Deletion of the ageing gene p66(Shc) reduces early stroke size following ischaemia/reperfusion brain injury.
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The endothelium abridges insulin resistance to premature agingThe Dual Function of Reactive Oxygen/Nitrogen Species in Bioenergetics and Cell Death: The Role of ATP SynthaseAging-related alterations in eNOS and nNOS responsiveness and smooth muscle reactivity of murine basilar arteries are modulated by apocynin and phosphorylation of myosin phosphatase targeting subunit-1Molecular mechanism of endothelial and vascular aging: implications for cardiovascular disease.Selective inhibition of protein kinase C β2 attenuates the adaptor P66 Shc-mediated intestinal ischemia-reperfusion injuryMonoamine oxidases as sources of oxidants in the heartDual role of endothelial nitric oxide synthase in oxidized LDL-induced, p66Shc-mediated oxidative stress in cultured human endothelial cells.Activated protein C ameliorates diabetic nephropathy by epigenetically inhibiting the redox enzyme p66ShcChronic nicotine exposure augments renal oxidative stress and injury through transcriptional activation of p66shc.Molecular pathways of arterial aging.P66Shc-SIRT1 Regulation of Oxidative Stress Protects Against Cardio-cerebral Vascular Disease.Ischemia/Reperfusion.Lack of Contribution of p66shc and Its Mitochondrial Translocation to Ischemia-Reperfusion Injury and Cardioprotection by Ischemic Preconditioning.Electron Paramagnetic Resonance Measurements of Reactive Oxygen Species by Cyclic Hydroxylamine Spin Probes.Post-ischaemic silencing of p66Shc reduces ischaemia/reperfusion brain injury and its expression correlates to clinical outcome in stroke.Neuroprotective effects of allicin on ischemia-reperfusion brain injury.Shooting vascular oxidative stress: new hopes for stroke patients?The p66ShcA adaptor protein regulates healing after myocardial infarction.Genetic deletion of the adaptor protein p66Shc increases susceptibility to short-term ischaemic myocardial injury via intracellular salvage pathways.Modulating the p66shc signaling pathway with protocatechuic acid protects the intestine from ischemia-reperfusion injury and alleviates secondary liver damage.
P2860
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P2860
Deletion of the ageing gene p66(Shc) reduces early stroke size following ischaemia/reperfusion brain injury.
description
2012 nî lūn-bûn
@nan
2012年の論文
@ja
2012年学术文章
@wuu
2012年学术文章
@zh
2012年学术文章
@zh-cn
2012年学术文章
@zh-hans
2012年学术文章
@zh-my
2012年学术文章
@zh-sg
2012年學術文章
@yue
2012年學術文章
@zh-hant
name
Deletion of the ageing gene p66
@nl
Deletion of the ageing gene p6 ...... emia/reperfusion brain injury.
@en
type
label
Deletion of the ageing gene p66
@nl
Deletion of the ageing gene p6 ...... emia/reperfusion brain injury.
@en
prefLabel
Deletion of the ageing gene p66
@nl
Deletion of the ageing gene p6 ...... emia/reperfusion brain injury.
@en
P2093
P2860
P50
P356
P1476
Deletion of the ageing gene p6 ...... emia/reperfusion brain injury.
@en
P2093
Bruno Weber
Christoph Hock
Francesco Paneni
Giovanni G Camici
Matthias M Wyss
Nadine Lauinger
Remo D Spescha
Roger M Nitsch
Stephan Keller
Susanne Wegener
P2860
P304
P356
10.1093/EURHEARTJ/EHS331
P577
2012-09-24T00:00:00Z