RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury - Wikidata - wikimedia - TriplyDB

RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

http://www.wikidata.org/entity/Q27021935

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The role of neutrophils in the development of liver diseases Immunomicrobial pathogenesis of periodontitis: keystones, pathobionts, and host response T cells in organ ischemia reperfusion injury.Angels and demons: Th17 cells represent a beneficial response, while neutrophil IL-17 is associated with poor prognosis in squamous cervical cancer Basic biology and role of interleukin-17 in immunity and inflammation Association between Serum Interleukin-17A Level and High-Altitude Deacclimatization Syndrome.IL-17-producing peripheral blood CD177+ neutrophils increase in allergic asthmatic subjects Phosphatase Wip1 as a new therapeutic target for intestinal ischemia-reperfusion injury.Bruton Tyrosine Kinase Inhibition Attenuates Liver Damage in a Mouse Warm Ischemia and Reperfusion Model.Interaction of Neutrophils with Macrophages Promotes IL-1β Maturation and Contributes to Hepatic Ischemia-Reperfusion Injury.Th17/Treg immunoregulation and implications in treatment of sulfur mustard gas-induced lung diseases.Revisiting the Page & Schroeder model: the good, the bad and the unknowns in the periodontal host response 40 years later.Neutrophils releasing IL-17A into NETs are essential to plasma cell differentiation in inflamed tissue dependent on IL-1R.Th-17 regulatory cytokines IL-21, IL-23, and IL-6 enhance neutrophil production of IL-17 cytokines during asthma.Lymphocyte-specific deletion of IKK2 or NEMO mediates an increase in intrarenal Th17 cells and accelerates renal damage in an ischemia-reperfusion injury mouse model.A Reappraisal on the Potential Ability of Human Neutrophils to Express and Produce IL-17 Family Members In Vitro: Failure to Reproducibly Detect It.Tc17/IL-17A Up-Regulated the Expression of MMP-9 via NF-κB Pathway in Nasal Epithelial Cells of Patients With Chronic Rhinosinusitis

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RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

http://www.wikidata.org/entity/Q27021935

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2013 nî lūn-bûn

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2013 թուականի Ապրիլին հրատարակուած գիտական յօդուած

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2013 թվականի ապրիլին հրատարակված գիտական հոդված

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2013年の論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年論文

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2013年论文

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name

RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

@ast

RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

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RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

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type

label

RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

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RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

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RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

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RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

@ast

RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

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RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

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Journal of molecular cell biology

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RORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injury

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B. Ryffel

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B. Sun

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L. Lu

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Q. Liu

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R. Jiang

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S. G. Zheng

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X. Wang

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Y. Wang

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Z. Tan

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P2860

IL-1 and IL-23 mediate early IL-17A production in pulmonary inflammation leading to late fibrosis

Neutrophils in the activation and regulation of innate and adaptive immunity

Innate IL-17-producing cells: the sentinels of the immune system.

Paneth cell-derived interleukin-17A causes multiorgan dysfunction after hepatic ischemia and reperfusion injury.

IL-17A expression is localised to both mononuclear and polymorphonuclear synovial cell infiltrates.

The role of neutrophils in the induction of glomerulonephritis by anti-myeloperoxidase antibodies.

Reactive oxygen species mediate liver injury through parenchymal nuclear factor-kappaB inactivation in prolonged ischemia/reperfusion.

Induction and effector functions of T(H)17 cells.

Inflammasome-IL-1-Th17 response in allergic lung inflammation.

IL-17, produced by lymphocytes and neutrophils, is necessary for lipopolysaccharide-induced airway neutrophilia: IL-15 as a possible trigger.

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10.1093/JMCB/MJS065

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reperfusion injury

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