Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic mice - Wikidata - wikimedia - TriplyDB

Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic mice

Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic mice

http://www.wikidata.org/entity/Q34512181

about

The role of O-GlcNAc transferase in regulating the gene transcription of developing and failing hearts Functional O-GlcNAc modifications: implications in molecular regulation and pathophysiology O-GlcNAcylation and oxidation of proteins: is signalling in the cardiovascular system becoming sweeter?Glucose Transporters in Cardiac Metabolism and Hypertrophy O-GlcNAcylation involvement in high glucose-induced cardiac hypertrophy via ERK1/2 and cyclin D2 Functional decorations: post-translational modifications and heart disease delineated by targeted proteomics.The role of O-GlcNAc signaling in the pathogenesis of diabetic retinopathy.Acute regulation of cardiac metabolism by the hexosamine biosynthesis pathway and protein O-GlcNAcylation.Consuming a Western diet for two weeks suppresses fetal genes in mouse hearts.MicroRNA-539 is up-regulated in failing heart, and suppresses O-GlcNAcase expression.Cardiomyocyte-specific BMAL1 plays critical roles in metabolism, signaling, and maintenance of contractile function of the heart.Protein O-GlcNAcylation and cardiovascular (patho)physiology.A systematic review of fetal genes as biomarkers of cardiac hypertrophy in rodent models of diabetes.High glucose induces mitochondrial dysfunction independently of protein O-GlcNAcylation.O-GlcNAcylation, novel post-translational modification linking myocardial metabolism and cardiomyocyte circadian clock.Metabolic effects of glutamine on the heart: anaplerosis versus the hexosamine biosynthetic pathway Alterations in left ventricular function during intermittent hypoxia: Possible involvement of O-GlcNAc protein and MAPK signaling.The roles of O-linked β-N-acetylglucosamine in cardiovascular physiology and disease.O-GlcNAc signaling is essential for NFAT-mediated transcriptional reprogramming during cardiomyocyte hypertrophy.Chronic ingestion of a Western diet increases O-linked-β-N-acetylglucosamine (O-GlcNAc) protein modification in the rat heart.The impact of thiamine treatment in the diabetes mellitus.Acute O-GlcNAcylation prevents inflammation-induced vascular dysfunction.Cardiac O-GlcNAcylation blunts autophagic signaling in the diabetic heart Modification of STIM1 by O-linked N-acetylglucosamine (O-GlcNAc) attenuates store-operated calcium entry in neonatal cardiomyocytes Overview: the maturing of proteomics in cardiovascular research.Post-translational protein modification by O-linked N-acetyl-glucosamine: its role in mediating the adverse effects of diabetes on the heart.Exercise and diabetes have opposite effects on the assembly and O-GlcNAc modification of the mSin3A/HDAC1/2 complex in the heart.Immediate effects of a single exercise bout on protein O-GlcNAcylation and chromatin regulation of cardiac hypertrophy.STIM1/Orai1-mediated SOCE: current perspectives and potential roles in cardiac function and pathology Cardiomyocyte Ogt is essential for postnatal viability.O-GlcNAc and the cardiovascular system.Stromal interaction molecule 1 is essential for normal cardiac homeostasis through modulation of ER and mitochondrial function.Protein O-linked β-N-acetylglucosamine: a novel effector of cardiomyocyte metabolism and function.Oxidative stress and myocardial injury in the diabetic heart Role of O-GlcNAcylation in nutritional sensing, insulin resistance and in mediating the benefits of exercise.New insights: A role for O-GlcNAcylation in diabetic complications.Changes in O-Linked N-Acetylglucosamine (O-GlcNAc) Homeostasis Activate the p53 Pathway in Ovarian Cancer Cells.Metabolism in cardiomyopathy: every substrate matters.O-GlcNAcylation and cardiovascular disease.Impact of protein O-GlcNAcylation on neural tube malformation in diabetic embryopathy.

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Activation of the hexosamine biosynthesis pathway and protein O-GlcNAcylation modulate hypertrophic and cell signaling pathways in cardiomyocytes from diabetic mice

http://www.wikidata.org/entity/Q34512181

description

2010 nî lūn-bûn

@nan

2010 թուականի Յուլիսին հրատարակուած գիտական յօդուած

@hyw

2010 թվականի հուլիսին հրատարակված գիտական հոդված

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2010年の論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年論文

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2010年论文

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name

Activation of the hexosamine b ...... diomyocytes from diabetic mice

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Activation of the hexosamine b ...... diomyocytes from diabetic mice

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Activation of the hexosamine b ...... diomyocytes from diabetic mice

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Activation of the hexosamine b ...... diomyocytes from diabetic mice

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Activation of the hexosamine b ...... diomyocytes from diabetic mice

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Activation of the hexosamine b ...... diomyocytes from diabetic mice

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Activation of the hexosamine b ...... diomyocytes from diabetic mice

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Activation of the hexosamine b ...... diomyocytes from diabetic mice

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Activation of the hexosamine b ...... diomyocytes from diabetic mice

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Activation of the hexosamine b ...... diomyocytes from diabetic mice

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John C Chatham

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Louis J Dell'Italia

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Susan A Marsh

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P2860

Effect of Rosiglitazone on the Risk of Myocardial Infarction and Death from Cardiovascular Causes

Hyperglycemia-induced mitochondrial superoxide overproduction activates the hexosamine pathway and induces plasminogen activator inhibitor-1 expression by increasing Sp1 glycosylation

Biochemistry and molecular cell biology of diabetic complications

Impact of Type 2 diabetes and aging on cardiomyocyte function and O-linked N-acetylglucosamine levels in the heart

The hexosamine signaling pathway: deciphering the "O-GlcNAc code"

Cycling of O-linked beta-N-acetylglucosamine on nucleocytoplasmic proteins

Glucosamine inhibits angiotensin II-induced cytoplasmic Ca2+ elevation in neonatal cardiomyocytes via protein-associated O-linked N-acetylglucosamine

Increased O-GlcNAc levels during reperfusion lead to improved functional recovery and reduced calpain proteolysis

Glucosamine protects neonatal cardiomyocytes from ischemia-reperfusion injury via increased protein-associated O-GlcNAc

Effects of intensive glucose lowering in type 2 diabetes

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