Aurora B overexpression associates with the thyroid carcinoma undifferentiated phenotype and is required for thyroid carcinoma cell proliferation - Wikidata - wikimedia - TriplyDB

Aurora B overexpression associates with the thyroid carcinoma undifferentiated phenotype and is required for thyroid carcinoma cell proliferation

Aurora B overexpression associates with the thyroid carcinoma undifferentiated phenotype and is required for thyroid carcinoma cell proliferation

http://www.wikidata.org/entity/Q28294945

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Aurora B expression correlates with aggressive behaviour in glioblastoma multiforme Aurora Kinase Inhibitors: Current Status and Outlook Aurora Kinases and Potential Medical Applications of Aurora Kinase Inhibitors: A Review Optimization of Imidazo[4,5- b ]pyridine-Based Kinase Inhibitors: Identification of a Dual FLT3/Aurora Kinase Inhibitor as an Orally Bioavailable Preclinical Development Candidate for the Treatment of Acute Myeloid Leukemia Aurora isoform selectivity: design and synthesis of imidazo[4,5-b]pyridine derivatives as highly selective inhibitors of Aurora-A kinase in cells.A novel histone deacetylase pathway regulates mitosis by modulating Aurora B kinase activity.Modulation of key regulators of mitosis linked to chromosomal instability is an early event in ochratoxin A carcinogenicity Aurora kinases in head and neck cancer Phosphatase inhibitor-2 balances protein phosphatase 1 and aurora B kinase for chromosome segregation and cytokinesis in human retinal epithelial cells.Protein tyrosine phosphatase Shp2 (Ptpn11) plays an important role in maintenance of chromosome stability Significance of Aurora B overexpression in hepatocellular carcinoma. Aurora B Overexpression in HCC.Update: the status of clinical trials with kinase inhibitors in thyroid cancer Anaplastic thyroid cancer: molecular pathogenesis and emerging therapies.Spindle proteins are differentially expressed in the various histological subtypes of testicular germ cell tumors.A new aurora in anaplastic thyroid cancer therapy.Inhibition of Aurora-B suppresses HepG2 cell invasion and migration via the PI3K/Akt/NF-κB signaling pathway in vitro The ARF tumor suppressor prevents chromosomal instability and ensures mitotic checkpoint fidelity through regulation of Aurora B.Understanding cytokinesis failure.Deregulated expression of Aurora kinases is not a prognostic biomarker in papillary thyroid cancer patients.A phase 1 dose escalation study of BI 831266, an inhibitor of Aurora kinase B, in patients with advanced solid tumors Knockdown of Aurora-B inhibits the growth of non-small cell lung cancer A549 cells.Transcriptional activation of human CDCA8 gene regulated by transcription factor NF-Y in embryonic stem cells and cancer cells Reversine Induced Multinucleated Cells, Cell Apoptosis and Autophagy in Human Non-Small Cell Lung Cancer Cells.Aurora B Overexpression Causes Aneuploidy and p21Cip1 Repression during Tumor Development.Tetraploidy/aneuploidy and stem cells in cancer promotion: The role of chromosome passenger proteins.Aurora A and B kinases as targets for cancer: will they be selective for tumors?Targeting Aurora kinases in ovarian cancer.Ceftriaxone, an FDA-approved cephalosporin antibiotic, suppresses lung cancer growth by targeting Aurora B.Poorly differentiated and anaplastic thyroid carcinomas: chromosomal and oligo-array profile of five new cell lines.Overexpression of aurora B kinase (AURKB) in primary non-small cell lung carcinoma is frequent, generally driven from one allele, and correlates with the level of genetic instability.FOXM1 is a molecular determinant of the mitogenic and invasive phenotype of anaplastic thyroid carcinoma A phase I study of BI 811283, an Aurora B kinase inhibitor, in patients with advanced solid tumors.Pazopanib enhances paclitaxel-induced mitotic catastrophe in anaplastic thyroid cancer Aurora kinase inhibitors: progress towards the clinic Mitotic failures in cancer: Aurora B kinase and its potential role in the development of aneuploidy.Regulation of chromatin structure via histone post-translational modification and the link to carcinogenesis.Mitosis as an anti-cancer drug target.Genetics and epigenetics of sporadic thyroid cancer.Targeting mitotic pathways for endocrine-related cancer therapeutics.Histone modifying enzymes: novel disease biomarkers and assay development.

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Aurora B overexpression associates with the thyroid carcinoma undifferentiated phenotype and is required for thyroid carcinoma cell proliferation

http://www.wikidata.org/entity/Q28294945

description

2005 nî lūn-bûn

@nan

2005 թուականի Փետրուարին հրատարակուած գիտական յօդուած

@hyw

2005 թվականի փետրվարին հրատարակված գիտական հոդված

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2005年の論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年論文

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2005年论文

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name

Aurora B overexpression associ ...... d carcinoma cell proliferation

@ast

Aurora B overexpression associ ...... d carcinoma cell proliferation

@en

Aurora B overexpression associ ...... d carcinoma cell proliferation

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type

label

Aurora B overexpression associ ...... d carcinoma cell proliferation

@ast

Aurora B overexpression associ ...... d carcinoma cell proliferation

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Aurora B overexpression associ ...... d carcinoma cell proliferation

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prefLabel

Aurora B overexpression associ ...... d carcinoma cell proliferation

@ast

Aurora B overexpression associ ...... d carcinoma cell proliferation

@en

Aurora B overexpression associ ...... d carcinoma cell proliferation

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The Journal of Clinical Endocrinology and Metabolism

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Aurora B overexpression associ ...... d carcinoma cell proliferation

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P2093

Daniela Spalletti-Cernia

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Giancarlo Troncone

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Giuseppe Portella

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Lucio Palombini

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Paolo Laccetti

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Pier Lorenzo Pallante

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Rosanna Sorrentino

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Silvana Libertini

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Vassilios Bavetsias

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scholarly article

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10.1210/JC.2004-1518

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2

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90

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Spiros Linardopoulos

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2004-11-23T00:00:00Z

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8165364

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15562011

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