Identification of pendrin as a common mediator for mucus production in bronchial asthma and chronic obstructive pulmonary disease
about
Genes associated with MUC5AC expression in small airway epithelium of human smokers and non-smokersMolecular insights into the progression of crystalline silica-induced pulmonary toxicity in ratsThe epithelial anion transporter pendrin is induced by allergy and rhinovirus infection, regulates airway surface liquid, and increases airway reactivity and inflammation in an asthma modelEpithelial Anion Transport as Modulator of Chemokine SignalingNovel Roles for Chloride Channels, Exchangers, and Regulators in Chronic Inflammatory Airway DiseasesEpithelial Sodium and Chloride Channels and AsthmaNovel therapies for the treatment of pertussis diseasePulmonary response to surface-coated nanotitanium dioxide particles includes induction of acute phase response genes, inflammatory cascades, and changes in microRNAs: a toxicogenomic studyDifferences in the pathogenicity of the p.H723R mutation of the common deafness-associated SLC26A4 gene in humans and miceMolecular mechanisms of pulmonary response progression in crystalline silica exposed ratsDo mutations of the Pendred syndrome gene, SLC26A4, confer resistance to asthma and hypertension?Integration of human and mouse genetics reveals pendrin function in hearing and deafnessTranscriptional regulation of the pendrin gene.Impact of female cigarette smoking on circulating B cells in vivo: the suppressed ICOSLG, TCF3, and VCAM1 gene functional network may inhibit normal cell function.Epithelial anion transporter pendrin contributes to inflammatory lung pathology in mouse models of Bordetella pertussis infectionModeling Pulmonary Disease Pathways Using Recombinant Adeno-Associated Virus 6.2.IL-17A induces Pendrin expression and chloride-bicarbonate exchange in human bronchial epithelial cellsMouse models for pendrin-associated loss of cochlear and vestibular functionThe molecular fingerprint of lung inflammation after blunt chest traumaIncreased expression of the epithelial anion transporter pendrin/SLC26A4 in nasal polyps of patients with chronic rhinosinusitisPertussis toxin exacerbates and prolongs airway inflammatory responses during Bordetella pertussis infection.Hypoxia-induced mitogenic factor (HIMF/FIZZ1/RELMα) in chronic hypoxia- and antigen-mediated pulmonary vascular remodeling.The SLC26 gene family of anion transporters and channels.Pendrin, an anion exchanger on lung epithelial cells, could be a novel target for lipopolysaccharide-induced acute lung injury mice.Inhibitors of pendrin anion exchange identified in a small molecule screen increase airway surface liquid volume in cystic fibrosis.Functional characterization of pendrin mutations found in the Israeli and Palestinian populations.Pendrin function and regulation in Xenopus oocytesIdentification of allelic variants of pendrin (SLC26A4) with loss and gain of function.Airway and lung pathology due to mucosal surface dehydration in {beta}-epithelial Na+ channel-overexpressing mice: role of TNF-{alpha} and IL-4R{alpha} signaling, influence of neonatal development, and limited efficacy of glucocorticoid treatmentBronchial airway gene expression in smokers with lung or head and neck cancer.Role of transcription factors in the pathogenesis of asthma and COPD.Pendrin, a novel transcriptional target of the uroguanylin system.Bicarbonate transport in health and disease.Interleukin-13 increases pendrin abundance to the cell surface in bronchial NCI-H292 cells via Rho/actin signaling.Role of anion exchangers in Cl- and HCO3- secretion by the human airway epithelial cell line Calu-3.Novel role for pendrin in orchestrating bicarbonate secretion in cystic fibrosis transmembrane conductance regulator (CFTR)-expressing airway serous cells.STAT6 links IL-4/IL-13 stimulation with pendrin expression in asthma and chronic obstructive pulmonary disease.Thick airway surface liquid volume and weak mucin expression in pendrin-deficient human airway epithelia.Concentration of the antibacterial precursor thiocyanate in cystic fibrosis airway secretions.IL-1β expression in the distal lung epithelium disrupts lung morphogenesis and epithelial cell differentiation in fetal mice.
P2860
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P2860
Identification of pendrin as a common mediator for mucus production in bronchial asthma and chronic obstructive pulmonary disease
description
2008 nî lūn-bûn
@nan
2008 թուականի Մայիսին հրատարակուած գիտական յօդուած
@hyw
2008 թվականի մայիսին հրատարակված գիտական հոդված
@hy
2008年の論文
@ja
2008年論文
@yue
2008年論文
@zh-hant
2008年論文
@zh-hk
2008年論文
@zh-mo
2008年論文
@zh-tw
2008年论文
@wuu
name
Identification of pendrin as a ...... obstructive pulmonary disease
@ast
Identification of pendrin as a ...... obstructive pulmonary disease
@en
Identification of pendrin as a ...... obstructive pulmonary disease
@nl
type
label
Identification of pendrin as a ...... obstructive pulmonary disease
@ast
Identification of pendrin as a ...... obstructive pulmonary disease
@en
Identification of pendrin as a ...... obstructive pulmonary disease
@nl
prefLabel
Identification of pendrin as a ...... obstructive pulmonary disease
@ast
Identification of pendrin as a ...... obstructive pulmonary disease
@en
Identification of pendrin as a ...... obstructive pulmonary disease
@nl
P2093
P3181
P1476
Identification of pendrin as a ...... obstructive pulmonary disease
@en
P2093
Aya Shiraki
Hidetomo Matsushita
Hiroaki Hayashi
Hiroichi Nagai
Hiromasa Inoue
Hironori Sagara
Hiroshi Kubo
Hiroyuki Tanaka
Hisamichi Aizawa
Isao Nakao
P304
P3181
P356
10.4049/JIMMUNOL.180.9.6262
P407
P577
2008-05-01T00:00:00Z