Induction of IRAK-M is associated with lipopolysaccharide tolerance in a human endotoxemia model
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Endotoxin tolerance impairs IL-1 receptor-associated kinase (IRAK) 4 and TGF-beta-activated kinase 1 activation, K63-linked polyubiquitination and assembly of IRAK1, TNF receptor-associated factor 6, and IkappaB kinase gamma and increases A20 expresTolerance and Cross-Tolerance following Toll-Like Receptor (TLR)-4 and -9 Activation Are Mediated by IRAK-M and Modulated by IL-7 in Murine SplenocytesIRAK-M expression limits dendritic cell activation and proinflammatory cytokine production in response to Helicobacter pyloriTopical treatment of melanoma metastases with imiquimod, plus administration of a cancer vaccine, promotes immune signatures in the metastases.Rescue from lethal Shiga toxin 2-induced renal failure with a cell-permeable peptide.Soluble ST2 levels are associated with bleeding in patients with severe Leptospirosis.Induction and termination of inflammatory signaling in group B streptococcal sepsis.Regulation of mammalian siderophore 2,5-DHBA in the innate immune response to infectionPathophysiology of endotoxin tolerance: mechanisms and clinical consequences.Elevation in body temperature to fever range enhances and prolongs subsequent responsiveness of macrophages to endotoxin challengeInterleukin-1 receptor-associated kinase-3 is a key inhibitor of inflammation in obesity and metabolic syndrome.Neutrophils activate alveolar macrophages by producing caspase-6-mediated cleavage of IL-1 receptor-associated kinase-MDaily exposure to dust alters innate immunityA Toll-like receptor-responsive kinase, protein kinase R, is inactivated in endotoxin tolerance through differential K63/K48 ubiquitination.STAT3 regulates monocyte TNF-alpha production in systemic inflammation caused by cardiac surgery with cardiopulmonary bypass.Network topologies and dynamics leading to endotoxin tolerance and priming in innate immune cells.Toll-like receptor tolerance as a mechanism for neuroprotection.TGF-β-induced IRAK-M expression in tumor-associated macrophages regulates lung tumor growth.Glucocorticoids suppress inflammation via the upregulation of negative regulator IRAK-M.Role of tumor necrosis factor-α in the human systemic endotoxin-induced transcriptome.A20 is critical for the induction of Pam3CSK4-tolerance in monocytic THP-1 cellsExpression of inhibitory regulators of innate immunity in patients with active tuberculosis.Contrasting ocular effects of local versus systemic endotoxin.A common haplotype of the TNF receptor 2 gene modulates endotoxin tolerance.Negative regulation of Toll-like receptor signaling plays an essential role in homeostasis of the intestine.Transvenous vagus nerve stimulation does not modulate the innate immune response during experimental human endotoxemia: a randomized controlled studyUpregulated Expression of A20 on Monocytes is Associated With Increased Severity of Acute-on-Chronic Hepatitis B Liver Failure: A Case-Control Study.Pellino-3 promotes endotoxin tolerance and acts as a negative regulator of TLR2 and TLR4 signaling.Naturally Occurring Subclinical Endotoxemia in Humans Alters Adaptive and Innate Immune Functions through Reduced MAPK and Increased STAT1 PhosphorylationEndothelial cell tolerance to lipopolysaccharide challenge is induced by monophosphoryl lipid A.IRAK-M modulates expression of IL-10 and cell surface markers CD80 and MHC II after bacterial re-stimulation of tolerized dendritic cellsAcute pulmonary lipopolysaccharide tolerance decreases TNF-alpha without reducing neutrophil recruitment.Inflammasome and toll-like receptor signaling in human monocytes after successful cardiopulmonary resuscitation.IRAK4 kinase activity is not required for induction of endotoxin tolerance but contributes to TLR2-mediated tolerancep21 mediates macrophage reprogramming through regulation of p50-p50 NF-κB and IFN-βPattern-recognition receptor signaling regulator mRNA expression in humans and mice, and in transient inflammation or progressive fibrosisToll-like receptors in skin infections and inflammatory diseases.The opposite effects of acute and chronic alcohol on lipopolysaccharide-induced inflammation are linked to IRAK-M in human monocytes.Molecular regulation of hepatic dendritic cell function and its relation to liver transplant outcome.Macrophage phenotype controls long-term AKI outcomes--kidney regeneration versus atrophy
P2860
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P2860
Induction of IRAK-M is associated with lipopolysaccharide tolerance in a human endotoxemia model
description
2007 nî lūn-bûn
@nan
2007 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2007 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
2007年の論文
@ja
2007年論文
@yue
2007年論文
@zh-hant
2007年論文
@zh-hk
2007年論文
@zh-mo
2007年論文
@zh-tw
2007年论文
@wuu
name
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@ast
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@en
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@nl
type
label
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@ast
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@en
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@nl
prefLabel
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@ast
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@en
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@nl
P2093
P3181
P1476
Induction of IRAK-M is associa ...... e in a human endotoxemia model
@en
P2093
Alex F de Vos
Cornelis van 't Veer
Eric S Stroes
Marieke A D van Zoelen
Martijn de Kruif
Petra S van den Pangaart
Rakesh S Birjmohun
P304
P3181
P356
10.4049/JIMMUNOL.179.10.7110
P407
P577
2007-11-15T00:00:00Z