Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes - Wikidata - wikimedia - TriplyDB

Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

http://www.wikidata.org/entity/Q35757083

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Atypical IκB proteins - nuclear modulators of NF-κB signaling Regulation of the Adaptive Immune Response by the IκB Family Protein Bcl-3 The tumor promoter and NF-κB modulator Bcl-3 regulates splenic B cell development.The NF-κB regulator Bcl-3 governs dendritic cell antigen presentation functions in adaptive immunity The oncoprotein and transcriptional regulator Bcl-3 governs plasticity and pathogenicity of autoimmune T cells Orbital sporadic Burkitt lymphoma in an adult diabetic African American female and a review of adult orbital cases.The IκB family member Bcl-3 coordinates the pulmonary defense against Klebsiella pneumoniae infection.Gene network inference using continuous time Bayesian networks: a comparative study and application to Th17 cell differentiation.The NF-κB regulator Bcl-3 modulates inflammation during contact hypersensitivity reactions in radioresistant cells.Induction of Bcl-3 by acute binge alcohol results in toll-like receptor 4/LPS tolerance.Though Active on RINm5F Insulinoma Cells and Cultured Pancreatic Islets, Recombinant IL-22 Fails to Modulate Cytotoxicity and Disease in a Protocol of Streptozotocin-Induced Experimental Diabetes xHeinz: an algorithm for mining cross-species network modules under a flexible conservation model.Mapping the Interaction of B Cell Leukemia 3 (BCL-3) and Nuclear Factor κB (NF-κB) p50 Identifies a BCL-3-mimetic Anti-inflammatory Peptide.Association of GWAS-Supported Variants rs556621 on Chromosome 6p21.1 with Large Artery Atherosclerotic Stroke in a Southern Chinese Han Population.Bcl-3 puts the brakes on contact hypersensitivity.AT-Rich-Interactive Domain-Containing Protein 5A Functions as a Negative Regulator of Retinoic Acid Receptor-Related Orphan Nuclear Receptor γt-Induced Th17 Cell Differentiation

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Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

http://www.wikidata.org/entity/Q35757083

description

2010 nî lūn-bûn

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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name

Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

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Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

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type

label

Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

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Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

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prefLabel

Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

@ast

Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

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Roles of Bcl-3 in the pathogenesis of murine type 1 diabetes

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Qingguo Ruan

http://www.w3.org/2001/XMLSchema#string

Ruaidhri J Carmody

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Scott Palmer

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Shi-Jun Zheng

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Youhai H Chen

http://www.w3.org/2001/XMLSchema#string

P2860

The bcl-3 proto-oncogene encodes a nuclear I kappa B-like molecule that preferentially interacts with NF-kappa B p50 and p52 in a phosphorylation-dependent manner

Selective inhibition of NF-kappaB activation by a peptide that blocks the interaction of NEMO with the IkappaB kinase complex

Diabetes-associated sustained activation of the transcription factor nuclear factor-kappaB

The oncoprotein Bcl-3 directly transactivates through kappa B motifs via association with DNA-binding p50B homodimers

Introduction to NF-kappaB: players, pathways, perspectives

A functional variant of SUMO4, a new I kappa B alpha modifier, is associated with type 1 diabetes

Immunological defects in mice with a targeted disruption in Bcl-3

Critical roles for the Bcl-3 oncoprotein in T cell-mediated immunity, splenic microarchitecture, and germinal center reactions

Opposing roles for RelB and Bcl-3 in regulation of T-box expressed in T cells, GATA-3, and Th effector differentiation

Distinct activities of p52/NF-kappa B required for proper secondary lymphoid organ microarchitecture: functions enhanced by Bcl-3

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45151189

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20622172

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type-1 diabetes

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3279524

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