Spreading depression and focal brain ischemia induce cyclooxygenase-2 in cortical neurons through N-methyl-D-aspartic acid-receptors and phospholipase A2 - Wikidata - wikimedia - TriplyDB

Spreading depression and focal brain ischemia induce cyclooxygenase-2 in cortical neurons through N-methyl-D-aspartic acid-receptors and phospholipase A2

Spreading depression and focal brain ischemia induce cyclooxygenase-2 in cortical neurons through N-methyl-D-aspartic acid-receptors and phospholipase A2

http://www.wikidata.org/entity/Q28240014

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Cannabinoid activation of PPAR alpha; a novel neuroprotective mechanism Serum deprivation and re-addition: effects on cyclooxygenase inhibitor sensitivity in cultured glia Glucocorticoid treatment in an ischaemic-like excitotoxic model of periventricular leucomalacia in mice.NSAIDs in the Acute Treatment of Migraine: A Review of Clinical and Experimental Data The selective cyclooxygenase-2 inhibitor rofecoxib reduces kainate-induced cell death in the rat hippocampus.Piroxicam and NS-398 rescue neurones from hypoxia/reoxygenation damage by a mechanism independent of cyclo-oxygenase inhibition.Differential effects of ethanol on the expression of cyclo-oxygenase in cultured cortical astrocytes and neurons.Clinically approved heterocyclics act on a mitochondrial target and reduce stroke-induced pathology.Effects of Cyclooxygenase Inhibitors on Apoptotic Neuroretinal Cells.Interaction between inducible nitric oxide synthase and cyclooxygenase-2 after cerebral ischemia Microsomal prostaglandin E synthase-1 and cyclooxygenase-2 are both required for ischaemic excitotoxicity Emerging roles of PGE2 receptors in models of neurological disease.Neuronal and nonneuronal COX-2 expression confers neurotoxic and neuroprotective phenotypes in response to excitotoxin challenge Multiple molecular penumbras after focal cerebral ischemia.Cytosolic phospholipase A2 alpha amplifies early cyclooxygenase-2 expression, oxidative stress and MAP kinase phosphorylation after cerebral ischemia in mice.A hypothesis to explain ganglion cell death caused by vascular insults at the optic nerve head: possible implication for the treatment of glaucoma Reduced susceptibility to ischemic brain injury and N-methyl-D-aspartate-mediated neurotoxicity in cyclooxygenase-2-deficient mice.Microsomal prostaglandin E synthase-1 is a critical factor of stroke-reperfusion injury.Neural controls of prostaglandin 2 pyrogenic, tachycardic, and anorexic actions are anatomically distributed.Central nervous system agents for ischemic stroke: neuroprotection mechanisms Differential effects of treadmill exercise on cyclooxygenase-2 in the rat hippocampus at early and chronic stages of diabetes.Preparation of polylactide-co-glycolide nanoparticles incorporating celecoxib and their antitumor activity against brain tumor cells.Prophylactic, prandial rofecoxib treatment lacks efficacy against acute PTZ-induced seizure generation and kindling acquisition.Pathogenic factors in vascular dementia and Alzheimer's disease. Multiple actions of heparin that probably are beneficial.Magnesium sulfate and nimesulide have synergistic effects on rescuing brain damage after transient focal ischemia.Contribution of prostanoid signaling to the evolution of spreading depolarization and the associated cerebral blood flow response.Investigational anti-inflammatory agents for the treatment of ischaemic brain injury.Focus on therapy of primary stabbing headache.The role of antioxidants in models of inflammation: emphasis on L-arginine and arachidonic acid metabolism.Bilateral common carotid artery ligation transiently changes brain lipid metabolism in rats.Interferon regulatory factor (IRF)-1 and IRF-2 regulate interferon gamma-dependent cyclooxygenase 2 expression.Lipid signaling and synaptic plasticity.A tetracycline derivative, minocycline, reduces inflammation and protects against focal cerebral ischemia with a wide therapeutic window Cyclooxygenase-2 in synaptic signaling.Prostaglandin E2 depresses solitary tract-mediated synaptic transmission in the nucleus tractus solitarius.Antiinflammatory and neuroprotective actions of COX2 inhibitors in the injured brain.Mechanisms involved in the cerebrovascular dilator effects of cortical spreading depression CSD, BBB and MMP-9 elevations: animal experiments versus clinical phenomena in migraine.Significance of brain tissue oxygenation and the arachidonic acid cascade in stroke.Inflammation after stroke: mechanisms and therapeutic approaches.

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P2860

Spreading depression and focal brain ischemia induce cyclooxygenase-2 in cortical neurons through N-methyl-D-aspartic acid-receptors and phospholipase A2

http://www.wikidata.org/entity/Q28240014

description

1997 nî lūn-bûn

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1997年の論文

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Physical association and functional antagonism between the p65 subunit of transcription factor NF-kappa B and the glucocorticoid receptor

Characterization of inducible cyclooxygenase in rat brain.

The biochemical basis of cerebral ischemic damage.

Sustained induction of prostaglandin endoperoxide synthase-2 by seizures in hippocampus. Inhibition by a platelet-activating factor antagonist.

Prostaglandins, aspirin-like drugs and analgesia.

Evidence for sensory nerve involvement in cutaneous reactive hyperemia in humans.

Transcriptional roles of nuclear factor kappa B and nuclear factor-interleukin-6 in the tumor necrosis factor alpha-dependent induction of cyclooxygenase-2 in MC3T3-E1 cells.

Effect of anoxia on ion distribution in the brain.

Characterization of mechanisms involved in transrepression of NF-kappa B by activated glucocorticoid receptors.

Inhibition of phospholipase.

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