Increased smad expression and activation are associated with apoptosis in normal and malignant prostate after castration
about
Interaction between Smad7 and beta-catenin: importance for transforming growth factor beta-induced apoptosisGonadotropin releasing hormone analogue (GnRHa) alters the expression and activation of Smad in human endometrial epithelial and stromal cellsOestrogens and oestrogen receptors in prostate cancerVariable expression of cystatin C in cultured trans-differentiating rat hepatic stellate cellsTMPRSS2-ERG -specific transcriptional modulation is associated with prostate cancer biomarkers and TGF-β signaling.Stromal transforming growth factor-beta signaling mediates prostatic response to androgen ablation by paracrine Wnt activityEstrogen opposes the apoptotic effects of bone morphogenetic protein 7 on tissue remodeling.Androgen receptor status predicts response to chemotherapy, not risk of breast cancer in Indian women.DHT selectively reverses Smad3-mediated/TGF-beta-induced responses through transcriptional down-regulation of Smad3 in prostate epithelial cellsThe loss of TGF-beta signaling promotes prostate cancer metastasis.Quinazoline-based alpha 1-adrenoceptor antagonists induce prostate cancer cell apoptosis via TGF-beta signalling and I kappa B alpha inductionAndrogenic control of transforming growth factor-beta signaling in prostate epithelial cells through transcriptional suppression of transforming growth factor-beta receptor II.Prostate cancer: the need for biomarkers and new therapeutic targets.The dual role of Smad7 in the control of cancer growth and metastasis.Androgen receptor and microRNA-21 axis downregulates transforming growth factor beta receptor II (TGFBR2) expression in prostate cancer.Amplification of PVT-1 is involved in poor prognosis via apoptosis inhibition in colorectal cancers.APC and Smad7 link TGFβ type I receptors to the microtubule system to promote cell migration.Overexpression of Smad7 results in severe pathological alterations in multiple epithelial tissues.Blockade of transforming growth factor beta/Smad signaling in T cells by overexpression of Smad7 enhances antigen-induced airway inflammation and airway reactivity.APIM-peptide targeting PCNA improves the efficacy of docetaxel treatment in the TRAMP mouse model of prostate cancer.
P2860
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P2860
Increased smad expression and activation are associated with apoptosis in normal and malignant prostate after castration
description
1999 թուականի Յունիսին հրատարակուած գիտական յօդուած
@hyw
1999 թվականի հունիսին հրատարակված գիտական հոդված
@hy
artículu científicu espublizáu en 1999
@ast
im Juni 1999 veröffentlichter wissenschaftlicher Artikel
@de
scientific journal article
@en
vedecký článok (publikovaný 1999/06/01)
@sk
vědecký článek publikovaný v roce 1999
@cs
wetenschappelijk artikel (gepubliceerd op 1999/06/01)
@nl
наукова стаття, опублікована в червні 1999
@uk
مقالة علمية (نشرت في يونيو 1999)
@ar
name
Increased smad expression and ...... nant prostate after castration
@ast
Increased smad expression and ...... nant prostate after castration
@en
Increased smad expression and ...... nant prostate after castration
@nl
type
label
Increased smad expression and ...... nant prostate after castration
@ast
Increased smad expression and ...... nant prostate after castration
@en
Increased smad expression and ...... nant prostate after castration
@nl
prefLabel
Increased smad expression and ...... nant prostate after castration
@ast
Increased smad expression and ...... nant prostate after castration
@en
Increased smad expression and ...... nant prostate after castration
@nl
P2093
P3181
P1433
P1476
Increased smad expression and ...... nant prostate after castration
@en
P2093
P304
P3181
P407
P577
1999-06-01T00:00:00Z