Essential role of TNF family molecule LIGHT as a cytokine in the pathogenesis of hepatitis. - Wikidata - wikimedia - TriplyDB

Essential role of TNF family molecule LIGHT as a cytokine in the pathogenesis of hepatitis.

Essential role of TNF family molecule LIGHT as a cytokine in the pathogenesis of hepatitis.

http://www.wikidata.org/entity/Q34468358

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Critical roles for LIGHT and its receptors in generating T cell-mediated immunity during Leishmania donovani infection Excess LIGHT contributes to placental impairment, increased secretion of vasoactive factors, hypertension, and proteinuria in preeclampsia.Expression patterns and action analysis of genes associated with hepatitis virus infection during rat liver regeneration.Anti-nucleocapsid protein immune responses counteract pathogenic effects of Rift Valley fever virus infection in mice.B7-H1/CD80 interaction is required for the induction and maintenance of peripheral T-cell tolerance Lymphotoxin, NF-ĸB, and cancer: the dark side of cytokines.Multiparametric analyses of human PBMCs loaded ex vivo with a candidate idiotype vaccine for HCV-related lymphoproliferative disorders.LIGHT/TNFSR14 can regulate hepatic lipase expression by hepatocytes independent of T cells and Kupffer cells.A lymphotoxin-driven pathway to hepatocellular carcinoma.Selective targeting of the LIGHT-HVEM costimulatory system for the treatment of graft-versus-host disease.The Tumor Necrosis Factor Superfamily Molecule LIGHT Promotes Keratinocyte Activity and Skin Fibrosis.Tumor necrosis factor superfamily 14 (LIGHT) controls thymic stromal lymphopoietin to drive pulmonary fibrosis.Immunoregulation by tumor necrosis factor superfamily member LIGHT Proinflammatory and prothrombotic effects on human vascular endothelial cells of immune-cell-derived LIGHT.Therapeutic blockade of LIGHT interaction with herpesvirus entry mediator and lymphotoxin β receptor attenuates in vivo cytotoxic allogeneic responses Increased Serum Levels of LIGHT/TNFSF14 in Nonalcoholic Fatty Liver Disease: Possible Role in Hepatic Inflammation.Targeting lymphocyte activation through the lymphotoxin and LIGHT pathways Expression of anti-HVEM single-chain antibody on tumor cells induces tumor-specific immunity with long-term memory.B7-H4-deficient mice display augmented neutrophil-mediated innate immunity.Immunotherapeutic targeting of LIGHT/LTβR/HVEM pathway fully recapitulates the reduced cytotoxic phenotype of LIGHT-deficient T cells.LIGHT/TNFSF14 is increased in patients with type 2 diabetes mellitus and promotes islet cell dysfunction and endothelial cell inflammation in vitro.A TNFRSF14-FcɛRI-mast cell pathway contributes to development of multiple features of asthma pathology in mice.Immune Response in Hepatitis B Virus Infection.Hematopoietic stem/progenitor cell differentiation towards myeloid lineage is modulated by LIGHT/LIGHT receptor signaling.Elevated plasma levels and platelet-associated expression of the pro-thrombotic and pro-inflammatory protein, TNFSF14 (LIGHT), in sickle cell disease.The tumor necrosis factor family member TNFSF14 (LIGHT) is required for resolution of intestinal inflammation in mice.Role of 2B4-mediated signals in the pathogenesis of a murine hepatitis model independent of Fas and Valpha14 NKT cells.Pathological significance and regulatory mechanism of lymphotoxin β receptor overexpression in T cells of patients with systemic lupus erythematosus.LIGHT-HVEM signaling in keratinocytes controls development of dermatitis.

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Essential role of TNF family molecule LIGHT as a cytokine in the pathogenesis of hepatitis.

http://www.wikidata.org/entity/Q34468358

description

2006 nî lūn-bûn

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2006 թուականի Մարտին հրատարակուած գիտական յօդուած

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2006 թվականի մարտին հրատարակված գիտական հոդված

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2006年の論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年論文

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2006年论文

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name

Essential role of TNF family m ...... the pathogenesis of hepatitis.

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Essential role of TNF family m ...... the pathogenesis of hepatitis.

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Essential role of TNF family m ...... the pathogenesis of hepatitis.

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Essential role of TNF family m ...... the pathogenesis of hepatitis.

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Essential role of TNF family m ...... the pathogenesis of hepatitis.

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Essential role of TNF family m ...... the pathogenesis of hepatitis.

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Essential role of TNF family m ...... the pathogenesis of hepatitis.

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Essential role of TNF family m ...... the pathogenesis of hepatitis.

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Journal of Clinical Investigation

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Essential role of TNF family m ...... the pathogenesis of hepatitis.

@en

P2093

Anja Hilliard

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Chyung-Ru Wang

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Dallas B Flies

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Drew M Pardoll

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Gefeng Zhu

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In-Hak Choi

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Kiyoshi Yoshimura

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Koji Tamada

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Lieping Chen

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Richard Schulick

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P2860

A newly identified member of tumor necrosis factor receptor superfamily (TR6) suppresses LIGHT-mediated apoptosis

The lymphotoxin-beta receptor is necessary and sufficient for LIGHT-mediated apoptosis of tumor cells

LIGHT, a new member of the TNF superfamily, and lymphotoxin alpha are ligands for herpesvirus entry mediator

LIGHT, a member of the tumor necrosis factor ligand superfamily, prevents tumor necrosis factor-alpha-mediated human primary hepatocyte apoptosis, but not Fas-mediated apoptosis

Hepatitis C virus core protein interacts with the cytoplasmic tail of lymphotoxin-beta receptor

LIGHT, a novel ligand for lymphotoxin beta receptor and TR2/HVEM induces apoptosis and suppresses in vivo tumor formation via gene transfer

Direct interaction of hepatitis C virus core protein with the cellular lymphotoxin-beta receptor modulates the signal pathway of the lymphotoxin-beta receptor

The regulation of T cell homeostasis and autoimmunity by T cell-derived LIGHT

Reciprocal expression of the TNF family receptor herpes virus entry mediator and its ligand LIGHT on activated T cells: LIGHT down-regulates its own receptor

Genomic characterization of LIGHT reveals linkage to an immune response locus on chromosome 19p13.3 and distinct isoforms generated by alternate splicing or proteolysis

P304

1045-1051

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scholarly article

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10.1172/JCI27083

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4

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116

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Andrew S. Flies

Sudarshan Anand

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2006-03-23T00:00:00Z

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16557300

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1409742

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