CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons - Wikidata - wikimedia - TriplyDB

CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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Recent progress in West Nile virus diagnosis and vaccination Feasibility of cross-protective vaccination against flaviviruses of the Japanese encephalitis serocomplex The Interferon-Stimulated Gene Ifi27l2a Restricts West Nile Virus Infection and Pathogenesis in a Cell-Type- and Region-Specific Manner Dual Function of Ccr5 during Langat Virus Encephalitis: Reduction in Neutrophil-Mediated Central Nervous System Inflammation and Increase in T Cell-Mediated Viral Clearance Acute systemic DNA damage in youth does not impair immune defense with aging West Nile Virus Infection in the Central Nervous System Role of natural killer and Gamma-delta T cells in West Nile virus infection Clearance of virus infection from the CNS Immunobiology of congenital cytomegalovirus infection of the central nervous system—the murine cytomegalovirus model Experimental infection of rhesus macaques and common marmosets with a European strain of West Nile virus IL-1β signaling promotes CNS-intrinsic immune control of West Nile virus infection Neurons are MHC class I-dependent targets for CD8 T cells upon neurotropic viral infection Gamma Interferon Plays a Crucial Early Antiviral Role in Protection against West Nile Virus Infection Pathogenesis of West Nile Virus Infection: a Balance between Virulence, Innate and Adaptive Immunity, and Viral Evasion CD4+ T-Cell Responses Are Required for Clearance of West Nile Virus from the Central Nervous System Antibody Recognition of Cell Surface-Associated NS1 Triggers Fc- Receptor-Mediated Phagocytosis and Clearance of West Nile Virus-Infected Cells CD40-CD40 Ligand Interactions Promote Trafficking of CD8+ T Cells into the Brain and Protection against West Nile Virus Encephalitis Fas Ligand Interactions Contribute to CD8+ T-Cell-Mediated Control of West Nile Virus Infection in the Central Nervous System Epitope discovery in West Nile virus infection: Identification and immune recognition of viral epitopes The relative contribution of antibody and CD8+ T cells to vaccine immunity against West Nile encephalitis virus Dengue Virus Replicon Expressing the Nonstructural Proteins Suffices To Enhance Membrane Expression of HLA Class I and Inhibit Lysis by Human NK Cells Drak2 contributes to West Nile virus entry into the brain and lethal encephalitis CXCR4 antagonism increases T cell trafficking in the central nervous system and improves survival from West Nile virus encephalitis Tumor Necrosis Factor Alpha Protects against Lethal West Nile Virus Infection by Promoting Trafficking of Mononuclear Leukocytes into the Central Nervous System Toll-Like Receptor 3 Has a Protective Role against West Nile Virus Infection Construction and Characterization of a Single-Cycle Chimeric Flavivirus Vaccine Candidate That Protects Mice against Lethal Challenge with Dengue Virus Type 2 The structural immunology of antibody protection against West Nile virus Toll-like Receptor 7 Mitigates Lethal West Nile Encephalitis via Interleukin 23-Dependent Immune Cell Infiltration and Homing IL-10 Signaling Blockade Controls Murine West Nile Virus Infection Progress on the development of therapeutics against West Nile virus NKp44 Receptor Mediates Interaction of the Envelope Glycoproteins from the West Nile and Dengue Viruses with NK Cells Tregs control the development of symptomatic West Nile virus infection in humans and mice Toll-like receptor 7-induced immune response to cutaneous West Nile virus infection Key role of T cell defects in age-related vulnerability to West Nile virus IPS-1 Is Essential for the Control of West Nile Virus Infection and Immunity Anti-West Nile virus activity of in vitro expanded human primary natural killer cells The dendritic and T cell responses to herpes simplex virus-1 are modulated by dietary vitamin E Interferon regulatory factor-1 (IRF-1) shapes both innate and CD8(+) T cell immune responses against West Nile virus infection A recombinant influenza A virus expressing domain III of West Nile virus induces protective immune responses against influenza and West Nile virus Chemokine Receptor Ccr7 Restricts Fatal West Nile Virus Encephalitis

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

http://www.wikidata.org/entity/Q27472780

description

2006 nî lūn-bûn

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2006 թուականի Յունուարին հրատարակուած գիտական յօդուած

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2006 թվականի հունվարին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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type

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

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JVI.80.1.119-129.2006

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Journal of Virology

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CD8+ T Cells Require Perforin To Clear West Nile Virus from Infected Neurons

@en

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Melanie A Samuel

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P2860

CD8+ T Cell Effector Mechanisms in Resistance to Infection

Rapid detection of west nile virus from human clinical specimens, field-collected mosquitoes, and avian samples by a TaqMan reverse transcriptase-PCR assay

Human dendritic cells are activated by dengue virus infection: enhancement by gamma interferon and implications for disease pathogenesis.

Neuronal CXCL10 directs CD8+ T-cell recruitment and control of West Nile virus encephalitis.

Alpha/beta interferon protects against lethal West Nile virus infection by restricting cellular tropism and enhancing neuronal survival

Lack of both Fas ligand and perforin protects from flavivirus-mediated encephalitis in mice.

B cells and antibody play critical roles in the immediate defense of disseminated infection by West Nile encephalitis virus.

Phenotypic changes in Langerhans' cells after infection with arboviruses: a role in the immune response to epidermally acquired viral infection?

Experimental encephalitis following peripheral inoculation of West Nile virus in mice of different ages

Morphologic and Molecular Characterization of New Cyclospora Species from Ethiopian Monkeys: C. cercopitheci sp.n., C. colobi sp.n., and C. papionis sp.n

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119-29

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scholarly article

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983654

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10.1128/JVI.80.1.119-129.2006

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1

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80

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Michael S. Diamond

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2006-01-01T00:00:00Z

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16352536

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West Nile virus

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1317548

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