Fas Ligand Interactions Contribute to CD8+ T-Cell-Mediated Control of West Nile Virus Infection in the Central Nervous System
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Dual Function of Ccr5 during Langat Virus Encephalitis: Reduction in Neutrophil-Mediated Central Nervous System Inflammation and Increase in T Cell-Mediated Viral ClearanceNeurons are MHC class I-dependent targets for CD8 T cells upon neurotropic viral infectionDrak2 contributes to West Nile virus entry into the brain and lethal encephalitisTumor Necrosis Factor Alpha Protects against Lethal West Nile Virus Infection by Promoting Trafficking of Mononuclear Leukocytes into the Central Nervous SystemThe structural immunology of antibody protection against West Nile virusToll-like Receptor 7 Mitigates Lethal West Nile Encephalitis via Interleukin 23-Dependent Immune Cell Infiltration and HomingProtective Role of Fas-FasL Signaling in Lethal Infection with Herpes Simplex Virus Type 2 in MiceApoptosis in animal models of virus-induced diseaseInterferon regulatory factor-1 (IRF-1) shapes both innate and CD8(+) T cell immune responses against West Nile virus infectionDevelopment of antigen-specific memory CD8+ T cells following live-attenuated chimeric West Nile virus vaccinationPivotal role of antibody and subsidiary contribution of CD8+ T cells to recovery from infection in a murine model of Japanese encephalitisQualitative differences in brain-infiltrating T cells are associated with a fatal outcome in mice infected with Japanese encephalitis virus.Fas/FasL pathway participates in regulation of antiviral and inflammatory response during mousepox infection of lungsIncreased protection from vaccinia virus infection in mice genetically prone to lymphoproliferative disorders.Differential Roles of Chemokines CCL2 and CCL7 in Monocytosis and Leukocyte Migration during West Nile Virus Infection.Human β-cell killing by autoreactive preproinsulin-specific CD8 T cells is predominantly granule-mediated with the potency dependent upon T-cell receptor avidity.Lymphocytes with cytotoxic activity induce rapid microtubule axonal destabilization independently and before signs of neuronal deathCD22 is required for protection against West Nile virus Infection.Effector CD4+ T-cell involvement in clearance of infectious herpes simplex virus type 1 from sensory ganglia and spinal cordsThe great balancing act: regulation and fate of antiviral T-cell interactionsInflammation on the mind: visualizing immunity in the central nervous system.Neuroprotective intervention by interferon-γ blockade prevents CD8+ T cell-mediated dendrite and synapse loss.Fas-mediated apoptotic signaling in the mouse brain following reovirus infectionChandipura virus induces neuronal death through Fas-mediated extrinsic apoptotic pathway.CNS infiltration of peripheral immune cells: D-Day for neurodegenerative disease?Interleukin-17A Promotes CD8+ T Cell Cytotoxicity To Facilitate West Nile Virus ClearanceDeath receptor-mediated apoptotic signaling is activated in the brain following infection with West Nile virus in the absence of a peripheral immune response.Reduced immune cell infiltration and increased pro-inflammatory mediators in the brain of Type 2 diabetic mouse model infected with West Nile virus.Tumor necrosis factor receptor/tumor necrosis factor family members in antiviral CD8 T-cell immunity.Immune responses to West Nile virus infection in the central nervous system.West Nile virus infection and immunity.CD8 and CD4 T cells in west nile virus immunity and pathogenesis.Neurons and T cells: Understanding this interaction for inflammatory neurological diseases.A lack of Fas/FasL signalling leads to disturbances in the antiviral response during ectromelia virus infection.The West Nile virus capsid protein blocks apoptosis through a phosphatidylinositol 3-kinase-dependent mechanism.Regulation of Apoptosis during Flavivirus Infection.Minocycline has anti-inflammatory effects and reduces cytotoxicity in an ex vivo spinal cord slice culture model of West Nile virus infection.Pattern recognition receptor MDA5 modulates CD8+ T cell-dependent clearance of West Nile virus from the central nervous system.CD8+ T cells use TRAIL to restrict West Nile virus pathogenesis by controlling infection in neurons.DICE: A novel tumor surveillance mechanism-a new therapy for cancer?
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P2860
Fas Ligand Interactions Contribute to CD8+ T-Cell-Mediated Control of West Nile Virus Infection in the Central Nervous System
description
2007 nî lūn-bûn
@nan
2007 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Fas Ligand Interactions Contri ...... in the Central Nervous System
@ast
Fas Ligand Interactions Contri ...... in the Central Nervous System
@en
Fas Ligand Interactions Contri ...... in the Central Nervous System
@nl
type
label
Fas Ligand Interactions Contri ...... in the Central Nervous System
@ast
Fas Ligand Interactions Contri ...... in the Central Nervous System
@en
Fas Ligand Interactions Contri ...... in the Central Nervous System
@nl
prefLabel
Fas Ligand Interactions Contri ...... in the Central Nervous System
@ast
Fas Ligand Interactions Contri ...... in the Central Nervous System
@en
Fas Ligand Interactions Contri ...... in the Central Nervous System
@nl
P2860
P356
P1433
P1476
Fas Ligand Interactions Contri ...... in the Central Nervous System
@en
P2860
P304
P356
10.1128/JVI.01136-07
P577
2007-11-01T00:00:00Z