGF and collagen I expressions are associated with angiogenesis and fibrogenesis in experimental cirrhosis

Enhancement of angiogenesis through stabilization of hypoxia-inducible factor-1 by silencing prolyl hydroxylase domain-2 gene

Molecular mechanisms and clinical applications of angiogenesis

Fate tracing reveals the pericyte and not epithelial origin of myofibroblasts in kidney fibrosis

Origin and function of myofibroblasts in kidney fibrosis.

Peptide blockade of HIFalpha degradation modulates cellular metabolism and angiogenesis

Mouse model for noninvasive imaging of HIF prolyl hydroxylase activity: assessment of an oral agent that stimulates erythropoietin production

Targeting endothelium-pericyte cross talk by inhibiting VEGF receptor signaling attenuates kidney microvascular rarefaction and fibrosis.

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10.1038/KISUP.2014.20

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