Estrogen withdrawal-induced NF-kappaB activity and bcl-3 expression in breast cancer cells: roles in growth and hormone independence
about
S100A7 and the progression of breast cancerA pathway for tumor necrosis factor-alpha-induced Bcl10 nuclear translocation. Bcl10 is up-regulated by NF-kappaB and phosphorylated by Akt1 and then complexes with Bcl3 to enter the nucleusThe Bik BH3-only protein is induced in estrogen-starved and antiestrogen-exposed breast cancer cells and provokes apoptosisNF-kappaB activation in inflammatory breast cancer is associated with oestrogen receptor downregulation, secondary to EGFR and/or ErbB2 overexpression and MAPK hyperactivationBCL2 and CASP8 regulation by NF-kappaB differentially affect mitochondrial function and cell fate in antiestrogen-sensitive and -resistant breast cancer cells.Enhanced NF kappa B and AP-1 transcriptional activity associated with antiestrogen resistant breast cancerBasal cancer cell survival involves JNK2 suppression of a novel JNK1/c-Jun/Bcl-3 apoptotic network.Multilevel support vector regression analysis to identify condition-specific regulatory networksEstrogen signaling crosstalk: Implications for endocrine resistance in ovarian cancer.IFNgamma restores breast cancer sensitivity to fulvestrant by regulating STAT1, IFN regulatory factor 1, NF-kappaB, BCL2 family members, and signaling to caspase-dependent apoptosis.Alpha-actinin 4 and tumorigenesis of breast cancer.Dynamic modelling of oestrogen signalling and cell fate in breast cancer cellsModeling the estrogen receptor to growth factor receptor signaling switch in human breast cancer cellsAnnexin-A1 regulates microRNA-26b* and microRNA-562 to directly target NF-κB and angiogenesis in breast cancer cells.The role of breast cancer stem cells in metastasis and therapeutic implications.NFkappaB1/p50 is not required for tumor necrosis factor-stimulated growth of primary mammary epithelial cells: implications for NFkappaB2/p52 and RelB.Bortezomib enhances the efficacy of fulvestrant by amplifying the aggregation of the estrogen receptor, which leads to a proapoptotic unfolded protein response.Robust identification of transcriptional regulatory networks using a Gibbs sampler on outlier sum statisticThe effect of 17β-estradiol on gene expression of calcitonin gene-related peptide and some pro-inflammatory mediators in peripheral blood mononuclear cells from patients with pure menstrual migraine.Vascular endothelial growth factor receptor-2 expression is down-regulated by 17beta-estradiol in MCF-7 breast cancer cells by estrogen receptor alpha/Sp proteinsStromal cells promote anti-estrogen resistance of breast cancer cells through an insulin-like growth factor binding protein 5 (IGFBP5)/B-cell leukemia/lymphoma 3 (Bcl-3) axisEndoplasmic reticulum stress, the unfolded protein response, and gene network modeling in antiestrogen resistant breast cancerRole of SP transcription factors in hormone-dependent modulation of genes in MCF-7 breast cancer cells: microarray and RNA interference studies.Differential dependency network analysis to identify condition-specific topological changes in biological networksNuclear factor-kappaB enhances ErbB2-induced mammary tumorigenesis and neoangiogenesis in vivo.COUP-TFII inhibits NFkappaB activation in endocrine-resistant breast cancer cells.Gene network signaling in hormone responsiveness modifies apoptosis and autophagy in breast cancer cellsBcl-3 regulates TGFβ signaling by stabilizing Smad3 during breast cancer pulmonary metastasis.Examining the role of cyclin D1 in breast cancer.Role of Bcl-3 in solid tumorscIAP2 represses IKKα/β-mediated activation of MDM2 to prevent p53 degradation.Ionizing radiation-induced NF-kappaB activation requires PARP-1 function to confer radioresistanceCyr61 expression confers resistance to apoptosis in breast cancer MCF-7 cells by a mechanism of NF-kappaB-dependent XIAP up-regulation.The canonical NF-kappaB pathway is required for formation of luminal mammary neoplasias and is activated in the mammary progenitor population.PARP inhibition sensitizes p53-deficient breast cancer cells to doxorubicin-induced apoptosis.Molecular signaling mechanisms behind polyphenol-induced bone anabolism.BIK/NBK gene as potential marker of prognostic and therapeutic target in breast cancer patients.
P2860
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P2860
Estrogen withdrawal-induced NF-kappaB activity and bcl-3 expression in breast cancer cells: roles in growth and hormone independence
description
2003 nî lūn-bûn
@nan
2003 թուականի Հոկտեմբերին հրատարակուած գիտական յօդուած
@hyw
2003 թվականի հոտեմբերին հրատարակված գիտական հոդված
@hy
2003年の論文
@ja
2003年論文
@yue
2003年論文
@zh-hant
2003年論文
@zh-hk
2003年論文
@zh-mo
2003年論文
@zh-tw
2003年论文
@wuu
name
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@ast
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@en
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@nl
type
label
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@ast
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@en
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@nl
prefLabel
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@ast
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@en
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@nl
P2093
P2860
P1476
Estrogen withdrawal-induced NF ...... rowth and hormone independence
@en
P2093
Dawn White
Gordon Yasvinski
M A Christine Pratt
Michel Ménard
Min Ying Niu
Robert Clarke
Tanya E Bishop
P2860
P304
P356
10.1128/MCB.23.19.6887-6900.2003
P407
P577
2003-10-01T00:00:00Z