Constitutive expression of LIGHT on T cells leads to lymphocyte activation, inflammation, and tissue destruction
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LIGHT, a member of the tumor necrosis factor ligand superfamily, prevents tumor necrosis factor-alpha-mediated human primary hepatocyte apoptosis, but not Fas-mediated apoptosisEvolutionarily divergent herpesviruses modulate T cell activation by targeting the herpesvirus entry mediator cosignaling pathwayThe intriguing biology of the tumour necrosis factor/tumour necrosis factor receptor superfamily: players, rules and the gamesDifferential expression of LIGHT and its receptors in human placental villi and amniochorion membranesGenetically engineered mouse models for studying inflammatory bowel diseaseThe Lymphotoxin Network: orchestrating a type I interferon response to optimize adaptive immunityMechanistic Basis for Functional Promiscuity in the TNF and TNF Receptor Superfamilies: Structure of the LIGHT:DcR3 AssemblyIncreased Heterologous Protein Expression in Drosophila S2 Cells for Massive Production of Immune Ligands/Receptors and Structural Analysis of Human HVEMMechanisms regulating expression of the tumor necrosis factor-related light gene. Role of calcium-signaling pathway in the transcriptional controlCritical roles for LIGHT and its receptors in generating T cell-mediated immunity during Leishmania donovani infectionTRAF2 plays a key, nonredundant role in LIGHT-lymphotoxin beta receptor signaling.The role of herpesvirus entry mediator as a negative regulator of T cell-mediated responses.Constitutive TL1A (TNFSF15) expression on lymphoid or myeloid cells leads to mild intestinal inflammation and fibrosisTumour necrosis factor superfamily members in the pathogenesis of inflammatory bowel diseaseRecent progress in herpes simplex virus immunobiology and vaccine researchPolymorphic variants of LIGHT (TNF superfamily-14) alter receptor avidity and bioavailability.Herpes simplex virus glycoprotein D interferes with binding of herpesvirus entry mediator to its ligands through downregulation and direct competitionEssential role of TNF family molecule LIGHT as a cytokine in the pathogenesis of hepatitis.The delay in the development of experimental colitis from isomaltosyloligosaccharides in rats is dependent on the degree of polymerizationCloning, expression, and characterization of TNFSF14 (LIGHT) gene in mefugu, Takifugu obscurus.Regulation of inflammation, autoimmunity, and infection immunity by HVEM-BTLA signaling.CD40L expressed from the canarypox vector, ALVAC, can boost immunogenicity of HIV-1 canarypox vaccine in mice and enhance the in vitro expansion of viral specific CD8+ T cell memory responses from HIV-1-infected and HIV-1-uninfected individualsMicroRNA regulate immune pathways in T-cells in multiple sclerosis (MS).Herpesvirus entry mediator (TNFRSF14) regulates the persistence of T helper memory cell populations.LIGHT/TNFSF14 increases osteoclastogenesis and decreases osteoblastogenesis in multiple myeloma-bone disease.CD95 ligand--death factor and costimulatory molecule?The impact of CCR7 and CXCR5 on lymphoid organ development and systemic immunity.Lymphoid microenvironment in the gut for immunoglobulin A and inflammation.Genetic evidence that intratumoral T-cell proliferation and activation are associated with recurrence and survival in patients with resected colorectal liver metastases.A role for surface lymphotoxin in experimental autoimmune encephalomyelitis independent of LIGHT.Tumor-specific T cells signal tumor destruction via the lymphotoxin beta receptorCD161 DEFINES EFFECTOR T CELLS THAT EXPRESS LIGHT AND RESPOND TO TL1A-DR3 SIGNALING.Rotavirus infection alters peripheral T-cell homeostasis in children with acute diarrhea.Selective targeting of the LIGHT-HVEM costimulatory system for the treatment of graft-versus-host disease.NK-cell activation by LIGHT triggers tumor-specific CD8+ T-cell immunity to reject established tumorsLymphotoxin and LIGHT signaling pathways and target genes.The signaling networks of the herpesvirus entry mediator (TNFRSF14) in immune regulationTumor necrosis factor family members and inflammatory bowel disease.The TNF-family cytokine TL1A drives IL-13-dependent small intestinal inflammation.LIGHTing up dendritic cell activation: Immune regulation and viral exploitation.
P2860
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P2860
Constitutive expression of LIGHT on T cells leads to lymphocyte activation, inflammation, and tissue destruction
description
2001 nî lūn-bûn
@nan
2001 թուականի Դեկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի դեկտեմբերին հրատարակված գիտական հոդված
@hy
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
name
Constitutive expression of LIG ...... mation, and tissue destruction
@ast
Constitutive expression of LIG ...... mation, and tissue destruction
@en
Constitutive expression of LIG ...... mation, and tissue destruction
@nl
type
label
Constitutive expression of LIG ...... mation, and tissue destruction
@ast
Constitutive expression of LIG ...... mation, and tissue destruction
@en
Constitutive expression of LIG ...... mation, and tissue destruction
@nl
prefLabel
Constitutive expression of LIG ...... mation, and tissue destruction
@ast
Constitutive expression of LIG ...... mation, and tissue destruction
@en
Constitutive expression of LIG ...... mation, and tissue destruction
@nl
P2093
P1476
Constitutive expression of LIG ...... mation, and tissue destruction
@en
P2093
H Cheroutre
K Butrovich
M Kronenberg
R B Shaikh
S W Granger
P304
P356
10.4049/JIMMUNOL.167.11.6330
P407
P577
2001-12-01T00:00:00Z