Viable hypomorphic signaling mutant of the Met receptor reveals a role for hepatocyte growth factor in postnatal cerebellar development
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MUC20 suppresses the hepatocyte growth factor-induced Grb2-Ras pathway by binding to a multifunctional docking site of metThe genetic and neurobiologic compass points toward common signaling dysfunctions in autism spectrum disordersA genetic variant that disrupts MET transcription is associated with autismGABAA receptor gamma 2 subunit knockdown mice have enhanced anxiety-like behavior but unaltered hypnotic response to benzodiazepines.Consensus paper: pathological role of the cerebellum in autismDecreased Serum Hepatocyte Growth Factor (HGF) in Autistic Children with Severe Gastrointestinal DiseaseEvidence of cell-nonautonomous changes in dendrite and dendritic spine morphology in the met-signaling-deficient mouse forebrainDistinct requirements for Gab1 in Met and EGF receptor signaling in vivo.Association of genetic variation in the MET proto-oncogene with schizophrenia and general cognitive ability.MET and AKT genetic influence on facial emotion perception.Inhibition of the MET Receptor Tyrosine Kinase as a Novel Therapeutic Strategy in MedulloblastomaGonadotropin-releasing hormone (GnRH) neuron migration: initiation, maintenance and cessation as critical steps to ensure normal reproductive functionCellular commitment in the developing cerebellumSocial Isolation Stress Induces Anxious-Depressive-Like Behavior and Alterations of Neuroplasticity-Related Genes in Adult Male Mice.Is autism a disease of the cerebellum? An integration of clinical and pre-clinical research.Excessive activation of tissue plasminogen activator makes a mouse nervous.Consensus paper: radiological biomarkers of cerebellar diseasesChicken Ovalbumin Upstream Promoter-Transcription Factor II (COUP-TFII) regulates growth and patterning of the postnatal mouse cerebellum.MET and autism susceptibility: family and case-control studies.New roles for the cerebellum in health and disease.Genetics of medulloblastoma: clues for novel therapies.MET receptor tyrosine kinase as an autism genetic risk factor.Specific Grb2-mediated interactions regulate clathrin-dependent endocytosis of the cMet-tyrosine kinase.SHH inhibitors for the treatment of medulloblastoma.Immune mediators in the brain and peripheral tissues in autism spectrum disorder.Distinct intracellular signaling mediates C-MET regulation of dendritic growth and synaptogenesis.Gene expression alterations during HGF-induced dedifferentiation of a renal tubular epithelial cell line (MDCK) using a novel canine DNA microarray.Hepatocyte growth factor and sonic Hedgehog expression in cerebellar neural progenitor cells costimulate medulloblastoma initiation and growth.The autism susceptibility gene met regulates zebrafish cerebellar development and facial motor neuron migrationModulation of p53 and met expression by Krüppel-like factor 8 regulates zebrafish cerebellar development.Assessment of de novo copy-number variations in Italian patients with schizophrenia: Detection of putative mutations involving regulatory enhancer elements.HGF-Met Pathway in Regeneration and Drug Discovery.The Human Gut Microbiome - A Potential Controller of Wellness and Disease
P2860
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P2860
Viable hypomorphic signaling mutant of the Met receptor reveals a role for hepatocyte growth factor in postnatal cerebellar development
description
2002 թուականի Նոյեմբերին հրատարակուած գիտական յօդուած
@hyw
2002 թվականի նոյեմբերին հրատարակված գիտական հոդված
@hy
article publié dans les Procee ...... f the United States of America
@fr
artículu científicu espublizáu en 2002
@ast
im November 2002 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2002/11/12)
@sk
vědecký článek publikovaný v roce 2002
@cs
wetenschappelijk artikel (gepubliceerd op 2002/11/12)
@nl
наукова стаття, опублікована в листопаді 2002
@uk
name
Viable hypomorphic signaling m ...... stnatal cerebellar development
@ast
Viable hypomorphic signaling m ...... stnatal cerebellar development
@en
Viable hypomorphic signaling m ...... stnatal cerebellar development
@nl
type
label
Viable hypomorphic signaling m ...... stnatal cerebellar development
@ast
Viable hypomorphic signaling m ...... stnatal cerebellar development
@en
Viable hypomorphic signaling m ...... stnatal cerebellar development
@nl
prefLabel
Viable hypomorphic signaling m ...... stnatal cerebellar development
@ast
Viable hypomorphic signaling m ...... stnatal cerebellar development
@en
Viable hypomorphic signaling m ...... stnatal cerebellar development
@nl
P2093
P2860
P356
P1476
Viable hypomorphic signaling m ...... stnatal cerebellar development
@en
P2093
Alessandro Ieraci
Carola Ponzetto
Paolo E. Forni
P2860
P304
15200–15205
P356
10.1073/PNAS.222362099
P407
P577
2002-11-12T00:00:00Z