The role of neutrophils in the induction of glomerulonephritis by anti-myeloperoxidase antibodies.
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The Prevalence and Management of Pauci-Immune Glomerulonephritis and Vasculitis in Western CountriesOverview of the Pathogenesis of ANCA-Associated VasculitisAnimal models of anti-neutrophil cytoplasmic antibody-associated vasculitisRORγt+IL-17+ neutrophils play a critical role in hepatic ischemia-reperfusion injuryThe neutrophil in antineutrophil cytoplasmic autoantibody-associated vasculitisBench-to-bedside review: Pulmonary–renal syndromes – an update for the intensivistExperimental Autoimmune VasculitisAnti-Neutrophil Cytoplasmic Antibody Pathogenesis in Small-Vessel VasculitisUpdates in ANCA-associated vasculitisPathophysiology of ANCA-associated small vessel vasculitisClassification of antineutrophil cytoplasmic autoantibody vasculitides: the role of antineutrophil cytoplasmic autoantibody specificity for myeloperoxidase or proteinase 3 in disease recognition and prognosisThe immunopathology of ANCA-associated vasculitisCritical but overlapping role of FcgammaRIII and FcgammaRIV in activation of murine neutrophils by immobilized immune complexesAnti-proteinase 3 anti-neutrophil cytoplasm autoantibodies recapitulate systemic vasculitis in mice with a humanized immune systemNeutrophils in a mouse model of autoantibody-mediated arthritis: critical producers of Fc receptor gamma, the receptor for C5a, and lymphocyte function-associated antigen 1Clinical applications of therapeutic apheresis.Spatiotemporal expression of chemokines and chemokine receptors in experimental anti-myeloperoxidase antibody-mediated glomerulonephritis.The use of small molecule high-throughput screening to identify inhibitors of the proteinase 3-NB1 interaction.Review: Complement and its regulatory proteins in kidney diseases.High mobility group box-1 contributes to anti-myeloperoxidase antibody-induced glomerular endothelial cell injury through a moesin-dependent route.Mechanistic connection between inflammation and fibrosisHMGB1 contributes to glomerular endothelial cell injury in ANCA-associated vasculitis through enhancing endothelium-neutrophil interactionsManagement of ANCA-associated vasculitis: Current trends and future prospectsTh17 cells promote autoimmune anti-myeloperoxidase glomerulonephritis.Epigenetic basis for aberrant upregulation of autoantigen genes in humans with ANCA vasculitisp38MAPK, ERK and PI3K signaling pathways are involved in C5a-primed neutrophils for ANCA-mediated activation.CD4+ T cells sensitized by vascular smooth muscle induce vasculitis, and interferon gamma is critical for the initiation of vascular pathology.The interaction between C5a and sphingosine-1-phosphate in neutrophils for antineutrophil cytoplasmic antibody mediated activation.Neutrophil surface presentation of the anti-neutrophil cytoplasmic antibody-antigen proteinase 3 depends on N-terminal processing.IgG glycan hydrolysis attenuates ANCA-mediated glomerulonephritis.Antineutrophil cytoplasmic antibodies, autoimmune neutropenia, and vasculitisHigh mobility group box 1 contributes to anti-neutrophil cytoplasmic antibody-induced neutrophils activation through receptor for advanced glycation end products (RAGE) and Toll-like receptor 4.Alternative complement pathway in the pathogenesis of disease mediated by anti-neutrophil cytoplasmic autoantibodiesNeutrophil-Related Gene Expression and Low-Density Granulocytes Associated With Disease Activity and Response to Treatment in Antineutrophil Cytoplasmic Antibody-Associated Vasculitis.Decreased CXCR1 and CXCR2 expression on neutrophils in anti-neutrophil cytoplasmic autoantibody-associated vasculitides potentially increases neutrophil adhesion and impairs migration.The immunodominant myeloperoxidase T-cell epitope induces local cell-mediated injury in antimyeloperoxidase glomerulonephritis.Cutting edge: IL-1β processing during Pseudomonas aeruginosa infection is mediated by neutrophil serine proteases and is independent of NLRC4 and caspase-1.Neutrophils contribute to development of a protective immune response during onset of infection with Leishmania donovani.Immune complexes isolated from patients with pulmonary tuberculosis modulate the activation and function of normal granulocytesPathogenic role and clinical relevance of antineutrophil cytoplasmic antibodies in vasculitides.
P2860
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P2860
The role of neutrophils in the induction of glomerulonephritis by anti-myeloperoxidase antibodies.
description
2005 nî lūn-bûn
@nan
2005 թուականի Յուլիսին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի հուլիսին հրատարակված գիտական հոդված
@hy
2005年の論文
@ja
2005年論文
@yue
2005年論文
@zh-hant
2005年論文
@zh-hk
2005年論文
@zh-mo
2005年論文
@zh-tw
2005年论文
@wuu
name
The role of neutrophils in the ...... ti-myeloperoxidase antibodies.
@ast
The role of neutrophils in the ...... ti-myeloperoxidase antibodies.
@en
type
label
The role of neutrophils in the ...... ti-myeloperoxidase antibodies.
@ast
The role of neutrophils in the ...... ti-myeloperoxidase antibodies.
@en
prefLabel
The role of neutrophils in the ...... ti-myeloperoxidase antibodies.
@ast
The role of neutrophils in the ...... ti-myeloperoxidase antibodies.
@en
P2093
P2860
P1476
The role of neutrophils in the ...... ti-myeloperoxidase antibodies.
@en
P2093
Dennis Huugen
J Charles Jennette
Nobuyo Maeda
Peter Heeringa
Ronald J Falk
P2860
P356
10.1016/S0002-9440(10)62951-3
P407
P577
2005-07-01T00:00:00Z