Essential role of the redox-sensitive kinase p66shc in determining energetic and oxidative status and cell fate in neuronal preconditioning
about
The multiple functions of cytochrome c and their regulation in life and death decisions of the mammalian cell: from respiration to apoptosisRedox modification of proteins as essential mediators of CNS autophagy and mitophagy.p53 and mitochondrial function in neurons.Proteomic alterations in mouse kidney induced by andrographolide sodium bisulfiteDeletion of p66Shc in mice increases the frequency of size-change mutations in the lacZ transgeneIsoprostane generation and function.The fatty acid oxidation product 15-A3t-isoprostane is a potent inhibitor of NFκB transcription and macrophage transformation.Voluntary exercise protects against methamphetamine-induced oxidative stress in brain microvasculature and disruption of the blood-brain barrier.New aspects of p66Shc in ischaemia reperfusion injury and other cardiovascular diseases.p66Shc signaling is involved in stress responses elicited by anthracycline treatment of rat cardiomyoblasts.Alteration of isocitrate dehydrogenase following acute ischemic injury as a means to improve cellular energetic status in neuroadaptation.Metabolic multianalyte microphysiometry reveals extracellular acidosis is an essential mediator of neuronal preconditioning.Neuron specific metabolic adaptations following multi-day exposures to oxygen glucose deprivation.CHIP Is an Essential Determinant of Neuronal Mitochondrial Stress Signaling.Lack of Contribution of p66shc and Its Mitochondrial Translocation to Ischemia-Reperfusion Injury and Cardioprotection by Ischemic Preconditioning.PRKCB/protein kinase C, beta and the mitochondrial axis as key regulators of autophagy.Prolonged swimming promotes cellular oxidative stress and p66Shc phosphorylation, but does not induce oxidative stress in mitochondria in the rat heart.Deletion of the ageing gene p66(Shc) reduces early stroke size following ischaemia/reperfusion brain injury.Shooting vascular oxidative stress: new hopes for stroke patients?
P2860
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P2860
Essential role of the redox-sensitive kinase p66shc in determining energetic and oxidative status and cell fate in neuronal preconditioning
description
2010 թուականի Ապրիլին հրատարակուած գիտական յօդուած
@hyw
2010 թվականի ապրիլին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 2010
@ast
im April 2010 veröffentlichter wissenschaftlicher Artikel
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scientific journal article
@en
vedecký článok (publikovaný 2010/04/14)
@sk
vědecký článek publikovaný v roce 2010
@cs
wetenschappelijk artikel (gepubliceerd op 2010/04/14)
@nl
наукова стаття, опублікована у квітні 2010
@uk
مقالة علمية (نشرت في 14-4-2010)
@ar
name
Essential role of the redox-se ...... te in neuronal preconditioning
@ast
Essential role of the redox-se ...... te in neuronal preconditioning
@en
Essential role of the redox-se ...... te in neuronal preconditioning
@nl
type
label
Essential role of the redox-se ...... te in neuronal preconditioning
@ast
Essential role of the redox-se ...... te in neuronal preconditioning
@en
Essential role of the redox-se ...... te in neuronal preconditioning
@nl
prefLabel
Essential role of the redox-se ...... te in neuronal preconditioning
@ast
Essential role of the redox-se ...... te in neuronal preconditioning
@en
Essential role of the redox-se ...... te in neuronal preconditioning
@nl
P2093
P2860
P1476
Essential role of the redox-se ...... te in neuronal preconditioning
@en
P2093
Benjamin Holt
Erik S. Musiek
Jacquelynn E. Brown
Jane C. Hettinger
Jason D. Morrow
Joshua D. Brooks
Stephanie L. H. Zeiger
P2860
P304
P356
10.1523/JNEUROSCI.6366-09.2010
P407
P577
2010-04-14T00:00:00Z