Peroxiredoxin-3 is overexpressed in prostate cancer and promotes cancer cell survival by protecting cells from oxidative stress
about
Orphan proteins of unknown function in the mitochondrial intermembrane space proteome: New pathways and metabolic cross-talkRedox Homeostasis and Cellular Antioxidant Systems: Crucial Players in Cancer Growth and TherapyPeroxiredoxins and the Regulation of Cell DeathThe Tumorigenic Roles of the Cellular REDOX Regulatory SystemsRedox Regulation in Cancer Stem CellsHigh-Protein Intake during Weight Loss Therapy Eliminates the Weight-Loss-Induced Improvement in Insulin Action in Obese Postmenopausal WomenTargeting multiple pro-apoptotic signaling pathways with curcumin in prostate cancer cellsMetallothionein-I/II Knockout Mice Aggravate Mitochondrial Superoxide Production and Peroxiredoxin 3 Expression in Thyroid after Excessive Iodide Exposure.Overexpression of Prdx1 in hilar cholangiocarcinoma: a predictor for recurrence and prognosis.Anti-cancer effect of snake venom toxin through down regulation of AP-1 mediated PRDX6 expressionSelenium- or Vitamin E-Related Gene Variants, Interaction with Supplementation, and Risk of High-Grade Prostate Cancer in SELECT.Epigenetic regulation of peroxiredoxins: Implications in the pathogenesis of cancer.Hitting the Bull's-Eye in Metastatic Cancers-NSAIDs Elevate ROS in Mitochondria, Inducing Malignant Cell Death.The functional role of peroxiredoxin 3 in reactive oxygen species, apoptosis, and chemoresistance of cancer cells.Mitochondrial ROS control of cancer.New Challenges to Study Heterogeneity in Cancer Redox Metabolism.microRNA-383 mediates high glucose-induced oxidative stress and apoptosis in retinal pigment epithelial cells by repressing peroxiredoxin 3.Oxidative stress and altered expression of peroxiredoxin genes family (PRDXS) and sulfiredoxin-1 (SRXN1) in human lung tissue following exposure to sulfur mustard.Mitochondrion-associated protein peroxiredoxin 3 promotes benign prostatic hyperplasia through autophagy suppression and pyroptosis activation.Narrower insight to SIRT1 role in cancer: A potential therapeutic target to control epithelial-mesenchymal transition in cancer cells.Circadian and Metabolic Perspectives in the Role Played by NADPH in Cancer.Overexpression of peroxiredoxin-3 and -5 is a potential biomarker for prognosis in endometrial cancer.GPx3-mediated redox signaling arrests the cell cycle and acts as a tumor suppressor in lung cancer cell lines
P2860
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P2860
Peroxiredoxin-3 is overexpressed in prostate cancer and promotes cancer cell survival by protecting cells from oxidative stress
description
2013 nî lūn-bûn
@nan
2013 թուականի Օգոստոսին հրատարակուած գիտական յօդուած
@hyw
2013 թվականի օգոստոսին հրատարակված գիտական հոդված
@hy
2013年の論文
@ja
2013年論文
@yue
2013年論文
@zh-hant
2013年論文
@zh-hk
2013年論文
@zh-mo
2013年論文
@zh-tw
2013年论文
@wuu
name
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@ast
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@en
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@nl
type
label
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@ast
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@en
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@nl
prefLabel
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@ast
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@en
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@nl
P2093
P2860
P3181
P356
P1476
Peroxiredoxin-3 is overexpress ...... ng cells from oxidative stress
@en
P2093
A Y Warren
H C Whitaker
H J Luxton
J C Marioni
J Mitchell
S Aldridge
P2860
P2888
P304
P3181
P356
10.1038/BJC.2013.396
P407
P577
2013-08-20T00:00:00Z
P5875
P6179
1020748921