Human spinal motoneurons express low relative abundance of GluR2 mRNA: an implication for excitotoxicity in ALS
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ALS Patient Stem Cells for Unveiling Disease Signatures of Motoneuron Susceptibility: Perspectives on the Deadly Mitochondria, ER Stress and Calcium TriadAdenosine-to-inosine RNA editing and human diseaseExcitotoxicity and ALS: what is unique about the AMPA receptors expressed on spinal motor neurons?Expression of AMPA and NMDA receptor subunits in the cervical spinal cord of wobbler miceGlutamate transporters and the excitotoxic path to motor neuron degeneration in amyotrophic lateral sclerosis.Editing of neurotransmitter receptor and ion channel RNAs in the nervous systemA-to-I RNA editing and human disease.Evidence for low GluR2 AMPA receptor subunit expression at synapses in the rat basolateral amygdala.Co-occurrence of TDP-43 mislocalization with reduced activity of an RNA editing enzyme, ADAR2, in aged mouse motor neuronsPersistent inward currents in spinal motoneurons: important for normal function but potentially harmful after spinal cord injury and in amyotrophic lateral sclerosis.Unravelling the enigma of selective vulnerability in neurodegeneration: motor neurons resistant to degeneration in ALS show distinct gene expression characteristics and decreased susceptibility to excitotoxicity.The molecular pharmacology and cell biology of alpha-amino-3-hydroxy-5-methyl-4-isoxazolepropionic acid receptorsAstrocytes regulate GluR2 expression in motor neurons and their vulnerability to excitotoxicity.Mitochondrial Ca(2+) and neurodegenerationGlycinergic innervation of motoneurons is deficient in amyotrophic lateral sclerosis mice: a quantitative confocal analysis.Altered presymptomatic AMPA and cannabinoid receptor trafficking in motor neurons of ALS model mice: implications for excitotoxicity.AMPA receptors as drug targets in neurological disease--advantages, caveats, and future outlook.Gene regulation and genetics in neurochemistry, past to future.When Does ALS Start? ADAR2-GluA2 Hypothesis for the Etiology of Sporadic ALS.Regulation of Human Endonuclease V Activity and Relocalization to Cytoplasmic Stress Granules.GluR2 deficiency accelerates motor neuron degeneration in a mouse model of amyotrophic lateral sclerosis.C9ORF72 repeat expansion causes vulnerability of motor neurons to Ca2+-permeable AMPA receptor-mediated excitotoxicity.A role for calpain-dependent cleavage of TDP-43 in amyotrophic lateral sclerosis pathology.The voltage-gated calcium channel blocker lomerizine is neuroprotective in motor neurons expressing mutant SOD1, but not TDP-43.
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P2860
Human spinal motoneurons express low relative abundance of GluR2 mRNA: an implication for excitotoxicity in ALS
description
2003 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի մայիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2003
@ast
scientific article (publication date: May 2003)
@en
vedecký článok (publikovaný 2003-05)
@sk
vědecký článek publikovaný v roce 2003
@cs
wetenschappelijk artikel (gepubliceerd in 2003-05)
@nl
наукова стаття, опублікована в травні 2003
@uk
مقالة علمية (نشرت في مايو 2003)
@ar
name
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@ast
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@en
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@nl
type
label
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@ast
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@en
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@nl
prefLabel
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@ast
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@en
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@nl
P2093
P1476
Human spinal motoneurons expre ...... tion for excitotoxicity in ALS
@en
P2093
Hideji Hashida
Hitoshi Aizawa
Ichiro Kanazawa
Seon-Yong Jeong
Yukio Kawahara
P356
10.1046/J.1471-4159.2003.01703.X
P407
P577
2003-05-01T00:00:00Z