Glucosamine inhibits angiotensin II-induced cytoplasmic Ca2+ elevation in neonatal cardiomyocytes via protein-associated O-linked N-acetylglucosamine
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Protrusive waves guide 3D cell migration along nanofibers.Activation of the intracellular renin-angiotensin system in cardiac fibroblasts by high glucose: role in extracellular matrix productionImpact of Type 2 diabetes and aging on cardiomyocyte function and O-linked N-acetylglucosamine levels in the heartIncreased O-GlcNAc levels during reperfusion lead to improved functional recovery and reduced calpain proteolysisGlucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAcNon-canonical glycosyltransferase modulates post-hypoxic cardiac myocyte death and mitochondrial permeability transitionO-GlcNAcylation involvement in high glucose-induced cardiac hypertrophy via ERK1/2 and cyclin D2Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein O-GlcNAc and increased mitochondrial Bcl-2Overexpression of TRPC3 increases apoptosis but not necrosis in response to ischemia-reperfusion in adult mouse cardiomyocytes.Glucosamine cardioprotection in perfused rat hearts associated with increased O-linked N-acetylglucosamine protein modification and altered p38 activation.O-GlcNAc signaling in the cardiovascular system.Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic miceGlobal mass spectrometry and transcriptomics array based drug profiling provides novel insight into glucosamine induced endoplasmic reticulum stressProtein O-GlcNAcylation and cardiovascular (patho)physiology.Lithium induces ER stress and N-glycan modification in galactose-grown Jurkat cells.Increased O-linked beta-N-acetylglucosamine levels on proteins improves survival, reduces inflammation and organ damage 24 hours after trauma-hemorrhage in ratsGlutamine-induced protection of isolated rat heart from ischemia/reperfusion injury is mediated via the hexosamine biosynthesis pathway and increased protein O-GlcNAc levels.The dynamic stress-induced "O-GlcNAc-ome" highlights functions for O-GlcNAc in regulating DNA damage/repair and other cellular pathways.The roles of O-linked β-N-acetylglucosamine in cardiovascular physiology and disease.The augmentation of O-GlcNAcylation reduces glyoxal-induced cell injury by attenuating oxidative stress in human retinal microvascular endothelial cells.Modification of STIM1 by O-linked N-acetylglucosamine (O-GlcNAc) attenuates store-operated calcium entry in neonatal cardiomyocytesPost-translational protein modification by O-linked N-acetyl-glucosamine: its role in mediating the adverse effects of diabetes on the heart.Role of protein O-linked N-acetyl-glucosamine in mediating cell function and survival in the cardiovascular system.Protein O-GlcNAcylation: a new signaling paradigm for the cardiovascular system.Dynamic O-GlcNAcylation and its roles in the cellular stress response and homeostasis.Unique hexosaminidase reduces metabolic survival signal and sensitizes cardiac myocytes to hypoxia/reoxygenation injury.O-GlcNAc signaling attenuates ER stress-induced cardiomyocyte death.STIM1/Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathologyThe role of protein O-linked beta-N-acetylglucosamine in mediating cardiac stress responses.Chemical tools to probe cellular O-GlcNAc signalling.Fatty acid synthase inhibits the O-GlcNAcase during oxidative stress.Oxidative stress induces transient O-GlcNAc elevation and tau dephosphorylation in SH-SY5Y cells.O-linked beta-N-acetylglucosamine (O-GlcNAc) regulates stress-induced heat shock protein expression in a GSK-3beta-dependent manner.The proinflammatory phenotype of PECAM-1-deficient mice results in atherogenic diet-induced steatohepatitis.Protein O-GlcNAcylation: A critical regulator of the cellular response to stress.Augmented O-GlcNAc signaling attenuates oxidative stress and calcium overload in cardiomyocytes.Enzymatic and non-enzymatic post-translational modifications linking diabetes and heart disease.Role of nitrosative stress in early neuropathy and vascular dysfunction in streptozotocin-diabetic rats.A proteolytic fragment of histone deacetylase 4 protects the heart from failure by regulating the hexosamine biosynthetic pathway.Roles of Glycoproteins in the Diagnosis and Differential Diagnosis of Chronic and Latent Keshan Disease.
P2860
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P2860
Glucosamine inhibits angiotensin II-induced cytoplasmic Ca2+ elevation in neonatal cardiomyocytes via protein-associated O-linked N-acetylglucosamine
description
2006 թուականի Յունուարին հրատարակուած գիտական յօդուած
@hyw
2006 թվականի հունվարին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2006
@ast
im Januar 2006 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 2006/01/01)
@sk
vědecký článek publikovaný v roce 2006
@cs
wetenschappelijk artikel (gepubliceerd op 2006/01/01)
@nl
наукова стаття, опублікована в січні 2006
@uk
مقالة علمية (نشرت عام 2006)
@ar
name
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@ast
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@en
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@nl
type
label
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@ast
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@en
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@nl
prefLabel
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@ast
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@en
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@nl
P2093
P2860
P3181
P1476
Glucosamine inhibits angiotens ...... d O-linked N-acetylglucosamine
@en
P2093
John C Chatham
Richard B Marchase
Voraratt Champattanachai
P2860
P304
P3181
P356
10.1152/AJPCELL.00263.2005
P50
P577
2005-08-17T00:00:00Z