Immunological defects in mice with a targeted disruption in Bcl-3
about
Gene function in early mouse embryonic stem cell differentiation.Atypical IκB proteins - nuclear modulators of NF-κB signalingPlasma cell differentiation requires the transcription factor XBP-1Expression of the Bcl-3 proto-oncogene suppresses p53 activationIdentification and characterization of BCL-3-binding protein: implications for transcription and DNA repair or recombinationSignal-dependent translation of a regulatory protein, Bcl-3, in activated human plateletsLead exposure inhibits fracture healing and is associated with increased chondrogenesis, delay in cartilage mineralization, and a decrease in osteoprogenitor frequency.Regulation of the Adaptive Immune Response by the IκB Family Protein Bcl-3NF-κB, the first quarter-century: remarkable progress and outstanding questionsCrystal structure of human pirin: an iron-binding nuclear protein and transcription cofactorBcl3, an IkappaB protein, stimulates activating protein-1 transactivation and cellular proliferationCOX-1 and COX-2 expression in osteoid osteomasCD8 T cells require Bcl-3 for maximal gamma interferon production upon secondary exposure to antigenMAIL, a novel nuclear I kappa B protein that potentiates LPS-induced IL-6 productionThe tumor promoter and NF-κB modulator Bcl-3 regulates splenic B cell development.Elevated levels of Bcl-3 inhibits Treg development and function resulting in spontaneous colitis.NF-kappaB-mediated inhibition of apoptosis is required for encephalomyocarditis virus virulence: a mechanism of resistance in p50 knockout mice.Genetic approaches in mice to understand Rel/NF-kappaB and IkappaB function: transgenics and knockouts.OCA-B integrates B cell antigen receptor-, CD40L- and IL 4-mediated signals for the germinal center pathway of B cell development.The repressing function of the oncoprotein BCL-3 requires CtBP, while its polyubiquitination and degradation involve the E3 ligase TBLR1.Positive and negative regulation of nuclear factor-kappaB-mediated transcription by IkappaB-zeta, an inducible nuclear protein.Bcl3 prevents acute inflammatory lung injury in mice by restraining emergency granulopoiesis.NF-kappaB family of transcription factors: central regulators of innate and adaptive immune functions.The IκB family member Bcl-3 coordinates the pulmonary defense against Klebsiella pneumoniae infection.Requirement for NF-kappaB in osteoclast and B-cell developmentmTOR-dependent synthesis of Bcl-3 controls the retraction of fibrin clots by activated human platelets.Roles of Bcl-3 in the pathogenesis of murine type 1 diabetesFunctions of NF-kappaB1 and NF-kappaB2 in immune cell biologyMice deficient in nuclear factor (NF)-kappa B/p52 present with defects in humoral responses, germinal center reactions, and splenic microarchitectureNuclear factor (NF)-kappa B2 (p100/p52) is required for normal splenic microarchitecture and B cell-mediated immune responsesFunctional redundancy of the nuclear factor kappa B inhibitors I kappa B alpha and I kappa B betaImpaired Bcl3 up-regulation leads to enhanced lipopolysaccharide-induced interleukin (IL)-23P19 gene expression in IL-10(-/-) mice.Quantitative mouse model of implant-associated osteomyelitis and the kinetics of microbial growth, osteolysis, and humoral immunityHigh expression of BCL3 in human myeloma cells is associated with increased proliferation and inferior prognosis.NF-kappaB regulation: the nuclear responseBcl-3 regulates TGFβ signaling by stabilizing Smad3 during breast cancer pulmonary metastasis.It takes two to tango: IκBs, the multifunctional partners of NF-κB.Molecular regulation of urothelial renewal and host defenses during infection with uropathogenic Escherichia coli.Comparing the fine specificity of DNA binding by NF-kappaB p50 and p52 using principal coordinates analysis.Bcl-3 expression promotes cell survival following interleukin-4 deprivation and is controlled by AP1 and AP1-like transcription factors.
P2860
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P2860
Immunological defects in mice with a targeted disruption in Bcl-3
description
1997 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
1997 թվականի հունվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1997
@ast
im Januar 1997 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
@en
vedecký článok (publikovaný 1997/01/15)
@sk
vědecký článek publikovaný v roce 1997
@cs
wetenschappelijk artikel (gepubliceerd op 1997/01/15)
@nl
наукова стаття, опублікована в січні 1997
@uk
مقالة علمية (نشرت في 15-1-1997)
@ar
name
Immunological defects in mice with a targeted disruption in Bcl-3
@ast
Immunological defects in mice with a targeted disruption in Bcl-3
@en
Immunological defects in mice with a targeted disruption in Bcl-3
@nl
type
label
Immunological defects in mice with a targeted disruption in Bcl-3
@ast
Immunological defects in mice with a targeted disruption in Bcl-3
@en
Immunological defects in mice with a targeted disruption in Bcl-3
@nl
prefLabel
Immunological defects in mice with a targeted disruption in Bcl-3
@ast
Immunological defects in mice with a targeted disruption in Bcl-3
@en
Immunological defects in mice with a targeted disruption in Bcl-3
@nl
P2093
P3181
P356
P1433
P1476
Immunological defects in mice with a targeted disruption in Bcl-3
@en
P2093
E. M. Schwarz
I. M. Verma
P. Krimpenfort
P304
P3181
P356
10.1101/GAD.11.2.187
P577
1997-01-15T00:00:00Z