AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.
about
Characterization of urinary exosomal release of aquaporin-1 and -2 after renal ischemia-reperfusion in rats.RTA-408 Protects Kidney from Ischemia-Reperfusion Injury in Mice via Activating Nrf2 and Downstream GSH Biosynthesis Gene.Lipopolysaccharide Mediates the Destruction of Intercellular Tight Junction among Renal Tubular Epithelial Cells via RhoT1/SMAD-4/JAM-3 Pathway.
P2860
AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.
description
2017 nî lūn-bûn
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2017年の論文
@ja
2017年学术文章
@wuu
2017年学术文章
@zh-cn
2017年学术文章
@zh-hans
2017年学术文章
@zh-my
2017年学术文章
@zh-sg
2017年學術文章
@yue
2017年學術文章
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2017年學術文章
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name
AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.
@en
AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.
@nl
type
label
AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.
@en
AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.
@nl
prefLabel
AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.
@en
AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.
@nl
P2093
P2860
P1433
P1476
AKI on CKD: heightened injury, suppressed repair, and the underlying mechanisms.
@en
P2093
Manjeri A Venkatachalam
Qingqing Wei
Youming Peng
P2860
P304
P356
10.1016/J.KINT.2017.06.030
P407
P577
2017-09-07T00:00:00Z