The FBP interacting repressor targets TFIIH to inhibit activated transcription
about
Control of pre-mRNA splicing by the general splicing factors PUF60 and U2AF(65)A yeast four-hybrid system identifies Cdk-activating kinase as a regulator of the XPD helicase, a subunit of transcription factor IIHThe FUSE binding proteins FBP1 and FBP3 are potential c-myc regulators in renal, but not in prostate and bladder cancerNuclear targeting determinants of the far upstream element binding protein, a c-myc transcription factorThe FUSE/FBP/FIR/TFIIH system is a molecular machine programming a pulse of c-myc expressionDifferentiation-induced colocalization of the KH-type splicing regulatory protein with polypyrimidine tract binding protein and the c-src pre-mRNAU2AF homology motifs: protein recognition in the RRM worldSingle-stranded DNA-binding proteins: multiple domains for multiple functionsDimerization of FIR upon FUSE DNA binding suggests a mechanism of c-myc inhibitionDimerization and protein binding specificity of the U2AF homology motif of the splicing factor Puf60The N-terminus of FILIA Forms an Atypical KH Domain with a Unique Extension Involved in Interaction with RNADownregulation of FUSE-binding protein and c-myc by tRNA synthetase cofactor p38 is required for lung cell differentiationAlleviation of PC4-mediated transcriptional repression by the ERCC3 helicase activity of general transcription factor TFIIHThe dynamic response of upstream DNA to transcription-generated torsional stressTFIIH operates through an expanded proximal promoter to fine-tune c-myc expression.Identification of candidate growth promoting genes in ovarian cancer through integrated copy number and expression analysis.Controlling the Master: Chromatin Dynamics at the MYC Promoter Integrate Developmental SignalingAlternative splicing of FBP-interacting repressor coordinates c-Myc, P27Kip1/cyclinE and Ku86/XRCC5 expression as a molecular sensor for bleomycin-induced DNA damage pathway.Myc in model organisms: a view from the flyroom.The splicing factor FUBP1 is required for the efficient splicing of oncogene MDM2 pre-mRNA.The regulatory role for the ERCC3 helicase of general transcription factor TFIIH during promoter escape in transcriptional activationFBP1 Is an Interacting Partner of Menin.An 8 nt RNA triggers a rate-limiting shift of RNA polymerase II complexes into elongation.Gene therapy of c-myc suppressor FUSE-binding protein-interacting repressor by Sendai virus delivery prevents tracheal stenosis.Regulation of BRCA1 transcription by specific single-stranded DNA binding factorsFBPs are calibrated molecular tools to adjust gene expressionHaploinsufficiency of the c-myc transcriptional repressor FIR, as a dominant negative-alternative splicing model, promoted p53-dependent T-cell acute lymphoblastic leukemia progression by activating Notch1.A proteomic analysis of the functional effects of fatty acids in NIH 3T3 fibroblastsOncoScape: Exploring the cancer aberration landscape by genomic data fusion.A subset of nuclear receptor coregulators act as coupling proteins during synthesis and maturation of RNA transcriptsFar upstream element-binding protein 1 is a prognostic biomarker and promotes nasopharyngeal carcinoma progression.Overexpression of the far upstream element binding protein 1 in hepatocellular carcinoma is required for tumor growth.Isolation and characterization of a novel H1.2 complex that acts as a repressor of p53-mediated transcription.Far Upstream Element Binding Protein Plays a Crucial Role in Embryonic Development, Hematopoiesis, and Stabilizing Myc Expression Levels.KSRP: a checkpoint for inflammatory cytokine production in astrocytesPUF60: a prominent new target of the autoimmune response in dermatomyositis and Sjögren's syndrome.Hierarchical mechanisms build the DNA-binding specificity of FUSE binding protein.Far upstream element-binding protein 1 and RNA secondary structure both mediate second-step splicing repression.Far upstream element binding protein 1: a commander of transcription, translation and beyondLoss of the xeroderma pigmentosum group B protein binding site impairs p210 BCR/ABL1 leukemogenic activity.
P2860
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P2860
The FBP interacting repressor targets TFIIH to inhibit activated transcription
description
2000 nî lūn-bûn
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2000 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2000 թվականի փետրվարին հրատարակված գիտական հոդված
@hy
2000年の論文
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2000年論文
@yue
2000年論文
@zh-hant
2000年論文
@zh-hk
2000年論文
@zh-mo
2000年論文
@zh-tw
2000年论文
@wuu
name
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@ast
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@en
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@nl
type
label
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@ast
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@en
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@nl
prefLabel
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@ast
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@en
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@nl
P2093
P3181
P1433
P1476
The FBP interacting repressor targets TFIIH to inhibit activated transcription
@en
P2093
P304
P3181
P356
10.1016/S1097-2765(00)80428-1
P407
P577
2000-02-01T00:00:00Z