The endoplasmic reticulum is a glucose-modulated high-affinity sink for Ca2+ in mouse pancreatic beta-cells
about
The central role of calcium in the effects of cytokines on beta-cell function: implications for type 1 and type 2 diabetesR-type Ca(2+)-channel-evoked CICR regulates glucose-induced somatostatin secretionStress-induced dissociations between intracellular calcium signaling and insulin secretion in pancreatic isletsEvidence of diminished glucose stimulation and endoplasmic reticulum function in nonoscillatory pancreatic islets.Bax inhibitor 1 increases cell adhesion through actin polymerization: involvement of calcium and actin binding.cAMP induces stromal interaction molecule 1 (STIM1) puncta but neither Orai1 protein clustering nor store-operated Ca2+ entry (SOCE) in islet cells.The Ca2+ dynamics of isolated mouse beta-cells and islets: implications for mathematical models.Filtering of calcium transients by the endoplasmic reticulum in pancreatic beta-cellsIonic mechanisms and Ca2+ dynamics underlying the glucose response of pancreatic β cells: a simulation study.Mechanisms of control of the free Ca2+ concentration in the endoplasmic reticulum of mouse pancreatic β-cells: interplay with cell metabolism and [Ca2+]c and role of SERCA2b and SERCA3.Glucose-Dependent Insulin Secretion in Pancreatic β-Cell Islets from Male Rats Requires Ca2+ Release via ROS-Stimulated Ryanodine Receptors.Glucose Evokes Rapid Ca2+ and Cyclic AMP Signals by Activating the Cell-Surface Glucose-Sensing Receptor in Pancreatic β-Cells.Bursting and calcium oscillations in pancreatic beta-cells: specific pacemakers for specific mechanisms.Calcium-activated K+ channels of mouse beta-cells are controlled by both store and cytoplasmic Ca2+: experimental and theoretical studies.Glucose-induced inhibition of insulin secretion.An emerging role for NAADP-mediated Ca2+ signaling in the pancreatic β-cell.PERK activation at low glucose concentration is mediated by SERCA pump inhibition and confers preemptive cytoprotection to pancreatic β-cellsInsulin secretion from beta-cells is affected by deletion of nicotinamide nucleotide transhydrogenase.The beneficial effect of growth hormone treatment on islet mass in streptozotocin-treated mice.TALK-1 channels control β cell endoplasmic reticulum Ca2+ homeostasis.Ligand-induced recruitment of Na+/H+-exchanger regulatory factor to the PDGF (platelet-derived growth factor) receptor regulates actin cytoskeleton reorganization by PDGF.Modeling of Ca2+ flux in pancreatic beta-cells: role of the plasma membrane and intracellular stores.Role of the glucose-sensing receptor in insulin secretion.
P2860
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P2860
The endoplasmic reticulum is a glucose-modulated high-affinity sink for Ca2+ in mouse pancreatic beta-cells
description
2001 nî lūn-bûn
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2001 թուականի Փետրուարին հրատարակուած գիտական յօդուած
@hyw
2001 թվականի փետրվարին հրատարակված գիտական հոդված
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2001年の論文
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2001年論文
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2001年論文
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2001年論文
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2001年論文
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2001年論文
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2001年论文
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The endoplasmic reticulum is a ...... in mouse pancreatic beta-cells
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The endoplasmic reticulum is a ...... in mouse pancreatic beta-cells
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The endoplasmic reticulum is a ...... in mouse pancreatic beta-cells
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The endoplasmic reticulum is a ...... in mouse pancreatic beta-cells
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The endoplasmic reticulum is a ...... in mouse pancreatic beta-cells
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The endoplasmic reticulum is a ...... in mouse pancreatic beta-cells
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The endoplasmic reticulum is a ...... in mouse pancreatic beta-cells
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P2860
P1476
The endoplasmic reticulum is a ...... in mouse pancreatic beta-cells
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P2093
P2860
P304
P356
10.1111/J.1469-7793.2001.0533K.X
P407
P50
P577
2001-02-01T00:00:00Z