Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
about
Chemical genetic screen identifies lithocholic acid as an anti-aging compound that extends yeast chronological life span in a TOR-independent manner, by modulating housekeeping longevity assurance processesRole of ROS and RNS Sources in Physiological and Pathological ConditionsOxidative Stress in the Healthy and Wounded Hepatocyte: A Cellular Organelles PerspectiveThe different facets of organelle interplay-an overview of organelle interactionsNo peroxisome is an island - Peroxisome contact sitesHypoxia signaling pathways: modulators of oxygen-related organellesPeroxisomes are required for lipid metabolism and muscle function in Drosophila melanogasterWhat is metabolic syndrome, and why are children getting it?Redox interplay between mitochondria and peroxisomesProtective Effects of α-Tocopherol, γ-Tocopherol and Oleic Acid, Three Compounds of Olive Oils, and No Effect of Trolox, on 7-Ketocholesterol-Induced Mitochondrial and Peroxisomal Dysfunction in Microglial BV-2 CellsCOMPARTMENTS: unification and visualization of protein subcellular localization evidencealpha-Synuclein abnormalities in mouse models of peroxisome biogenesis disorders.Peroxisomal biogenesis is genetically and biochemically linked to carbohydrate metabolism in Drosophila and mouse.Toward a unifying hypothesis of metabolic syndrome.PEX13 deficiency in mouse brain as a model of Zellweger syndrome: abnormal cerebellum formation, reactive gliosis and oxidative stress.Nonylphenol-mediated CYP induction is PXR-dependent: The use of humanized mice and human hepatocytes suggests that hPXR is less sensitive than mouse PXR to nonylphenol treatment.Peroxisomal multifunctional protein-2 deficiency causes motor deficits and glial lesions in the adult central nervous system.Carbohydrate metabolism is perturbed in peroxisome-deficient hepatocytes due to mitochondrial dysfunction, AMP-activated protein kinase (AMPK) activation, and peroxisome proliferator-activated receptor γ coactivator 1α (PGC-1α) suppression.Deletion of a single allele of the Pex11β gene is sufficient to cause oxidative stress, delayed differentiation and neuronal death in mouse brainPeroxisome deficiency-induced ER stress and SREBP-2 pathway activation in the liver of newborn PEX2 knock-out miceInduced pluripotent stem cell models of Zellweger spectrum disorder show impaired peroxisome assembly and cell type-specific lipid abnormalities.Induction of mitochondrial changes associated with oxidative stress on very long chain fatty acids (C22:0, C24:0, or C26:0)-treated human neuronal cells (SK-NB-E).Peroxisome proliferator-activated receptor and age-related macular degenerationEndothelial peroxisomal dysfunction and impaired pexophagy promotes oxidative damage in lipopolysaccharide-induced acute kidney injury.Emerging roles of mitochondria in the evolution, biogenesis, and function of peroxisomes.Peroxisome deficient invertebrate and vertebrate animal modelsBile acids: the role of peroxisomes.Developmental roles of D-bifunctional protein-A zebrafish model of peroxisome dysfunction.Tauroursodeoxycholic bile acid arrests axonal degeneration by inhibiting the unfolded protein response in X-linked adrenoleukodystrophy.Peroxisome biogenesis disorders: Biological, clinical and pathophysiological perspectives.Mitochondrial free radical theory of aging: who moved my premise?Peroxisome-mitochondria interplay and disease.Dysregulation of redox pathways in liver fibrosis.Microporation is an efficient method for siRNA-induced knockdown of PEX5 in HepG2 cells: evaluation of the transfection efficiency, the PEX5 mRNA and protein levels and induction of peroxisomal deficiency.Target acquired: Selective autophagy in cardiometabolic disease.The Peroxisome-Mitochondria Connection: How and Why?Fis1 depletion in osteoarthritis impairs chondrocyte survival and peroxisomal and lysosomal function.Pathophysiology and fate of hepatocytes in a mouse model of mitochondrial hepatopathies.Effects of peroxisomal catalase inhibition on mitochondrial function.Peroxisome deficiency causes a complex phenotype because of hepatic SREBP/Insig dysregulation associated with endoplasmic reticulum stress.
P2860
Q24616983-C167696E-C15F-435F-BCD0-CA325DE94DC0Q26740342-ADB796B9-802C-4C5D-940F-308B1C77111DQ26770805-E73E89E3-115E-441D-AEEA-D840F6CF412DQ26781052-43F3B343-7818-405A-9261-6A3709E3E0CFQ26783227-258A909C-F740-4E8A-8CC6-857A1251831BQ26799950-4B2FD39F-EBDB-4EA4-BA98-A7BD5BD31A9BQ27322788-934926E5-1BF7-4487-9D19-6F119BF0A845Q28385817-8402DFF1-7FDB-416E-9767-C5230A89976DQ28394944-1A0701C4-3A42-4418-B987-03A965A77254Q28397314-DEF3BEC6-8F35-4EC0-8D1F-31A543C931CEQ30002352-CD6408A3-EB34-4922-9A5F-3DEB5AC450A0Q33643679-DBE7B82C-51C5-4558-9B26-0BE9E4C95625Q33825204-212FA81A-79F4-4E98-A5FE-70D968673D78Q34255878-EAFEDEF0-7E1E-4B8F-853F-877F59D07AFBQ34458791-ADE82597-956C-4DB0-912F-C149C022FAA7Q34909409-3570E80F-052D-4FCC-ABBA-2405296F4C37Q35088180-9AC568F8-8DDE-4B91-B7FE-827D8D6F7E9DQ35604967-26338D5B-F95C-43DA-86AE-C4743006D553Q35665063-F4C9C2EC-C06B-4839-92D2-D3CDFF33C222Q36004816-E4BAC683-A21D-4F55-903F-5F3E105A2D36Q36005581-FA7BD6EA-F4CD-438A-9EBB-FAA42E1224F9Q36167749-47868569-3A15-4E95-BDC7-B0954F7B32AAQ36446030-1615A470-0D4F-4C11-8A21-0BA653ABC4E9Q36953507-43165AD1-0AF2-44A6-AE17-9654E8ED3E8AQ37199592-9C9E6C3F-ED88-470F-A3B8-7DD361B33A0CQ37334200-AC1D0835-9674-4237-9894-221076326E25Q37382119-22A41CDF-A51E-44CF-88F5-F700FA35DD64Q37532699-C27858C9-383C-4439-8297-4F5661FFDD4EQ37596791-7B9646A7-8159-4741-8233-1E1F06DD311EQ38116808-6098116D-12D0-49A0-A5C5-60465C4A302EQ38205924-5062C328-E59E-454C-9222-09F313FCBBB9Q38357539-0092C208-134A-472C-8BFA-938052213A43Q38937340-3C78870F-0302-495A-A5D6-C46289926D46Q38956337-CF0352CD-C731-4B80-BF3D-38E08CC4D5D4Q39155198-79790C60-79E0-41BF-B662-D61E07126FAAQ39326795-0BD0447C-4F33-4C4E-8C0F-869BE3DCBD21Q39516538-DD913356-D8C2-4F04-89CD-A281D3749BE8Q41919172-972F9D11-5EFC-4967-A643-8DC72EBB7813Q42535813-4C504152-5A1A-4860-A50F-25B1EA04CCC8Q43167089-F89B11DE-1B2F-44AA-8489-B6FCFE9EDD35
P2860
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
description
2005 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2005 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2005
@ast
im April 2005 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2005/04/01)
@sk
vědecký článek publikovaný v roce 2005
@cs
wetenschappelijk artikel (gepubliceerd op 2005/04/01)
@nl
наукова стаття, опублікована у квітні 2005
@uk
مقالة علمية (نشرت في أبريل 2005)
@ar
name
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@ast
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@en
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@nl
type
label
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@ast
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@en
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@nl
prefLabel
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@ast
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@en
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@nl
P2093
P2860
P3181
P356
P1433
P1476
Absence of peroxisomes in mouse hepatocytes causes mitochondrial and ER abnormalities
@en
P2093
Arno Schad
Dariush Fahimi
Katrin Martens
Markus Grabenbauer
Myriam Baes
Paul P. Van Veldhoven
Peter Declercq
Ruud Dirkx
P2860
P304
P3181
P356
10.1002/HEP.20628
P407
P577
2005-04-01T00:00:00Z