Difluorinated-curcumin (CDF) restores PTEN expression in colon cancer cells by down-regulating miR-21 - Wikidata - wikimedia - TriplyDB

Difluorinated-curcumin (CDF) restores PTEN expression in colon cancer cells by down-regulating miR-21

Difluorinated-curcumin (CDF) restores PTEN expression in colon cancer cells by down-regulating miR-21

http://www.wikidata.org/entity/Q28534834

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The critical roles of miR-21 in anti-cancer effects of curcumin Non-coding landscapes of colorectal cancer Targeting colorectal cancer stem cells using curcumin and curcumin analogues: insights into the mechanism of the therapeutic efficacy MicroRNAs and Chinese Medicinal Herbs: New Possibilities in Cancer Therapy MicroRNAs potential utility in colon cancer: Early detection, prognosis, and chemosensitivity Lifestyle Modifications and Colorectal Cancer Antisense oligonucleotides against microRNA-21 reduced the proliferation and migration of human colon carcinoma cells Colorectal Cancer: From the Genetic Model to Posttranscriptional Regulation by Noncoding RNAs.miR-145 downregulates the expression of cyclin-dependent kinase 6 in human cervical carcinoma cells.Prognostic value of miR-21 in various cancers: an updating meta-analysis.Pathological unfoldomics of uncontrolled chaos: intrinsically disordered proteins and human diseases Molecular targets of naturopathy in cancer research: bridge to modern medicine.Omega-3 fatty acid is a potential preventive agent for recurrent colon cancer.Prediction of poor prognosis in breast cancer patients based on microRNA-21 expression: a meta-analysis.miR-21 and miR-145 cooperation in regulation of colon cancer stem cells.Manipulating miRNA Expression: A Novel Approach for Colon Cancer Prevention and Chemotherapy Contribution of microRNAs in understanding the pancreatic tumor microenvironment involving cancer associated stellate and fibroblast cells.Metformin: a potential therapeutic agent for recurrent colon cancer.The Structure and Clinical Roles of MicroRNA in Colorectal Cancer.Linking Diet to Colorectal Cancer: The Emerging Role of MicroRNA in the Communication between Plant and Animal Kingdoms.Dietary phytochemicals alter epigenetic events and signaling pathways for inhibition of metastasis cascade: phytoblockers of metastasis cascade.Novel Holistic Approaches for Overcoming Therapy Resistance in Pancreatic and Colon Cancers.Modulatory roles of microRNAs in the regulation of different signalling pathways in large bowel cancer stem cells.The bounty of nature for changing the cancer landscape.MicroRNA-454 functions as an oncogene by regulating PTEN in uveal melanoma.Curcumin inhibits proliferation-migration of NSCLC by steering crosstalk between a Wnt signaling pathway and an adherens junction via EGR-1.Involvement of Non-coding RNAs in the Signaling Pathways of Colorectal Cancer.Cancer Chemoprevention by Phytochemicals: Nature's Healing Touch.MicroRNAs in HPV associated cancers: small players with big consequences.Effects of resveratrol, curcumin, berberine and other nutraceuticals on aging, cancer development, cancer stem cells and microRNAs.MicroRNA: A novel target of curcumin in cancer therapy.Silencing miR-21 sensitizes non-small cell lung cancer A549 cells to ionizing radiation through inhibition of PI3K/Akt.Diet and cancer prevention: Dietary compounds, dietary MicroRNAs, and dietary exosomes.Effects of microRNA-21 on apoptosis by regulating the expression of PTEN in diffuse large B-cell lymphoma.Curcumin inhibits in vitro and in vivo chronic myelogenous leukemia cells growth: a possible role for exosomal disposal of miR-21.Molecular Crosstalking among Noncoding RNAs: A New Network Layer of Genome Regulation in Cancer.Role of phytochemicals in the modulation of miRNA expression in cancer.Impact of curcumin on the regulation of microRNAs in colorectal cancer.Folic acid conjugated polymeric micelles loaded with a curcumin difluorinated analog for targeting cervical and ovarian cancers.

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Difluorinated-curcumin (CDF) restores PTEN expression in colon cancer cells by down-regulating miR-21

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Difluorinated-curcumin (CDF) r ...... ells by down-regulating miR-21

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Difluorinated-curcumin (CDF) r ...... ells by down-regulating miR-21

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Difluorinated-curcumin (CDF) r ...... ells by down-regulating miR-21

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Difluorinated-curcumin (CDF) r ...... ells by down-regulating miR-21

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Difluorinated-curcumin (CDF) r ...... ells by down-regulating miR-21

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Adhip P N Majumdar

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Fazlul H Sarkar

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Sanchita Roy

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Yingjie Yu

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P2860

Gemcitabine sensitivity can be induced in pancreatic cancer cells through modulation of miR-200 and miR-21 expression by curcumin or its analogue CDF

PTEN activation contributes to tumor inhibition by trastuzumab, and loss of PTEN predicts trastuzumab resistance in patients.

A functional genetic approach identifies the PI3K pathway as a major determinant of trastuzumab resistance in breast cancer.

A human colon cancer cell capable of initiating tumour growth in immunodeficient mice

MicroRNAs: genomics, biogenesis, mechanism, and function

Tumor-suppressive miR-34a induces senescence-like growth arrest through modulation of the E2F pathway in human colon cancer cells

Anti-tumor activity of a novel compound-CDF is mediated by regulating miR-21, miR-200, and PTEN in pancreatic cancer

Pten is essential for embryonic development and tumour suppression

MicroRNA-21 regulates expression of the PTEN tumor suppressor gene in human hepatocellular cancer

miR-21-mediated tumor growth

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