Proteolysis of proBDNF is a key regulator in the formation of memory
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The neurotrophic compound J147 reverses cognitive impairment in aged Alzheimer's disease micePhysiological and pathological roles of tissue plasminogen activator and its inhibitor neuroserpin in the nervous systemThe Regulation of Cytokine Networks in Hippocampal CA1 Differentiates Extinction from Those Required for the Maintenance of Contextual Fear Memory after RecallChronic Microdose Lithium Treatment Prevented Memory Loss and Neurohistopathological Changes in a Transgenic Mouse Model of Alzheimer's DiseaseSubacute intranasal administration of tissue plasminogen activator promotes neuroplasticity and improves functional recovery following traumatic brain injury in ratsBDNF-dependent consolidation of fear memories in the perirhinal cortex.Rescue of long-term memory after reconsolidation blockade.Tissue plasminogen activator inhibits NMDA-receptor-mediated increases in calcium levels in cultured hippocampal neurons.Increasing tPA activity in astrocytes induced by multipotent mesenchymal stromal cells facilitate neurite outgrowth after stroke in the mouseQuantitatively and qualitatively different cellular processes are engaged in CA1 during the consolidation and reconsolidation of contextual fear memory.Treadmill exercise improves spatial learning ability by enhancing brain-derived neurotrophic factor expression in the attention-deficit/hyperactivity disorder rats.Xenon impairs reconsolidation of fear memories in a rat model of post-traumatic stress disorder (PTSD)A simple role for BDNF in learning and memory?Abnormalities in the zinc-metalloprotease-BDNF axis may contribute to megalencephaly and cortical hyperconnectivity in young autism spectrum disorder patients.The role of NR4A transcription factors in memory formationAging and a peripheral immune challenge interact to reduce mature brain-derived neurotrophic factor and activation of TrkB, PLCgamma1, and ERK in hippocampal synaptoneurosomes.Prenatal stress differentially alters brain-derived neurotrophic factor expression and signaling across rat strains.Behavior training reverses asymmetry in hippocampal transcriptome of the cav3.2 knockout mice.Extracellular proteolysis of reelin by tissue plasminogen activator following synaptic potentiationTreatment of TBI with collagen scaffolds and human marrow stromal cells increases the expression of tissue plasminogen activator.The sonic hedgehog pathway mediates brain plasticity and subsequent functional recovery after bone marrow stromal cell treatment of stroke in mice.Catalpol Induces Neuroprotection and Prevents Memory Dysfunction through the Cholinergic System and BDNF.Molecular specificity of multiple hippocampal processes governing fear extinctionModulation of fear memory by retrieval and extinction: a clue for memory deconsolidation.BDNF-TrkB signalling in fear learning: from genetics to neural networks.Is altered BDNF biosynthesis a general feature in patients with cognitive dysfunctions?An association study of the genetic polymorphisms in 13 neural plasticity-related genes with semantic and episodic memories.PROneurotrophins and CONSequences.Voluntary wheel running, but not a diet containing (-)-epigallocatechin-3-gallate and β-alanine, improves learning, memory and hippocampal neurogenesis in aged mice.Increased Hippocampal ProBDNF Contributes to Memory Impairments in Aged Mice.Pro-neurotrophins secreted from retinal ganglion cell axons are necessary for ephrinA-p75NTR-mediated axon guidance.Schizophrenia copy number variants and associative learningBDNF contributes to the facilitation of hippocampal synaptic plasticity and learning enabled by environmental enrichment.Hippocampal Regulation of Postsynaptic Density Homer1 by Associative Learning.The effect of ketamine on the consolidation and extinction of contextual fear memory.AMPA receptors control fear extinction through an Arc-dependent mechanism.Dose-dependent decrease in mortality with no cognitive or muscle function improvements due to dietary EGCG supplementation in aged mice.Aqueous extracts from asparagus stems prevent memory impairments in scopolamine-treated mice.Acute Administration of Branched-Chain Amino Acids Increases the Pro-BDNF/Total-BDNF Ratio in the Rat Brain.Tissue plasminogen activator does not alter development of acquired epilepsy.
P2860
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P2860
Proteolysis of proBDNF is a key regulator in the formation of memory
description
2008 nî lūn-bûn
@nan
2008 թուականին հրատարակուած գիտական յօդուած
@hyw
2008 թվականին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Proteolysis of proBDNF is a key regulator in the formation of memory
@ast
Proteolysis of proBDNF is a key regulator in the formation of memory
@en
Proteolysis of proBDNF is a key regulator in the formation of memory
@nl
type
label
Proteolysis of proBDNF is a key regulator in the formation of memory
@ast
Proteolysis of proBDNF is a key regulator in the formation of memory
@en
Proteolysis of proBDNF is a key regulator in the formation of memory
@nl
prefLabel
Proteolysis of proBDNF is a key regulator in the formation of memory
@ast
Proteolysis of proBDNF is a key regulator in the formation of memory
@en
Proteolysis of proBDNF is a key regulator in the formation of memory
@nl
P2860
P3181
P1433
P1476
Proteolysis of proBDNF is a key regulator in the formation of memory
@en
P2093
Philip Barnes
P2860
P3181
P356
10.1371/JOURNAL.PONE.0003248
P407
P577
2008-01-01T00:00:00Z