Inflammation after stroke: mechanisms and therapeutic approaches. - Wikidata - wikimedia - TriplyDB

Inflammation after stroke: mechanisms and therapeutic approaches.

Inflammation after stroke: mechanisms and therapeutic approaches.

http://www.wikidata.org/entity/Q37803131

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Fingolimod for multiple sclerosis and emerging indications: appropriate patient selection, safety precautions, and special considerations Neutralization of chemokine-like factor 1, a novel C-C chemokine, protects against focal cerebral ischemia by inhibiting neutrophil infiltration via MAPK pathways in rats.Proteomic discovery and verification of serum amyloid A as a predictor marker of patients at risk of post-stroke infection: a pilot study.Impact of an immune modulator fingolimod on acute ischemic stroke.Prolonged exposure to isoflurane ameliorates infarction severity in the rat pup model of neonatal hypoxia-ischemia Recombinant T cell receptor ligands improve outcome after experimental cerebral ischemia.Inhibition of protein kinase Cβ reverses increased blood-brain barrier permeability during hyperglycemic stroke and prevents edema formation in vivo.Vascular Protection Following Cerebral Ischemia and Reperfusion.Combination of the Immune Modulator Fingolimod With Alteplase in Acute Ischemic Stroke: A Pilot Trial.Inhibition of immunoproteasome reduces infarction volume and attenuates inflammatory reaction in a rat model of ischemic stroke.Fast neuroprotection (fast-NPRX) for acute ischemic stroke victims: the time for treatment is now.Cerebral ischemic damage in diabetes: an inflammatory perspective.E2-25K SUMOylation inhibits proteasome for cell death during cerebral ischemia/reperfusion Mesenchymal stem cells secretome: a new paradigm for central nervous system regeneration?Nitric oxide donors as neuroprotective agents after an ischemic stroke-related inflammatory reaction Valproic acid: a new candidate of therapeutic application for the acute central nervous system injuries.PKC-β exacerbates in vitro brain barrier damage in hyperglycemic settings via regulation of RhoA/Rho-kinase/MLC2 pathway Tissue kallikrein protects against ischemic stroke by suppressing TLR4/NF-κB and activating Nrf2 signaling pathway in rats Anti-inflammatory effects of Chinese medicinal herbs on cerebral ischemia.Association Between Splenic Contraction and the Systemic Inflammatory Response After Acute Ischemic Stroke Varies with Age and Race.MiR-126 Affects Brain-Heart Interaction after Cerebral Ischemic Stroke.Using oxidant and antioxidant levels to predict the duration of both acute peripheral and mesenteric ischemia.

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Inflammation after stroke: mechanisms and therapeutic approaches.

http://www.wikidata.org/entity/Q37803131

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on June 2010

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Inflammation after stroke: mechanisms and therapeutic approaches.

@en

Inflammation after stroke: mechanisms and therapeutic approaches.

@nl

type

label

Inflammation after stroke: mechanisms and therapeutic approaches.

@en

Inflammation after stroke: mechanisms and therapeutic approaches.

@nl

prefLabel

Inflammation after stroke: mechanisms and therapeutic approaches.

@en

Inflammation after stroke: mechanisms and therapeutic approaches.

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S12975-010-0023-7

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Translational stroke research

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Inflammation after stroke: mechanisms and therapeutic approaches.

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Muzamil Ahmad

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Steven H Graham

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P2860

Identification and molecular characterization of fractalkine receptor CX3CR1, which mediates both leukocyte migration and adhesion

Peak plasma interleukin-6 and other peripheral markers of inflammation in the first week of ischaemic stroke correlate with brain infarct volume, stroke severity and long-term outcome

Characterization of fractalkine in rat brain cells: migratory and activation signals for CX3CR-1-expressing microglia

Unfractionated heparin is associated with a lower rise of serum vascular cell adhesion molecule-1 in acute ischemic stroke patients

Inflammatory cell adhesion molecules in ischemic cerebrovascular disease

Spreading depression and focal brain ischemia induce cyclooxygenase-2 in cortical neurons through N-methyl-D-aspartic acid-receptors and phospholipase A2

Neurotactin, a membrane-anchored chemokine upregulated in brain inflammation

Identification of CX3CR1. A chemotactic receptor for the human CX3C chemokine fractalkine and a fusion coreceptor for HIV-1

A new class of membrane-bound chemokine with a CX3C motif

Role for neuronally derived fractalkine in mediating interactions between neurons and CX3CR1-expressing microglia

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s12975-010-0023-7

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scholarly article

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10.1007/S12975-010-0023-7

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