Severe renal mass reduction impairs recovery and promotes fibrosis after AKI - Wikidata - wikimedia - TriplyDB

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

http://www.wikidata.org/entity/Q28388800

about

Hypoxia: The Force that Drives Chronic Kidney Disease Acute kidney injury: preclinical innovations, challenges, and opportunities for translation Progression after AKI: Understanding Maladaptive Repair Processes to Predict and Identify Therapeutic Treatments.Th-17 cell activation in response to high salt following acute kidney injury is associated with progressive fibrosis and attenuated by AT-1R antagonism.Elevated BP after AKI.Losartan reduces ensuing chronic kidney disease and mortality after acute kidney injury.Renal disease pathophysiology and treatment: contributions from the rat.Tubular Peroxiredoxin 3 as a Predictor of Renal Recovery from Acute Tubular Necrosis in Patients with Chronic Kidney Disease.Hypoxia as a key player in the AKI-to-CKD transition.Failed Tubule Recovery, AKI-CKD Transition, and Kidney Disease Progression.Stop adding insult to injury-identifying and managing risk factors for the progression of acute kidney injury in children.Hypoxia and Dysregulated Angiogenesis in Kidney Disease.Progression of Chronic Kidney Disease After Acute Kidney Injury: Role of Self-Perpetuating Versus Hemodynamic-Induced Fibrosis αKlotho and Chronic Kidney Disease.Mitochondrial Pathology and Glycolytic Shift during Proximal Tubule Atrophy after Ischemic AKI.The Use of Cytochrome C Oxidase Enzyme Activity and Immunohistochemistry in Defining Mitochondrial Injury in Kidney Disease αKlotho Mitigates Progression of AKI to CKD through Activation of Autophagy Pre-clinical model of severe glutathione peroxidase-3 deficiency and chronic kidney disease results in coronary artery thrombosis and depressed left ventricular function.Renal fibrosis: Primacy of the proximal tubule.AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.Acute Kidney Injury.Unilateral Renal Ischemia as a Model of Acute Kidney Injury and Renal Fibrosis in Cats.

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P2860

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

http://www.wikidata.org/entity/Q28388800

description

2014 nî lūn-bûn

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2014 թուականի Յուլիսին հրատարակուած գիտական յօդուած

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2014 թվականի հուլիսին հրատարակված գիտական հոդված

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2014年の論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年論文

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2014年论文

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name

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

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Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

@en

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

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type

label

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

@ast

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

@en

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

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prefLabel

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

@ast

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

@en

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

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Journal of the American Society of Nephrology

P1476

Severe renal mass reduction impairs recovery and promotes fibrosis after AKI

@en

P2093

Aaron J Polichnowski

http://www.w3.org/2001/XMLSchema#string

Anil K Bidani

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Hui Geng

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Karen A Griffin

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Manjeri A Venkatachalam

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Rongpei Lan

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P2860

Nephron number determines susceptibility to renal mass reduction-induced CKD in Lewis and Fisher 344 rats: implications for development of experimentally induced chronic allograft nephropathy

Does AKI truly lead to CKD?

Coexpression of keratin and vimentin in damaged and regenerating tubular epithelia of the kidney

Inhibition of autoregulated TGFbeta signaling simultaneously enhances proliferation and differentiation of kidney epithelium and promotes repair following renal ischemia.

Recovery from renal ischemia-reperfusion injury is associated with altered renal hemodynamics, blunted pressure natriuresis, and sodium-sensitive hypertension.

Acute kidney injury: a springboard for progression in chronic kidney disease

Is there a common mechanism for the progression of different types of renal diseases other than proteinuria? Towards the unifying theme of chronic hypoxia.

Localization of proliferating cell nuclear antigen, vimentin, c-Fos, and clusterin in the postischemic kidney. Evidence for a heterogenous genetic response among nephron segments, and a large pool of mitotically active and dedifferentiated cells.

Morphometric and visual evaluation of fibrosis in renal biopsies

Acute kidney injury increases risk of ESRD among elderly.

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1496-507

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scholarly article

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3307805

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10.1681/ASN.2013040359

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7

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25

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2014-07-01T00:00:00Z

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24511135

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4073422

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