Metabolic profiling of hypoxic cells revealed a catabolic signature required for cell survival - Wikidata - wikimedia - TriplyDB

Metabolic profiling of hypoxic cells revealed a catabolic signature required for cell survival

Metabolic profiling of hypoxic cells revealed a catabolic signature required for cell survival

http://www.wikidata.org/entity/Q28476795

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Mitochondrial metabolic remodeling in response to genetic and environmental perturbations Hypoxia and metabolic adaptation of cancer cells Cancer metabolism and oxidative stress: Insights into carcinogenesis and chemotherapy via the non-dihydrofolate reductase effects of methotrexate Autophagy: a targetable linchpin of cancer cell metabolism Changing perspective on oncometabolites: from metabolic signature of cancer to tumorigenic and immunosuppressive agents Metabolic reprogramming: a cancer hallmark even warburg did not anticipate Spatial reorganization of Saccharomyces cerevisiae enolase to alter carbon metabolism under hypoxia.Hypoxia-induced changes in Ca(2+) mobilization and protein phosphorylation implicated in impaired wound healing.Dietary supplementation of heat-treated Gracilaria and Ulva seaweeds enhanced acute hypoxia tolerance in gilthead sea bream (Sparus aurata).Cancer-associated isocitrate dehydrogenase 1 (IDH1) R132H mutation and d-2-hydroxyglutarate stimulate glutamine metabolism under hypoxia.Generalized portrait of cancer metabolic pathways inferred from a list of genes overexpressed in cancer Enhanced heme function and mitochondrial respiration promote the progression of lung cancer cells Mitochondrial bioenergetics of metastatic breast cancer cells in response to dynamic changes in oxygen tension: effects of HIF-1α High throughput synthetic lethality screen reveals a tumorigenic role of adenylate cyclase in fumarate hydratase-deficient cancer cells.Energy metabolism in Mycobacterium gilvum PYR-GCK: insights from transcript expression analyses following two states of induction.Global profiling of metabolic adaptation to hypoxic stress in human glioblastoma cells.Comparative metabolic flux profiling of melanoma cell lines: beyond the Warburg effect.Hypoxia promotes isocitrate dehydrogenase-dependent carboxylation of α-ketoglutarate to citrate to support cell growth and viability Hypoxia Induces Autophagy through Translational Up-Regulation of Lysosomal Proteins in Human Colon Cancer Cells A Compendium of Volatile Organic Compounds (VOCs) Released By Human Cell Lines.Loss of NADH Oxidase Activity in Streptococcus mutans Leads to Rex-Mediated Overcompensation in NAD+ Regeneration by Lactate Dehydrogenase.Dual PI3K/mTOR inhibitor NVP-BEZ235 suppresses hypoxia-inducible factor (HIF)-1α expression by blocking protein translation and increases cell death under hypoxia.Targeting cancer metabolism.Development of an Optimized Protocol for NMR Metabolomics Studies of Human Colon Cancer Cell Lines and First Insight from Testing of the Protocol Using DNA G-Quadruplex Ligands as Novel Anti-Cancer Drugs.EGLN1 Inhibition and Rerouting of α-Ketoglutarate Suffice for Remote Ischemic Protection.Synergistic Inhibitory Effects of Hypoxia and Iron Deficiency on Hepatic Glucose Response in Mouse Liver.Cell metabolomics.Hypoxia-inducible factor 1 alpha is required for the tumourigenic and aggressive phenotype associated with Rab25 expression in ovarian cancer.Phosphorylation of DYNLT1 at serine 82 regulates microtubule stability and mitochondrial permeabilization in hypoxia.Metabolomic Profiling of the Effects of Melittin on Cisplatin Resistant and Cisplatin Sensitive Ovarian Cancer Cells Using Mass Spectrometry and Biolog Microarray Technology.MiRNA-210 modulates a nickel-induced cellular energy metabolism shift by repressing the iron-sulfur cluster assembly proteins ISCU1/2 in Neuro-2a cells.Untargeted metabolomics approach for unraveling robust biomarkers of nutritional status in fasted gilthead sea bream (Sparus aurata).The growing complexity of HIF-1α's role in tumorigenesis: DNA repair and beyond.Mammalian cell metabolomics: experimental design and sample preparation.HIF expression and the role of hypoxic microenvironments within primary tumours as protective sites driving cancer stem cell renewal and metastatic progression.Biological detection by optical oxygen sensing.Hypoxia signaling pathways in cancer metabolism: the importance of co-selecting interconnected physiological pathways Cancer may be a pathway to cell survival under persistent hypoxia and elevated ROS: a model for solid-cancer initiation and early development.Hypoxia-induced nitric oxide production and tumour perfusion is inhibited by pegylated arginine deiminase (ADI-PEG20).Extending metabolome coverage for untargeted metabolite profiling of adherent cultured hepatic cells.

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Metabolic profiling of hypoxic cells revealed a catabolic signature required for cell survival

http://www.wikidata.org/entity/Q28476795

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Metabolic profiling of hypoxic ...... ure required for cell survival

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Metabolic profiling of hypoxic ...... ure required for cell survival

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Metabolic profiling of hypoxic ...... ure required for cell survival

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Metabolic profiling of hypoxic ...... ure required for cell survival

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Barbara A Hedley

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David G Watson

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Gabriela Kalna

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Liang Zheng

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P2860

HIF-1 regulates cytochrome oxidase subunits to optimize efficiency of respiration in hypoxic cells

Pyruvate kinase M2 is a PHD3-stimulated coactivator for hypoxia-inducible factor 1

miR-210 is overexpressed in late stages of lung cancer and mediates mitochondrial alterations associated with modulation of HIF-1 activity

LC3, a mammalian homologue of yeast Apg8p, is localized in autophagosome membranes after processing

Tumor hypoxia: definitions and current clinical, biologic, and molecular aspects

HIF-1-mediated expression of pyruvate dehydrogenase kinase: a metabolic switch required for cellular adaptation to hypoxia

HIF-1 mediates adaptation to hypoxia by actively downregulating mitochondrial oxygen consumption

Mitochondrial autophagy is an HIF-1-dependent adaptive metabolic response to hypoxia

Autophagy and the integrated stress response

Effect of oxygen tension on cellular energetics.

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