Impact of TLR4 on behavioral and cognitive dysfunctions associated with alcohol-induced neuroinflammatory damage - Wikidata - wikimedia - TriplyDB

Impact of TLR4 on behavioral and cognitive dysfunctions associated with alcohol-induced neuroinflammatory damage

Impact of TLR4 on behavioral and cognitive dysfunctions associated with alcohol-induced neuroinflammatory damage

http://www.wikidata.org/entity/Q28306144

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Neuroimmune Function and the Consequences of Alcohol Exposure Behavioral Neuroadaptation to Alcohol: From Glucocorticoids to Histone Acetylation Mechanisms of neuroimmune gene induction in alcoholism Glucuronic acid and the ethanol metabolite ethyl-glucuronide cause toll-like receptor 4 activation and enhanced pain Protection of Gastrointestinal Mucosa from Acute Heavy Alcohol Consumption: The Effect of Berberine and Its Correlation with TLR2, 4/IL1β-TNFα Signaling TLR2 and TLR4 Expression and Inflammatory Cytokines are Altered in the Airway Epithelium of Those with Alcohol Use Disorders Interactions between inflammation, sex steroids, and Alzheimer's disease risk factors CRF-amplified neuronal TLR4/MCP-1 signaling regulates alcohol self-administration.Exposure of neonatal rats to alcohol has differential effects on neuroinflammation and neuronal survival in the cerebellum and hippocampus.Effects of Intermittent Alcohol Exposure on Emotion and Cognition: A Potential Role for the Endogenous Cannabinoid System and Neuroinflammation.Toll-like receptor signaling and stages of addiction.Electroacupuncture Alleviates Postoperative Cognitive Dysfunction in Aged Rats by Inhibiting Hippocampal Neuroinflammation Activated via Microglia/TLRs Pathway.Innate immune factors modulate ethanol interaction with GABAergic transmission in mouse central amygdala.Molecular and behavioral aspects of the actions of alcohol on the adult and developing brain.Toll-like receptor 4 participates in the myelin disruptions associated with chronic alcohol abuse.Temporal changes in innate immune signals in a rat model of alcohol withdrawal in emotional and cardiorespiratory homeostatic nuclei.Activation of adult rat CNS endothelial cells by opioid-induced toll-like receptor 4 (TLR4) signaling induces proinflammatory, biochemical, morphological, and behavioral sequelae Neuroimmune pathways in alcohol consumption: evidence from behavioral and genetic studies in rodents and humans Release of neuronal HMGB1 by ethanol through decreased HDAC activity activates brain neuroimmune signaling.The epigenetic landscape of alcoholism.Fetal alcohol spectrum disorders and neuroimmune changes Alcohol-induced sedation and synergistic interactions between alcohol and morphine: a key mechanistic role for Toll-like receptors and MyD88-dependent signaling.Neurotransmitter receptors and cognitive dysfunction in Alzheimer's disease and Parkinson's disease.TLR4-MyD88 signalling: a molecular target for alcohol actions Inhibiting the TLR4-MyD88 signalling cascade by genetic or pharmacological strategies reduces acute alcohol-induced sedation and motor impairment in mice Neuroimmune mechanisms in fetal alcohol spectrum disorder Neuroinflammatory pathways in binge alcohol-induced neuronal degeneration: oxidative stress cascade involving aquaporin, brain edema, and phospholipase A2 activation.Adolescent intermittent ethanol exposure enhances ethanol activation of the nucleus accumbens while blunting the prefrontal cortex responses in adult rat.Persistent adaptation by chronic alcohol is facilitated by neuroimmune activation linked to stress and CRF Substance use disorders: psychoneuroimmunological mechanisms and new targets for therapy.Docosahexaenoic acid (DHA) prevents binge ethanol-dependent aquaporin-4 elevations while inhibiting neurodegeneration: experiments in rat adult-age entorhino-hippocampal slice cultures Adolescent binge-like ethanol exposure reduces basal α-MSH expression in the hypothalamus and the amygdala of adult rats.Environmentally induced epigenetic toxicity: potential public health concerns.The cytokine mRNA increase induced by withdrawal from chronic ethanol in the sterile environment of brain is mediated by CRF and HMGB1 release.TLR4 signaling in VTA dopaminergic neurons regulates impulsivity through tyrosine hydroxylase modulation.Immune function genes, genetics, and the neurobiology of addiction.TLR4-dependent metabolic changes are associated with cognitive impairment in an animal model of type 1 diabetes.Genes and Alcohol Consumption: Studies with Mutant Mice.White matter integrity is associated with alcohol cue reactivity in heavy drinkers Neuroimmune signaling: a key component of alcohol abuse.

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P2860

Impact of TLR4 on behavioral and cognitive dysfunctions associated with alcohol-induced neuroinflammatory damage

http://www.wikidata.org/entity/Q28306144

description

2011 nî lūn-bûn

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2011 թուականի Յունիսին հրատարակուած գիտական յօդուած

@hyw

2011 թվականի հունիսին հրատարակված գիտական հոդված

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2011年の論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年論文

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2011年论文

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name

Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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J.BBI.2011.02.012

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Brain, Behavior, and Immunity

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Impact of TLR4 on behavioral a ...... duced neuroinflammatory damage

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Carlos M G Aragón

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Consuelo Guerri

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María Pascual

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Pablo Baliño

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Silvia Alfonso-Loeches

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S80-91

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scholarly article

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3103840

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10.1016/J.BBI.2011.02.012

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25 Suppl 1

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2011-06-01T00:00:00Z

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50212702

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21352907

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Toll-like receptor